Insights into battles between Mycobacterium tuberculosis and macrophages. - Wikidata - awgldk - TriplyDB

Insights into battles between Mycobacterium tuberculosis and macrophages.

Insights into battles between Mycobacterium tuberculosis and macrophages.

http://www.wikidata.org/entity/Q34268873

about

Mycobacterium tuberculosis PE_PGRS41 Enhances the Intracellular Survival of M. smegmatis within Macrophages Via Blocking Innate Immunity and Inhibition of Host Defense.Mycobacterial infection induces a specific human innate immune response.Genetic polymorphisms of CCL1 rs2072069 G/A and TLR2 rs3804099 T/C in pulmonary or meningeal tuberculosis patients Mycobacterium bovis Induces Endoplasmic Reticulum Stress Mediated-Apoptosis by Activating IRF3 in a Murine Macrophage Cell Line.Mycobacterial Dormancy Systems and Host Responses in Tuberculosis.Patho-epigenetics of Infectious Diseases Caused by Intracellular Bacteria.Mycobacterium tuberculosis suppresses innate immunity by coopting the host ubiquitin system.Recombinant TB9.8 of Mycobacterium bovis Triggers the Production of IL-12 p40 and IL-6 in RAW264.7 Macrophages via Activation of the p38, ERK, and NF-κB Signaling Pathways.Role of Interferons in the Development of Diagnostics, Vaccines, and Therapy for Tuberculosis.Quantitative proteomic analysis of host responses triggered by Mycobacterium tuberculosis infection in human macrophage cells.Activation of TLR7 Inhibition of Mycobacterium Tuberculosis Survival by Autophagy in RAW 264.7 Macrophages.Systematic Identification of Mycobacterium tuberculosis Effectors Reveals that BfrB Suppresses Innate Immunity.Analysis of host-pathogen modulators of autophagy during Mycobacterium Tuberculosis infection and therapeutic repercussions.Integrin CD11b negatively regulates Mincle-induced signaling via the Lyn-SIRPα-SHP1 complex.A pore-forming toxin enables Serratia a nonlytic egress from host cells.De Novo Fatty Acid Synthesis During Mycobacterial Infection Is a Prerequisite for the Function of Highly Proliferative T Cells, But Not for Dendritic Cells or Macrophages.Vitamin B5 Reduces Bacterial Growth via Regulating Innate Immunity and Adaptive Immunity in Mice Infected with Mycobacterium tuberculosis.MptpB Promotes Mycobacteria Survival by Inhibiting the Expression of Inflammatory Mediators and Cell Apoptosis in Macrophages.The functional interplay of low molecular weight thiols in Mycobacterium tuberculosis.Rv2346c enhances mycobacterial survival within macrophages by inhibiting TNF-α and IL-6 production via the p38/miRNA/NF-κB pathway Vitamin B1 Helps to Limit Growth Regulating Innate Immunity in a Peroxisome Proliferator-Activated Receptor-γ-Dependent Manner

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Insights into battles between Mycobacterium tuberculosis and macrophages.

http://www.wikidata.org/entity/Q34268873

description

2014 nî lūn-bûn

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2014 թուականի Յունիսին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի հունիսին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

@yue

2014年論文

@zh-hant

2014年論文

@zh-hk

2014年論文

@zh-mo

2014年論文

@zh-tw

2014年论文

@wuu

name

Insights into battles between Mycobacterium tuberculosis and macrophages.

@ast

Insights into battles between Mycobacterium tuberculosis and macrophages.

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Insights into battles between Mycobacterium tuberculosis and macrophages.

@nl

type

label

Insights into battles between Mycobacterium tuberculosis and macrophages.

@ast

Insights into battles between Mycobacterium tuberculosis and macrophages.

@en

Insights into battles between Mycobacterium tuberculosis and macrophages.

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Insights into battles between Mycobacterium tuberculosis and macrophages.

@ast

Insights into battles between Mycobacterium tuberculosis and macrophages.

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Insights into battles between Mycobacterium tuberculosis and macrophages.

@nl

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Insights into battles between Mycobacterium tuberculosis and macrophages.

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Cui Hua Liu

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Guanghua Xu

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Jing Wang

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P2860

The mitochondrial phosphatase PGAM5 functions at the convergence point of multiple necrotic death pathways

1,25-Dihydroxyvitamin D3 induces nitric oxide synthase and suppresses growth of Mycobacterium tuberculosis in a human macrophage-like cell line

Immunomodulatory functions of type I interferons

Critical role of type I interferon-induced macrophage necroptosis during infection with Salmonella enterica serovar Typhimurium

The innate immune response to bacterial flagellin is mediated by Toll-like receptor 5

RIPK3 contributes to TNFR1-mediated RIPK1 kinase-dependent apoptosis in conditions of cIAP1/2 depletion or TAK1 kinase inhibition

Toll-like receptor triggering of a vitamin D-mediated human antimicrobial response

Mycobacterium tuberculosis nuoG is a virulence gene that inhibits apoptosis of infected host cells

Protein kinase E of Mycobacterium tuberculosis has a role in the nitric oxide stress response and apoptosis in a human macrophage model of infection

Apoptosis and interferons: role of interferon-stimulated genes as mediators of apoptosis

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