Different roles for nonhomologous end joining and homologous recombination following replication arrest in mammalian cells.
about
WRN interacts physically and functionally with the recombination mediator protein RAD52RNAi-based screening identifies the Mms22L-Nfkbil2 complex as a novel regulator of DNA replication in human cells.Metnase promotes restart and repair of stalled and collapsed replication forksFunctional interaction between the Bloom's syndrome helicase and the RAD51 paralog, RAD51L3 (RAD51D)The cell-cycle checkpoint kinase Chk1 is required for mammalian homologous recombination repairMethyl methanesulfonate (MMS) produces heat-labile DNA damage but no detectable in vivo DNA double-strand breaks.Chk1 and p21 cooperate to prevent apoptosis during DNA replication fork stress6-thioguanine selectively kills BRCA2-defective tumors and overcomes PARP inhibitor resistanceRNA interference inhibition of Mus81 reduces mitotic recombination in human cellsPoly(ADP-ribose) polymerase (PARP-1) has a controlling role in homologous recombinationCreation of non-human primate neurogenetic disease models by gene targeting and nuclear transferSystematic quantification of gene interactions by phenotypic array analysisCharacterisation of cytotoxicity and DNA damage induced by the topoisomerase II-directed bisdioxopiperazine anti-cancer agent ICRF-187 (dexrazoxane) in yeast and mammalian cellsImpaired DNA replication within progenitor cell pools promotes leukemogenesis.Repression of mutagenesis by Rad51D-mediated homologous recombination.Ctf18 is required for homologous recombination-mediated double-strand break repairBalancing self-renewal against genome preservation in stem cells: How do they manage to have the cake and eat it too?Rad54 and DNA Ligase IV cooperate to maintain mammalian chromatid stabilityThe structure-specific endonuclease Ercc1-Xpf is required to resolve DNA interstrand cross-link-induced double-strand breaksPathways of DNA double-strand break repair during the mammalian cell cycleTargeting nucleophosmin 1 represents a rational strategy for radiation sensitization.Inhibition of human Chk1 causes increased initiation of DNA replication, phosphorylation of ATR targets, and DNA breakageDNA lesion-specific co-localization of the Mre11/Rad50/Nbs1 (MRN) complex and replication protein A (RPA) to repair foci.Differential involvement of phosphatidylinositol 3-kinase-related protein kinases in hyperphosphorylation of replication protein A2 in response to replication-mediated DNA double-strand breaks.Apoptosis induced by replication inhibitors in Chk1-depleted cells is dependent upon the helicase cofactor Cdc45.Impaired replication stress response in cells from immunodeficiency patients carrying Cernunnos/XLF mutations.Cellular pathways for DNA repair and damage tolerance of formaldehyde-induced DNA-protein crosslinksThe role of RPA2 phosphorylation in homologous recombination in response to replication arrestDifferential requirement for H2AX and 53BP1 in organismal development and genome maintenance in the absence of poly(ADP)ribosyl polymerase 1.Induction of homologous recombination following in utero exposure to DNA-damaging agents.Transcription inhibition by DRB potentiates recombinational repair of UV lesions in mammalian cells.Control of dTTP pool size by anaphase promoting complex/cyclosome is essential for the maintenance of genetic stabilityInfluence of homologous recombinational repair on cell survival and chromosomal aberration induction during the cell cycle in gamma-irradiated CHO cellsDNA-PK phosphorylation of RPA32 Ser4/Ser8 regulates replication stress checkpoint activation, fork restart, homologous recombination and mitotic catastrophe.More forks on the road to replication stress recoveryHydroxycarbamide: a user's guide for chronic myeloproliferative disorders.An intranucleolar body associated with rDNA.Tax impairs DNA replication forks and increases DNA breaks in specific oncogenic genome regionsHydroxyurea-stalled replication forks become progressively inactivated and require two different RAD51-mediated pathways for restart and repair.Redundancy of mammalian Y family DNA polymerases in cellular responses to genomic DNA lesions induced by ultraviolet light
P2860
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P2860
Different roles for nonhomologous end joining and homologous recombination following replication arrest in mammalian cells.
description
2002 nî lūn-bûn
@nan
2002 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2002 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2002年の論文
@ja
2002年論文
@yue
2002年論文
@zh-hant
2002年論文
@zh-hk
2002年論文
@zh-mo
2002年論文
@zh-tw
2002年论文
@wuu
name
Different roles for nonhomolog ...... ion arrest in mammalian cells.
@ast
Different roles for nonhomolog ...... ion arrest in mammalian cells.
@en
Different roles for nonhomolog ...... ion arrest in mammalian cells.
@nl
type
label
Different roles for nonhomolog ...... ion arrest in mammalian cells.
@ast
Different roles for nonhomolog ...... ion arrest in mammalian cells.
@en
Different roles for nonhomolog ...... ion arrest in mammalian cells.
@nl
prefLabel
Different roles for nonhomolog ...... ion arrest in mammalian cells.
@ast
Different roles for nonhomolog ...... ion arrest in mammalian cells.
@en
Different roles for nonhomolog ...... ion arrest in mammalian cells.
@nl
P2093
P2860
P1476
Different roles for nonhomolog ...... ion arrest in mammalian cells.
@en
P2093
Catherine Arnaudeau
Cecilia Lundin
Dag Jenssen
Klaus Erixon
Mark Meuth
Niklas Schultz
P2860
P304
P356
10.1128/MCB.22.16.5869-5878.2002
P407
P577
2002-08-01T00:00:00Z