IL-29 is the dominant type III interferon produced by hepatocytes during acute hepatitis C virus infection - Wikidata - awgldk - TriplyDB

IL-29 is the dominant type III interferon produced by hepatocytes during acute hepatitis C virus infection

IL-29 is the dominant type III interferon produced by hepatocytes during acute hepatitis C virus infection

http://www.wikidata.org/entity/Q34282251

about

IFNL3 mRNA structure is remodeled by a functional non-coding polymorphism associated with hepatitis C virus clearance The Dual Role of Exosomes in Hepatitis A and C Virus Transmission and Viral Immune Activation Hepatitis B and Delta Virus: Advances on Studies about Interactions between the Two Viruses and the Infected Hepatocyte Interferon Response in Hepatitis C Virus (HCV) Infection: Lessons from Cell Culture Systems of HCV Infection Interferon-λ: Immune Functions at Barrier Surfaces and Beyond Immune responses to HCV and other hepatitis viruses Emerging concepts in immunity to hepatitis C virus infection.Genetic variants at the IFNL3 locus and their association with hepatitis C virus infections reveal novel insights into host-virus interactions Interferon-λ4 is a cell-autonomous type III interferon associated with pre-treatment hepatitis C virus burden Elevation of Alanine Aminotransferase Activity Occurs after Activation of the Cell-Death Signaling Initiated by Pattern-Recognition Receptors ‎but before Activation of Cytolytic Effectors in NK or CD8+ T Cells in the Liver During Acute HCV Infection Hepatitis E virus persists in the presence of a type III interferon response.Zinc is a potent and specific inhibitor of IFN-λ3 signalling.Elucidating the host-pathogen interaction between human colorectal cells and invading Enterovirus 71 using transcriptomics profiling Protective immunity against hepatitis C: many shades of gray.IFN-λ4 potently blocks IFN-α signalling by ISG15 and USP18 in hepatitis C virus infection.Interferon lambda alleles predict innate antiviral immune responses and hepatitis C virus permissiveness An interferon response gene signature is associated with the therapeutic response of hepatitis C patients Immune control and failure in HCV infection--tipping the balance.HepG2 cells mount an effective antiviral interferon-lambda based innate immune response to hepatitis C virus infection.Interferon lambda: opportunities, risks, and uncertainties in the fight against HCV.Evaluation of IL-28B polymorphisms and serum IP-10 in hepatitis C infected chimpanzees.Albumin fusion of interleukin-28B: production and characterization of its biological activities and protein stability.Permissivity of primary human hepatocytes and different hepatoma cell lines to cell culture adapted hepatitis C virus.Control of hepatitis C virus replication in mouse liver-derived cells by MAVS-dependent production of type I and type III interferons.Hepatitis C Virus. Strategies to Evade Antiviral Responses Activation and evasion of antiviral innate immunity by hepatitis C virus.Monocytes activate natural killer cells via inflammasome-induced interleukin 18 in response to hepatitis C virus replication.Hepatitis C Virus Driven AXL Expression Suppresses the Hepatic Type I Interferon Response Activation of Type I and III Interferon Response by Mitochondrial and Peroxisomal MAVS and Inhibition by Hepatitis C Virus.Roles of unphosphorylated ISGF3 in HCV infection and interferon responsiveness.The Effect of Chronic Hepatitis B Virus Infection on BDCA3+ Dendritic Cell Frequency and Function.Permissivity of primary hepatocytes and hepatoma cell lines to support hepatitis C virus infection.Immunity and hepatitis C: a review.Hepatitis C virus infection induces autocrine interferon signaling by human liver endothelial cells and release of exosomes, which inhibits viral replication MicroRNA-548 down-regulates host antiviral response via direct targeting of IFN-λ1.Independent, parallel pathways to CXCL10 induction in HCV-infected hepatocytes.IFN-λ3 polymorphism indirectly influences NK cell phenotype and function during acute HCV infection HCV core protein inhibits polarization and activity of both M1 and M2 macrophages through the TLR2 signaling pathway.Type III Interferons in Hepatitis C Virus Infection.A paradox of transcriptional and functional innate interferon responses of human intestinal enteroids to enteric virus infection.

