Calcium-activated potassium current modulates ventricular repolarization in chronic heart failure. - Wikidata - awgldk - TriplyDB

Calcium-activated potassium current modulates ventricular repolarization in chronic heart failure.

Calcium-activated potassium current modulates ventricular repolarization in chronic heart failure.

http://www.wikidata.org/entity/Q34278261

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Small-conductance Ca2+ -activated K+ channels and cardiac arrhythmias SK channels and ventricular arrhythmias in heart failure Apamin-sensitive calcium-activated potassium currents in rabbit ventricles with chronic myocardial infarction.Small conductance calcium-activated potassium current is important in transmural repolarization of failing human ventricles.Patients with Dilated Cardiomyopathy and Sustained Monomorphic Ventricular Tachycardia Show Up-Regulation of KCNN3 and KCNJ2 Genes and CACNG8-Linked Left Ventricular Dysfunction The chemopreventive potential of lycopene against atrazine-induced cardiotoxicity: modulation of ionic homeostasis Small conductance calcium-activated potassium current and the mechanism of atrial arrhythmia in mice with dysfunctional melanocyte-like cells.Dysfunction of the β2-spectrin-based pathway in human heart failure.Sarcoplasmic reticulum Ca²⁺ release is both necessary and sufficient for SK channel activation in ventricular myocytes.A novel mechanism underlies atrazine toxicity in quails (Coturnix Coturnix coturnix): triggering ionic disorder via disruption of ATPases.Role of Calcium-activated Potassium Channels in Atrial Fibrillation Pathophysiology and Therapy.SK channel enhancers attenuate Ca2+-dependent arrhythmia in hypertrophic hearts by regulating mito-ROS-dependent oxidation and activity of RyR.Are SK channels a logical target for treating ventricular arrhythmias? First, do no harm.Termination of Vernakalant-Resistant Atrial Fibrillation by Inhibition of Small-Conductance Ca2+-Activated K+ Channels in Pigs.Functional interaction of Junctophilin 2 with small- conductance Ca2+ -activated potassium channel subtype 2(SK2) in mouse cardiac myocytes.Role of Apamin-Sensitive Calcium-Activated Small-Conductance Potassium Currents on the Mechanisms of Ventricular Fibrillation in Pacing-Induced Failing Rabbit Hearts.Apamin-Sensitive Small Conductance Calcium-Activated Potassium Channels were Negatively Regulated by Captopril in Volume-Overload Heart Failure Rats.Small-conductance Ca2+-activated K+ channels: insights into their roles in cardiovascular disease.Calcium in the Pathophysiology of Atrial Fibrillation and Heart Failure

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Calcium-activated potassium current modulates ventricular repolarization in chronic heart failure.

http://www.wikidata.org/entity/Q34278261

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2014 nî lūn-bûn

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2014 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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Ahmet Kilic

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Andriy Belevych

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Cynthia A Carnes

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Jae Yoo

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Kent Mowrey

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Paulus M L Janssen

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Philip F Binkley

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P2860

Ablation of a Ca2+-activated K+ channel (SK2 channel) results in action potential prolongation in atrial myocytes and atrial fibrillation

Calcium-activated potassium channels

Small conductance calcium-activated potassium channels: from structure to function

International Union of Pharmacology. LII. Nomenclature and molecular relationships of calcium-activated potassium channels

Differential expression of small-conductance Ca2+-activated K+ channels SK1, SK2, and SK3 in mouse atrial and ventricular myocytes

Molecular properties of small-conductance Ca2+-activated K+ channels expressed in murine colonic smooth muscle

Apamin interacts with all subtypes of cloned small-conductance Ca2+-activated K+ channels.

Apamin does not inhibit human cardiac Na+ current, L-type Ca2+ current or other major K+ currents

Common variants in KCNN3 are associated with lone atrial fibrillation.

Abnormal intrastore calcium signaling in chronic heart failure.

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266381635

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