Increased degranulation of natural killer cells during acute HCV correlates with the magnitude of virus-specific T cell responses. - Wikidata - awgldk - TriplyDB

Increased degranulation of natural killer cells during acute HCV correlates with the magnitude of virus-specific T cell responses.

Increased degranulation of natural killer cells during acute HCV correlates with the magnitude of virus-specific T cell responses.

http://www.wikidata.org/entity/Q34258994

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Pathogenesis of hepatitis C during pregnancy and childhood Human NK Cell Diversity in Viral Infection: Ramifications of Ramification Natural killer cells in hepatitis C: Current progress Vertically acquired hepatitis C virus infection: Correlates of transmission and disease progression NK cell function and receptor diversity in the context of HCV infection Vitamin D and chronic hepatitis C: effects on success rate and prevention of side effects associated with pegylated interferon-α and ribavirin Natural killer cell responses during viral infections: flexibility and conditioning of innate immunity by experience Natural killer cells in liver disease Extracellular HCV-core protein induces an immature regulatory phenotype in NK cells: implications for outcome of acute infection Long-term effect on natural killer cells by interferon-α therapy on the outcomes of HCV infection.Protective immunity against hepatitis C: many shades of gray.Chronic HCV infection affects the NK cell phenotype in the blood more than in the liver.Immune control and failure in HCV infection--tipping the balance.Natural Killer Cells in Viral Hepatitis.KIR/HLA interactions and pathogen immunity PEG-IFN alpha but not ribavirin alters NK cell phenotype and function in patients with chronic hepatitis C Functions of liver natural killer cells are dependent on the severity of liver inflammation and fibrosis in chronic hepatitis C.Activins and Follistatin in Chronic Hepatitis C and Its Treatment with Pegylated-Interferon-α Based Therapy.Antiviral treatment alters the frequency of activating and inhibitory receptor-expressing natural killer cells in chronic hepatitis B virus infected patients.Natural killer and natural killer T cells in liver fibrosis Sustained hyperresponsiveness of dendritic cells is associated with spontaneous resolution of acute hepatitis C.Innate immune responses in hepatitis C virus-exposed healthcare workers who do not develop acute infection Reduced frequencies of NKp30+NKp46+, CD161+, and NKG2D+ NK cells in acute HCV infection may predict viral clearance.Natural killer cells: multifaceted players with key roles in hepatitis C immunity.IFN-λ3 polymorphism indirectly influences NK cell phenotype and function during acute HCV infection The natural killer cell response to HCV infection.Acute Liver Damage Associated with Innate Immune Activation in a Small Nonhuman Primate Model of Hepacivirus Infection.Between Scylla and Charybdis: the role of the human immune system in the pathogenesis of hepatitis C.Involvement of activating NK cell receptors and their modulation in pathogen immunity NK cells prevalence, subsets and function in viral hepatitis C.Activation of natural killer cells during microbial infections Hepatitis C virus-mediated modulation of cellular immunity.Organ system view of the hepatic innate immunity in HCV infection.Immune regulation in chronic hepatitis C virus infection.An effective interferon-gamma-mediated inhibition of hepatitis C virus replication by natural killer cells is associated with spontaneous clearance of acute hepatitis C in human immunodeficiency virus-positive patients.IFN-γ production by human natural killer cells in response to HCV-infected hepatoma cells is dependent on accessory cells.Natural killer cells in highly exposed hepatitis C-seronegative injecting drug users.Innate and Adaptive Immune Responses in Chronic HCV Infection.NKp46+ expression on NK cells as a biomarker for liver pathology and IFN-responiveness in HCV infection.The 3rd Canadian Symposium on Hepatitis C Virus: expanding care in the interferon-free era.

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P2860

Increased degranulation of natural killer cells during acute HCV correlates with the magnitude of virus-specific T cell responses.

http://www.wikidata.org/entity/Q34258994

description

2010 nî lūn-bûn

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2010 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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2010 թվականի հուլիսին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Increased degranulation of nat ...... rus-specific T cell responses.

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Increased degranulation of nat ...... rus-specific T cell responses.

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Increased degranulation of nat ...... rus-specific T cell responses.

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type

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Increased degranulation of nat ...... rus-specific T cell responses.

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Increased degranulation of nat ...... rus-specific T cell responses.

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Increased degranulation of nat ...... rus-specific T cell responses.

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Increased degranulation of nat ...... rus-specific T cell responses.

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Increased degranulation of nat ...... rus-specific T cell responses.

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Increased degranulation of nat ...... rus-specific T cell responses.

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P356

J.JHEP.2010.05.013

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Journal of Hepatology

P1476

Increased degranulation of nat ...... rus-specific T cell responses.

@en

P2093

Christian Drouin

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Julie Bruneau

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Naglaa H Shoukry

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Nathalie Bédard

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Salim I Khakoo

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Sandy Pelletier

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P2860

Human dendritic cells activate resting natural killer (NK) cells and are recognized via the NKp30 receptor by activated NK cells

Full-breadth analysis of CD8+ T-cell responses in acute hepatitis C virus infection and early therapy.

Impairment of natural killer (NK) cytotoxic activity in hepatitis C virus (HCV) infection

Inhibition of Natural Killer Cells through Engagement of CD81 by the Major Hepatitis C Virus Envelope Protein

Binding of the Hepatitis C Virus Envelope Protein E2 to CD81 Inhibits Natural Killer Cell Functions

Interaction of NK inhibitory receptor genes with HLA-C and MHC class II alleles in Hepatitis C virus infection outcome

Early Interferon Therapy for Hepatitis C Virus Infection Rescues Polyfunctional, Long-Lived CD8+ Memory T Cells

NK-dependent DC maturation is mediated by TNFalpha and IFNgamma released upon engagement of the NKp30 triggering receptor

Course and outcome of hepatitis C

Natural killer cells are polarized toward cytotoxicity in chronic hepatitis C in an interferon-alfa-dependent manner

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805-816

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scholarly article

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10.1016/J.JHEP.2010.05.013

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5

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53

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2010-07-17T00:00:00Z

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45507811

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20688412

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4178223

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