Poly(I:C) treatment leads to interferon-dependent clearance of hepatitis B virus in a hydrodynamic injection mouse model.
about
Toll-like receptor (TLR)-mediated innate immune responses in the control of hepatitis B virus (HBV) infection.Hepatitis B virus polymerase disrupts K63-linked ubiquitination of STING to block innate cytosolic DNA-sensing pathwaysCharacterization of the Treg Response in the Hepatitis B Virus Hydrodynamic Injection Mouse Model.Canonical Inflammasomes Drive IFN-γ to Prime Caspase-11 in Defense against a Cytosol-Invasive Bacterium.Transposon mouse models to elucidate the genetic mechanisms of hepatitis B viral induced hepatocellular carcinoma.Regulation of TLR3 Activation by S100A9.Hepatitis B virus genome replication triggers toll-like receptor 3-dependent interferon responses in the absence of hepatitis B surface antigenContribution of Toll-like receptors to the control of hepatitis B virus infection by initiating antiviral innate responses and promoting specific adaptive immune responses.Treatment of chronic hepatitis B with pattern recognition receptor agonists: Current status and potential for a cureToward RNA nanoparticle vaccines: synergizing RNA and inorganic nanoparticles to achieve immunopotentiation.Activating the innate immune response to counter chronic hepatitis B virus infection.Immune Evasion Strategies during Chronic Hepatitis B and C Virus InfectionHIV-hepatitis B virus co-infection: epidemiology, pathogenesis, and treatment.The IL-1R/TLR signaling pathway is essential for efficient CD8(+) T-cell responses against hepatitis B virus in the hydrodynamic injection mouse model.Matrix Metalloproteinase 9 Facilitates Hepatitis B Virus Replication through Binding with Type I Interferon (IFN) Receptor 1 To Repress IFN/JAK/STAT Signaling.TLR2 Expression in Peripheral CD4+ T Cells Promotes Th17 Response and Is Associated with Disease Aggravation of Hepatitis B Virus-Related Acute-On-Chronic Liver Failure.Interaction between Hepatitis B Virus and Toll-Like Receptors: Current Status and Potential Therapeutic Use for Chronic Hepatitis B.Raf Kinase Inhibitor Protein Preferentially Promotes TLR3-Triggered Signaling and Inflammation.Animal models for the study of hepatitis B virus infection.Drugs in Development for Hepatitis B.Hepatitis B Virus-Specific CD8+ T Cells Maintain Functional Exhaustion after Antigen Reexposure in an Acute Activation Immune Environment.
P2860
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P2860
Poly(I:C) treatment leads to interferon-dependent clearance of hepatitis B virus in a hydrodynamic injection mouse model.
description
2014 nî lūn-bûn
@nan
2014 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
name
Poly(I:C) treatment leads to i ...... dynamic injection mouse model.
@ast
Poly(I:C) treatment leads to i ...... dynamic injection mouse model.
@en
type
label
Poly(I:C) treatment leads to i ...... dynamic injection mouse model.
@ast
Poly(I:C) treatment leads to i ...... dynamic injection mouse model.
@en
prefLabel
Poly(I:C) treatment leads to i ...... dynamic injection mouse model.
@ast
Poly(I:C) treatment leads to i ...... dynamic injection mouse model.
@en
P2093
P2860
P356
P1433
P1476
Poly(I:C) treatment leads to i ...... dynamic injection mouse model.
@en
P2093
Baoju Wang
Dongliang Yang
Ejuan Zhang
Jingjiao Song
Joerg F Schlaak
Junzhong Wang
Mingfa Chen
P2860
P304
10421-10431
P356
10.1128/JVI.00996-14
P407
P577
2014-06-11T00:00:00Z