Networked T cell death following macrophage infection by Mycobacterium tuberculosis. - Wikidata - awgldk - TriplyDB

Networked T cell death following macrophage infection by Mycobacterium tuberculosis.

Networked T cell death following macrophage infection by Mycobacterium tuberculosis.

http://www.wikidata.org/entity/Q34295929

about

Effects of chalcone derivatives on players of the immune system Mycobacterium tuberculosis ESAT-6 is a leukocidin causing Ca2+ influx, necrosis and neutrophil extracellular trap formation.Inhibition of nuclear factor-kappa B activation decreases survival of Mycobacterium tuberculosis in human macrophages.Analysis of host responses to Mycobacterium tuberculosis antigens in a multi-site study of subjects with different TB and HIV infection states in sub-Saharan Africa The within host dynamics of Mycobacterium avium ssp. paratuberculosis infection in cattle: where time and place matter Severe Paradoxical Reaction During Treatment of Disseminated Tuberculosis in a Patient With Neutralizing Anti-IFNγ Autoantibodies.Mycobacterial ESX-1 secretion system mediates host cell lysis through bacterium contact-dependent gross membrane disruptions.Mathematical Model of Innate and Adaptive Immunity of Sepsis: A Modeling and Simulation Study of Infectious Disease.Cigarette smoking impairs human pulmonary immunity to Mycobacterium tuberculosis.B-cells with a FasL expressing regulatory phenotype are induced following successful anti-tuberculosis treatment.The effects of exogenous lipid on THP-1 cells: an in vitro model of airway aspiration?Response: Disseminated tuberculosis.

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Networked T cell death following macrophage infection by Mycobacterium tuberculosis.

http://www.wikidata.org/entity/Q34295929

description

2012 nî lūn-bûn

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2012 թուականի Յունիսին հրատարակուած գիտական յօդուած

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2012 թվականի հունիսին հրատարակված գիտական հոդված

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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name

Networked T cell death following macrophage infection by Mycobacterium tuberculosis.

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Networked T cell death following macrophage infection by Mycobacterium tuberculosis.

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Networked T cell death following macrophage infection by Mycobacterium tuberculosis.

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type

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Networked T cell death following macrophage infection by Mycobacterium tuberculosis.

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Networked T cell death following macrophage infection by Mycobacterium tuberculosis.

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Networked T cell death following macrophage infection by Mycobacterium tuberculosis.

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Networked T cell death following macrophage infection by Mycobacterium tuberculosis.

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Networked T cell death following macrophage infection by Mycobacterium tuberculosis.

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Networked T cell death following macrophage infection by Mycobacterium tuberculosis.

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Networked T cell death following macrophage infection by Mycobacterium tuberculosis.

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Anne-Marie McLaughlin

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Elliott Woodward

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Parthiban Nadarajan

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Stephen H-F Macdonald

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Wui-Mei Chew

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P2860

Structure and function of the complex formed by the tuberculosis virulence factors CFP-10 and ESAT-6

Interferon gamma activated macrophages kill mycobacteria by nitric oxide induced apoptosis

Control of M. tuberculosis ESAT-6 secretion and specific T cell recognition by PhoP

Secreted Mycobacterium tuberculosis Rv3654c and Rv3655c proteins participate in the suppression of macrophage apoptosis

Mycobacterium tuberculosis ESX-1 system-secreted protein ESAT-6 but not CFP10 inhibits human T-cell immune responses

Autophagy is a defense mechanism inhibiting BCG and Mycobacterium tuberculosis survival in infected macrophages

Macrophage and T cell dynamics during the development and disintegration of mycobacterial granulomas

Virulent Mycobacterium tuberculosis strains evade apoptosis of infected alveolar macrophages.

Mycobacterium tuberculosis infection induces non-apoptotic cell death of human dendritic cells.

Mycobacterium tuberculosis upregulates microglial matrix metalloproteinase-1 and -3 expression and secretion via NF-kappaB- and Activator Protein-1-dependent monocyte networks.

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Michelle Coleman

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Mycobacterium tuberculosis

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