Gammaherpesvirus latency accentuates EAE pathogenesis: relevance to Epstein-Barr virus and multiple sclerosis.
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Infection as an Environmental Trigger of Multiple Sclerosis Disease ExacerbationEnhanced response of T cells from murine gammaherpesvirus 68-infected mice lacking the suppressor of T cell receptor signaling molecules Sts-1 and Sts-2The Multibiome: The Intestinal Ecosystem's Influence on Immune Homeostasis, Health, and DiseaseCoinfections: Another Variable in the Herpesvirus Latency-Reactivation DynamicGene expression profiling of the response to interferon beta in Epstein-Barr-transformed and primary B cells of patients with multiple sclerosisLatent gammaherpesvirus 68 infection induces distinct transcriptional changes in different organsKingdom-agnostic metagenomics and the importance of complete characterization of enteric microbial communities.Gammaherpesvirus latency induces antibody-associated thrombocytopenia in mice.Cytomegalovirus infection exacerbates autoimmune mediated neuroinflammation.Induction of encephalitis in rhesus monkeys infused with lymphocryptovirus-infected B-cells presenting MOG(34-56) peptide.The Role of Latently Infected B Cells in CNS Autoimmunity.Th40 cells (CD4+CD40+ Tcells) drive a more severe form of Experimental Autoimmune Encephalomyelitis than conventional CD4 T cells.Cytokine expression profiles of immune imbalance in post-mononucleosis chronic fatigue.Virus-induced CD8+ T cells accelerate the onset of experimental autoimmune encephalomyelitis: implications for how viral infections might trigger multiple sclerosis exacerbations.Environmental factors acting during development to influence MS risk: insights from animal studies.Murine Cytomegalovirus Infection Induces Susceptibility to EAE in Resistant BALB/c Mice.The Primate EAE Model Points at EBV-Infected B Cells as a Preferential Therapy Target in Multiple Sclerosis.Latent virus infection upregulates CD40 expression facilitating enhanced autoimmunity in a model of multiple sclerosis.Gammaherpesvirus latency differentially impacts the generation of primary versus secondary memory CD8+ T cells during subsequent infection.The different clinical effects of anti-BLyS, anti-APRIL and anti-CD20 antibodies point at a critical pathogenic role of γ-herpesvirus infected B cells in the marmoset EAE model.Beta and Gamma Human Herpesviruses: Agonistic and Antagonistic Interactions with the Host Immune System.Murine Gammaherpesvirus 68: A Small Animal Model for Gammaherpesvirus-Associated Diseases.Herpesvirus trigger accelerates neuroinflammation in a nonhuman primate model of multiple sclerosisEvodiamine alleviates severe pneumonia induced by methicillin-susceptible Staphylococcus aureus following cytomegalovirus reactivation through suppressing NF-κB and MAPKs
P2860
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P2860
Gammaherpesvirus latency accentuates EAE pathogenesis: relevance to Epstein-Barr virus and multiple sclerosis.
description
2012 nî lūn-bûn
@nan
2012 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2012 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
name
Gammaherpesvirus latency accen ...... virus and multiple sclerosis.
@ast
Gammaherpesvirus latency accen ...... virus and multiple sclerosis.
@en
Gammaherpesvirus latency accen ...... virus and multiple sclerosis.
@nl
type
label
Gammaherpesvirus latency accen ...... virus and multiple sclerosis.
@ast
Gammaherpesvirus latency accen ...... virus and multiple sclerosis.
@en
Gammaherpesvirus latency accen ...... virus and multiple sclerosis.
@nl
prefLabel
Gammaherpesvirus latency accen ...... virus and multiple sclerosis.
@ast
Gammaherpesvirus latency accen ...... virus and multiple sclerosis.
@en
Gammaherpesvirus latency accen ...... virus and multiple sclerosis.
@nl
P2093
P2860
P1433
P1476
Gammaherpesvirus latency accen ...... r virus and multiple sclerosis
@en
P2093
Iryna Shanina
Marc S Horwitz
Marcia A Blackman
Michael L Freeman
Sehyun Cho
P2860
P304
P356
10.1371/JOURNAL.PPAT.1002715
P577
2012-05-17T00:00:00Z