Excitotoxic potential of the cyanotoxin β-methyl-amino-L-alanine (BMAA) in primary human neurons.
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Toxic picoplanktonic cyanobacteria--reviewEquilibrium Dynamics of β-N-Methylamino-L-Alanine (BMAA) and Its Carbamate Adducts at Physiological Conditionsβ-N-Methylamino-L-Alanine Toxicity in PC12: Excitotoxicity vs. Misincorporation.European contribution to the study of ROS: A summary of the findings and prospects for the future from the COST action BM1203 (EU-ROS).Proteome analysis of body fluids for amyotrophic lateral sclerosis biomarker discovery.The effects of the toxic cyanobacterium Limnothrix (strain AC0243) on Bufo marinus larvae.A critical review of the postulated role of the non-essential amino acid, β-N-methylamino-L-alanine, in neurodegenerative disease in humans.β-N-Methylamino-L-alanine (BMAA) perturbs alanine, aspartate and glutamate metabolism pathways in human neuroblastoma cells as determined by metabolic profiling.Gliotoxicity of the cyanotoxin, β-methyl-amino-L-alanine (BMAA).Chemistry and Chemical Equilibrium Dynamics of BMAA and Its Carbamate Adducts.Metabolic profiling of zebrafish (Danio rerio) embryos by NMR spectroscopy reveals multifaceted toxicity of β-methylamino-L-alanine (BMAA).Cytotoxic Effects of Environmental Toxins on Human Glial Cells.Investigating β-N-Methylamino-l-alanine Misincorporation in Human Cell Cultures: A Comparative Study with Known Amino Acid Analogues.Investigation of the interaction of β-methylamino-L-alanine with eukaryotic and prokaryotic proteins.Mechanisms of L-Serine Neuroprotection in vitro Include ER Proteostasis Regulation.Perinatal Exposure to the Cyanotoxin β-N-Méthylamino-L-Alanine (BMAA) Results in Long-Lasting Behavioral Changes in Offspring-Potential Involvement of DNA Damage and Oxidative Stress.L-Serine-Mediated Neuroprotection Includes the Upregulation of the ER Stress Chaperone Protein Disulfide Isomerase (PDI).Mechanisms and Effects Posed by Neurotoxic Products of Cyanobacteria/Microbial Eukaryotes/Dinoflagellates in Algae Blooms: a Review.Detection of the Cyanotoxins L-BMAA Uptake and Accumulation in Primary Neurons and Astrocytes.Assessing the Combined Toxicity of BMAA and Its Isomers 2,4-DAB and AEG In Vitro Using Human Neuroblastoma Cells.Neuroprotective effects of rosmarinic acid on ciguatoxin in primary human neurons.Neurotoxicity of the Cyanotoxin BMAA Through Axonal Degeneration and Intercellular Spreading.
P2860
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P2860
Excitotoxic potential of the cyanotoxin β-methyl-amino-L-alanine (BMAA) in primary human neurons.
description
2012 nî lūn-bûn
@nan
2012 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2012 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
name
Excitotoxic potential of the c ...... MAA) in primary human neurons.
@ast
Excitotoxic potential of the c ...... MAA) in primary human neurons.
@en
Excitotoxic potential of the cyanotoxin β-methyl-amino-L-alanine
@nl
type
label
Excitotoxic potential of the c ...... MAA) in primary human neurons.
@ast
Excitotoxic potential of the c ...... MAA) in primary human neurons.
@en
Excitotoxic potential of the cyanotoxin β-methyl-amino-L-alanine
@nl
prefLabel
Excitotoxic potential of the c ...... MAA) in primary human neurons.
@ast
Excitotoxic potential of the c ...... MAA) in primary human neurons.
@en
Excitotoxic potential of the cyanotoxin β-methyl-amino-L-alanine
@nl
P50
P1433
P1476
Excitotoxic potential of the c ...... BMAA) in primary human neurons
@en
P2093
Alexander S Chiu
Jeffrey H Welch
P304
P356
10.1016/J.TOXICON.2012.07.169
P577
2012-08-09T00:00:00Z