α-Thalassemia impairs the cytoadherence of Plasmodium falciparum-infected erythrocytes
about
Biochemical and immunological mechanisms by which sickle cell trait protects against malariaHemoglobinopathies: slicing the Gordian knot of Plasmodium falciparum malaria pathogenesisThe influence of host genetics on erythrocytes and malaria infection: is there therapeutic potential?Abnormal PfEMP1/knob display on Plasmodium falciparum-infected erythrocytes containing hemoglobin variants: fresh insights into malaria pathogenesis and protectionModeling cytoadhesion of Plasmodium falciparum-infected erythrocytes and leukocytes-common principles and distinctive featuresHemoglobinopathic erythrocytes affect the intraerythrocytic multiplication of Plasmodium falciparum in vitroThe density of knobs on Plasmodium falciparum-infected erythrocytes depends on developmental age and varies among isolates.Ex-vivo cytoadherence phenotypes of Plasmodium falciparum strains from Malian children with hemoglobins A, S, and C.Genetic heterogeneity of the β-globin gene in various geographic populations of Yunnan in southwestern ChinaMechanistic Studies of the Negative Epistatic Malaria-protective Interaction Between Sickle Cell Trait and α(+)thalassemiaα(+)Thalassemia Antagonizes the Malaria-protective Effects of Sickle-Cell TraitHemoglobin Constant Spring among Southeast Asian Populations: Haplotypic Heterogeneities and Phylogenetic Analysis.Ticket to ride: export of proteins to the Plasmodium falciparum-infected erythrocyte.The Prevalence of α-Thalassemia and Its Relation to Plasmodium falciparum Infection in Patients Presenting to Clinics in Two Distinct Ecological Zones in Ghana.The role of the red blood cell in host defence against falciparum malaria: an expanding repertoire of evolutionary alterations.Hemoglobin S and C affect protein export in Plasmodium falciparum-infected erythrocytes.Haemoglobin S and C affect the motion of Maurer's clefts in Plasmodium falciparum-infected erythrocytes.Analyzing the Formation, Morphology, and Integrity of Erythroblastic Islands.Two complement receptor one alleles have opposing associations with cerebral malaria and interact with α+thalassaemia.Erythrocyte β spectrin can be genetically targeted to protect mice from malariaAssociations between erythrocyte polymorphisms and risks of uncomplicated and severe malaria in Ugandan children: A case control study
P2860
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P2860
α-Thalassemia impairs the cytoadherence of Plasmodium falciparum-infected erythrocytes
description
2012 nî lūn-bûn
@nan
2012 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2012 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
name
α-Thalassemia impairs the cytoadherence of Plasmodium falciparum-infected erythrocytes
@ast
α-Thalassemia impairs the cytoadherence of Plasmodium falciparum-infected erythrocytes
@en
α-Thalassemia impairs the cytoadherence of Plasmodium falciparum-infected erythrocytes
@nl
type
label
α-Thalassemia impairs the cytoadherence of Plasmodium falciparum-infected erythrocytes
@ast
α-Thalassemia impairs the cytoadherence of Plasmodium falciparum-infected erythrocytes
@en
α-Thalassemia impairs the cytoadherence of Plasmodium falciparum-infected erythrocytes
@nl
prefLabel
α-Thalassemia impairs the cytoadherence of Plasmodium falciparum-infected erythrocytes
@ast
α-Thalassemia impairs the cytoadherence of Plasmodium falciparum-infected erythrocytes
@en
α-Thalassemia impairs the cytoadherence of Plasmodium falciparum-infected erythrocytes
@nl
P2093
P2860
P1433
P1476
α-Thalassemia impairs the cytoadherence of Plasmodium falciparum-infected erythrocytes
@en
P2093
Chanaki Amaratunga
Drissa Konate
Jeffrey R Keefer
Karim Traore
Michael A Krause
Saibou Doumbia
Seidina A S Diakite
Takayuki Arie
Tatiana M Lopera-Mesa
P2860
P304
P356
10.1371/JOURNAL.PONE.0037214
P407
P577
2012-05-18T00:00:00Z