P2860

Q22121973-9BC82BB4-4431-453D-8BB0-1A7B56206EF6 Q26773325-1F98C1EF-D4CD-454D-9E78-0AB3382D608A Q26775635-9BE9B16E-6288-4DB1-8BEE-15E0148AE472 Q26784252-CC8D1BCC-9A4D-47FE-A74F-461E30A59F61 Q26801049-1D965538-3E12-4873-BAA3-D0470E918849 Q26866955-DBDAF9B2-4F75-47E2-8D24-1245E9BE0702 Q27002588-9E5F5EA5-1D95-4B30-8F96-3B9C613732D0 Q27014831-08C9BB4A-5642-4936-A0A7-7F1A844D4999 Q28255239-ABFB31B1-E67C-4E91-A90C-367402A9B0A7 Q28553005-C6CB990B-F8E1-4A22-B59A-2DCEEDE1CDC5 Q30100969-EF49ADFA-D12B-43A4-A4E3-DD15628B394F Q33720876-CE59D70B-2890-4CB8-867F-92A1320F2DC0 Q33729001-E8C40291-5B2F-4CA9-93B0-D178B84794AF Q33759224-1A3C155D-93D5-4F95-A9A0-E8F4C4B4A51B Q33812521-6345F460-F6F0-4830-AFEB-155BD511307E Q33922537-1FF78BC4-A4DD-4B4B-B4B8-AA2778B5C4E2 Q34027569-CD865FFD-6869-4DBA-808C-8E5D0ED9B527 Q34178628-CE6162EA-B38F-44FB-93A0-27205A13984B Q34250323-ED5C4FC1-1A4F-42DC-B411-607B363D3F1F Q34433087-149FD34C-0B47-4DC9-B1B9-3A150AB3B5A3 Q34464406-EF31DD5A-E1D9-482B-AE25-50052303C3FB Q34760439-BEC1C353-3F1F-4764-8E39-E7EBD55FA5DC Q34936072-F910213B-0548-4320-9092-EA7CBB28EA15 Q35488054-72B04104-E2AB-40F0-BDD8-158A378F61CE Q35604054-73783EEE-DAA0-4EFB-A14A-118D23929062 Q35606341-245523DE-F5A0-4FA3-851D-3DC08D80678A Q35748893-BE0304F3-9C93-440C-92A5-50D42D2D6A99 Q35757367-BB7CDC69-4B5F-4657-B8DF-E0D1D6A4FB29 Q35847364-CAF08203-0BED-4F69-A228-27DF52089CCE Q35989977-24ADCCEB-5F0C-4C70-9D9B-18C82D8B0DCE Q36104804-6A9EAC59-06BE-4829-817B-26F2F0F4993B Q36255092-0A292C4D-ADFB-465E-90F8-6C884390C4D6 Q36594965-8AC3ABA7-28DF-473E-90FF-45F3A6FE3CD2 Q36612971-E29B9AC4-6BFE-4E8B-BC0D-9E18DF6D5859 Q36922908-F4169D43-0D7E-4786-AF49-BF7CF56656C7 Q37188844-2098EF28-DE14-4513-9EA4-AFB3CEB9FC4E Q37216063-1963F1A3-E1AB-4141-AEAE-F8C4176829F5 Q37370720-2616714A-988A-441B-A5CF-034B3D001294 Q37528605-4E7C2D78-59F8-498D-9A70-CA05080CCDD0 Q37612580-D012D6CF-A0DE-4ACE-919F-2488E2554206

P2860

IL-29 is the dominant type III interferon produced by hepatocytes during acute hepatitis C virus infection

