Altered retinoid uptake and action contributes to cell survival in endometriosis.
about
The role of lipoxin A4 in endometrial biology and endometriosisEndometriosis: hormone regulation and clinical consequences of chemotaxis and apoptosisPathogenesis of Endometriosis: Roles of Retinoids and Inflammatory PathwaysThe dynamics of nuclear receptors and nuclear receptor coregulators in the pathogenesis of endometriosis.Progesterone action in endometrial cancer, endometriosis, uterine fibroids, and breast cancer.Fenretinide: a novel treatment for endometrial cancer.Endometriosis expresses a molecular pattern consistent with decreased retinoid uptake, metabolism and action.Genome-wide DNA methylation analysis predicts an epigenetic switch for GATA factor expression in endometriosis.Retinoic acid biosynthesis is impaired in human and murine endometriosis.Retinoic acid inhibits endometrial cancer cell growth via multiple genomic mechanismsRetinoic acid has the potential to suppress endometriosis development.Retinoic acid suppresses growth of lesions, inhibits peritoneal cytokine secretion, and promotes macrophage differentiation in an immunocompetent mouse model of endometriosis.Reprogramming of the retinoic acid pathway in decidualizing human endometrial stromal cells.Effects of simvastatin on retinoic acid system in primary human endometrial stromal cells and in a chimeric model of human endometriosis.Local and systemic factors and implantation: what is the evidence?An overview of early drug development for endometriosis.KRAS Activation and over-expression of SIRT1/BCL6 Contributes to the Pathogenesis of Endometriosis and Progesterone Resistance.Progesterone resistance in endometriosis: origins, consequences and interventions.Retinoic acid regulates cell-shape and -death of E-FABP (FABP5)-immunoreactive septoclasts in the growth plate cartilage of mice.Pre- and post-partum mild underfeeding influences gene expression in the reproductive tract of cyclic dairy cows.Retinoic acid regulates gap junction intercellular communication in human endometrial stromal cells through modulation of the phosphorylation status of connexin 43.The efficacy of bevacizumab, sorafenib, and retinoic acid on rat endometriosis model.Fenretinide: A Potential Treatment for Endometriosis.
P2860
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P2860
Altered retinoid uptake and action contributes to cell survival in endometriosis.
description
2010 nî lūn-bûn
@nan
2010 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Altered retinoid uptake and action contributes to cell survival in endometriosis.
@ast
Altered retinoid uptake and action contributes to cell survival in endometriosis.
@en
Altered retinoid uptake and action contributes to cell survival in endometriosis.
@nl
type
label
Altered retinoid uptake and action contributes to cell survival in endometriosis.
@ast
Altered retinoid uptake and action contributes to cell survival in endometriosis.
@en
Altered retinoid uptake and action contributes to cell survival in endometriosis.
@nl
prefLabel
Altered retinoid uptake and action contributes to cell survival in endometriosis.
@ast
Altered retinoid uptake and action contributes to cell survival in endometriosis.
@en
Altered retinoid uptake and action contributes to cell survival in endometriosis.
@nl
P2093
P2860
P356
P1476
Altered retinoid uptake and action contributes to cell survival in endometriosis
@en
P2093
Scott Reierstad
Serdar E Bulun
You-Hong Cheng
P2860
P304
P356
10.1210/JC.2010-0459
P407
P577
2010-08-11T00:00:00Z