Intracellular methicillin selection of Listeria monocytogenes mutants unable to replicate in a macrophage cell line
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Common themes in microbial pathogenicity revisitedIsolation of Listeria monocytogenes small-plaque mutants defective for intracellular growth and cell-to-cell spreadDynamics of intracellular bacterial replication at the single cell level.Glutathione provides a source of cysteine essential for intracellular multiplication of Francisella tularensis.Regulation of the prfA transcriptional activator of Listeria monocytogenes: multiple promoter elements contribute to intracellular growth and cell-to-cell spread.Pleiotropic enhancement of bacterial pathogenesis resulting from the constitutive activation of the Listeria monocytogenes regulatory factor PrfA.Cloning of the listeriolysin O gene and development of specific gene probes for Listeria monocytogenes.RNAi screen reveals host cell kinases specifically involved in Listeria monocytogenes spread from cell to cell.Probing the role of protein surface charge in the activation of PrfA, the central regulator of Listeria monocytogenes pathogenesis.Intracellular induction of Listeria monocytogenes actA expressionListeria monocytogenes PrsA2 is required for virulence factor secretion and bacterial viability within the host cell cytosol.Functional analysis of the Listeria monocytogenes secretion chaperone PrsA2 and its multiple contributions to bacterial virulence.A nonvirulent mutant of Listeria monocytogenes does not move intracellularly but still induces polymerization of actin.Nonhemolytic Listeria monocytogenes mutants that are also noninvasive for mammalian cells in culture: evidence for coordinate regulation of virulence.Identification of a peptide-pheromone that enhances Listeria monocytogenes escape from host cell vacuoles.The broad-range phospholipase C and a metalloprotease mediate listeriolysin O-independent escape of Listeria monocytogenes from a primary vacuole in human epithelial cellsA novel C-terminal mutation resulting in constitutive activation of the Listeria monocytogenes central virulence regulatory factor PrfA.Microbial strategies to prevent oxygen-dependent killing by phagocytes.Insertional mutagenesis of Listeria monocytogenes with a novel Tn917 derivative that allows direct cloning of DNA flanking transposon insertions.A differential fluorescence-based genetic screen identifies Listeria monocytogenes determinants required for intracellular replication.A novel prfA mutation that promotes Listeria monocytogenes cytosol entry but reduces bacterial spread and cytotoxicity.Functional impact of mutational activation on the Listeria monocytogenes central virulence regulator PrfA.The posttranslocation chaperone PrsA2 contributes to multiple facets of Listeria monocytogenes pathogenesisA novel mutation within the central Listeria monocytogenes regulator PrfA that results in constitutive expression of virulence gene products.The globally disseminated M1T1 clone of group A Streptococcus evades autophagy for intracellular replication.Intracellular replication is essential for the virulence of Salmonella typhimurium.A Genetic Screen Reveals that Synthesis of 1,4-Dihydroxy-2-Naphthoate (DHNA), but Not Full-Length Menaquinone, Is Required for Listeria monocytogenes Cytosolic Survival.Cytotoxicity of bacterial metabolic products, including listeriolysin O, on leukocyte targets.Listeria monocytogenes cytosolic metabolism promotes replication, survival, and evasion of innate immunity.Secretion Chaperones PrsA2 and HtrA Are Required for Listeria monocytogenes Replication following Intracellular Induction of Virulence Factor Secretion.Human T-cell recognition of Listeria monocytogenes: recognition of listeriolysin O by TcR alpha beta + and TcR gamma delta + T cellsBacterium-host cell interactions at the cellular level: fluorescent labeling of bacteria and analysis of short-term bacterium-phagocyte interaction by flow cytometry.Generating tetracycline-inducible auxotrophy in Escherichia coli and Salmonella enterica serovar Typhimurium by using an insertion element and a hyperactive transposase.Contrasting regulation of macrophage iron homeostasis in response to infection with Listeria monocytogenes depending on localization of bacteria.Th1 dominance in the immune response to live Salmonella typhimurium requires bacterial invasiveness but not persistence.The ethanolamine permease EutH promotes vacuole adaptation of Salmonella enterica and Listeria monocytogenes during macrophage infection.Identification of a second Listeria secA gene associated with protein secretion and the rough phenotype.
P2860
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P2860
Intracellular methicillin selection of Listeria monocytogenes mutants unable to replicate in a macrophage cell line
description
1989 nî lūn-bûn
@nan
1989 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
1989 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
1989年の論文
@ja
1989年論文
@yue
1989年論文
@zh-hant
1989年論文
@zh-hk
1989年論文
@zh-mo
1989年論文
@zh-tw
1989年论文
@wuu
name
Intracellular methicillin sele ...... cate in a macrophage cell line
@ast
Intracellular methicillin sele ...... cate in a macrophage cell line
@en
Intracellular methicillin sele ...... cate in a macrophage cell line
@nl
type
label
Intracellular methicillin sele ...... cate in a macrophage cell line
@ast
Intracellular methicillin sele ...... cate in a macrophage cell line
@en
Intracellular methicillin sele ...... cate in a macrophage cell line
@nl
prefLabel
Intracellular methicillin sele ...... cate in a macrophage cell line
@ast
Intracellular methicillin sele ...... cate in a macrophage cell line
@en
Intracellular methicillin sele ...... cate in a macrophage cell line
@nl
P2093
P2860
P356
P1476
Intracellular methicillin sele ...... cate in a macrophage cell line
@en
P2093
C R Paynton
D A Portnoy
P2860
P304
P356
10.1073/PNAS.86.14.5522
P407
P577
1989-07-01T00:00:00Z