http://www.wikidata.org/entity/Q34282251

description

2012 nî lūn-bûn

@nan

2012 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

@hyw

2012 թվականի նոյեմբերին հրատարակված գիտական հոդված

@hy

2012年の論文

@ja

2012年論文

@yue

2012年論文

@zh-hant

2012年論文

@zh-hk

2012年論文

@zh-mo

2012年論文

@zh-tw

2012年论文

@wuu

name

IL-29 is the dominant type III ...... te hepatitis C virus infection

@ast

IL-29 is the dominant type III ...... te hepatitis C virus infection

@en

IL-29 is the dominant type III ...... te hepatitis C virus infection

@nl

type

label

IL-29 is the dominant type III ...... te hepatitis C virus infection

@ast

IL-29 is the dominant type III ...... te hepatitis C virus infection

@en

IL-29 is the dominant type III ...... te hepatitis C virus infection

@nl

prefLabel

IL-29 is the dominant type III ...... te hepatitis C virus infection

@ast

IL-29 is the dominant type III ...... te hepatitis C virus infection

@en

IL-29 is the dominant type III ...... te hepatitis C virus infection

@nl

P1433

Q34282251-2700779F-DD67-4EF5-8EB2-7BAD86DBD75B

P1476

Q34282251-08C411B4-92EA-4308-8BD9-1FE0431FBC23

P2093

Q34282251-0316C83D-7DFE-458D-B899-C3FFE884671E

Q34282251-06710615-6B3C-4FFF-BB53-76BF6F49C934

Q34282251-5742CC61-C0E7-4D0D-A4AA-4ACDAE053757

Q34282251-593336FC-EA39-42E3-87FB-57ED8B1505C8

Q34282251-608EC95B-CD06-49BA-8D1A-514B4EF4BC49

Q34282251-B767F8B5-D1AF-437D-9EDE-8FA509B51B81

Q34282251-D19FDA99-1D72-4953-A425-6CAA86056400

P2860

Q34282251-05865109-180D-41B2-9953-1668CF6944DB

Q34282251-28586C4A-5D6B-46B1-B5B1-5D16BC68DB34

Q34282251-29A81DCA-EDF1-4A99-BD45-77B48C70583B

Q34282251-2C44EAC2-ECDE-4D1B-9C90-732B6D372A57

Q34282251-2DC8270B-7197-41A7-B2EA-AFA92ADD23AC

Q34282251-35B81C53-5AEC-4E98-BFEE-6DCF5AD7A73B

Q34282251-4583BAED-E711-4A86-9492-1CDE1CF2D342

Q34282251-4ED640F9-49FB-4AB0-A526-52CE5B73141B

Q34282251-59649C52-2109-41A1-BBA5-AFDDE54C2B8B

Q34282251-65EDA5C0-E395-4974-A119-3E4856A79147

P304

Q34282251-96336D77-C482-4100-BE7D-2F0816325623

P31

Q34282251-2EE8B4A9-B971-4418-9319-AFBE86556851

P356

Q34282251-934B426C-9ABA-45E2-8916-AAF74E8C0837

P407

Q34282251-D9DCF65E-05C2-4E54-B58E-2B34D7086C63

P433

Q34282251-6042BB7D-62AE-4F76-B264-A69997468B51

P478

Q34282251-EC886FE6-899A-41B7-B1C9-226B1559C500

P50

Q34282251-25E40069-FDEB-4BF5-8ED0-477280554141

P577

Q34282251-53420EF7-1B3E-4AC2-94EA-687A64795499

P5875

Q34282251-7A176335-E786-41F4-A116-4C5F2D02F5FA

P698

Q34282251-46FD001C-C31A-4E1E-86F8-EBD6BD5F44CC

P932

Q34282251-9BB932E6-E75B-44CB-A890-95059C4E482F

P356

P1433

P1476

IL-29 is the dominant type III ...... te hepatitis C virus infection

@en

P2093

Antonella Folgori

http://www.w3.org/2001/XMLSchema#string

Barbara Rehermann

http://www.w3.org/2001/XMLSchema#string

Brian Muchmore

http://www.w3.org/2001/XMLSchema#string

Elisavet Serti

http://www.w3.org/2001/XMLSchema#string

Heiyoung Park

http://www.w3.org/2001/XMLSchema#string

Onyinyechi Eke

http://www.w3.org/2001/XMLSchema#string

Stefania Capone

http://www.w3.org/2001/XMLSchema#string

P2860

IFN-lambdas mediate antiviral protection through a distinct class II cytokine receptor complex

Genetic variation in IL28B and spontaneous clearance of hepatitis C virus

A diverse range of gene products are effectors of the type I interferon antiviral response

Lambda interferon inhibits hepatitis B and C virus replication.

DNA microarray analysis of chimpanzee liver during acute resolving hepatitis C virus infection.

Nonlinear partial differential equations and applications: Genomic analysis of the host response to hepatitis C virus infection

Hepatitis C virus versus innate and adaptive immune responses: a tale of coevolution and coexistence

Production of infectious hepatitis C virus in tissue culture from a cloned viral genome

IFN regulatory factor family members differentially regulate the expression of type III IFN (IFN-lambda) genes

Genetic variation in IL28B predicts hepatitis C treatment-induced viral clearance

P304

2060-2070

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1002/HEP.25897

http://www.w3.org/2001/XMLSchema#string

P407

P433

6

http://www.w3.org/2001/XMLSchema#string

P478

56

http://www.w3.org/2001/XMLSchema#string

P50

Ludmila Prokunina-Olsson

P577

2012-11-19T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

227176489

http://www.w3.org/2001/XMLSchema#string

P698

22706965

http://www.w3.org/2001/XMLSchema#string

P932

3581145

http://www.w3.org/2001/XMLSchema#string