CSF and plasma amyloid-β temporal profiles and relationships with neurological status and mortality after severe traumatic brain injury
about
Traumatic Axonal Injury: Mechanisms and Translational OpportunitiesCerebrovascular disease in ageing and Alzheimer's diseaseFluid Biomarkers of Traumatic Brain Injury and Intended Context of UseSerum Concentrations of Ubiquitin C-Terminal Hydrolase-L1 and Glial Fibrillary Acidic Protein after Pediatric Traumatic Brain InjuryPathophysiology of Hypoperfusion of the Precuneus in Early Alzheimer's Disease.Active duty service members who sustain a traumatic brain injury have chronically elevated peripheral concentrations of Aβ40 and lower ratios of Aβ42/40.Physical activity predicts reduced plasma β amyloid in the Cardiovascular Health Study.Biomarkers of Traumatic Brain Injury: Temporal Changes in Body FluidsDetection of Plasma Biomarkers Using Immunomagnetic Reduction: A Promising Method for the Early Diagnosis of Alzheimer's Disease.sAβPPα is a Potent Endogenous Inhibitor of BACE1.Analytical performance of reagent for assaying tau protein in human plasma and feasibility study screening neurodegenerative diseasesCurrent and Emerging Technologies for Probing Molecular Signatures of Traumatic Brain Injury.Rapid amyloid-β oligomer and protofibril accumulation in traumatic brain injury.Inflammation Relates to Chronic Behavioral and Neurological Symptoms in Military Personnel with Traumatic Brain Injuries.Current Opportunities for Clinical Monitoring of Axonal Pathology in Traumatic Brain Injury.Elevated serum miR-93, miR-191, and miR-499 are noninvasive biomarkers for the presence and progression of traumatic brain injury.Moderate blast exposure results in increased IL-6 and TNFα in peripheral blood.Blood biomarkers indicate mild neuroaxonal injury and increased amyloid β production after transient hypoxia during breath-hold diving.Cerebral Hypoperfusion and the Energy Deficit in Alzheimer's Disease.Increases of Plasma Levels of Glial Fibrillary Acidic Protein, Tau, and Amyloid β up to 90 Days after Traumatic Brain Injury.MicroRNA Signature of Traumatic Brain Injury: From the Biomarker Discovery to the Point-of-Care.Finding effective biomarkers for pediatric traumatic brain injuryIntegration of Biomarkers Into a Signature Profile of Persistent Traumatic Brain Injury Involving Autoimmune Processes Following Water Hammer Injury From Repetitive Head Impacts
P2860
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P2860
CSF and plasma amyloid-β temporal profiles and relationships with neurological status and mortality after severe traumatic brain injury
description
2014 nî lūn-bûn
@nan
2014 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
name
CSF and plasma amyloid-β tempo ...... severe traumatic brain injury
@ast
CSF and plasma amyloid-β tempo ...... severe traumatic brain injury
@en
CSF and plasma amyloid-β tempo ...... severe traumatic brain injury
@nl
type
label
CSF and plasma amyloid-β tempo ...... severe traumatic brain injury
@ast
CSF and plasma amyloid-β tempo ...... severe traumatic brain injury
@en
CSF and plasma amyloid-β tempo ...... severe traumatic brain injury
@nl
prefLabel
CSF and plasma amyloid-β tempo ...... severe traumatic brain injury
@ast
CSF and plasma amyloid-β tempo ...... severe traumatic brain injury
@en
CSF and plasma amyloid-β tempo ...... severe traumatic brain injury
@nl
P2093
P2860
P356
P1433
P1476
CSF and plasma amyloid-β tempo ...... severe traumatic brain injury
@en
P2093
Andras Buki
Andreas Jeromin
David Hanlon
David Wilson
Gail Provuncher
Jeff Randall
P2860
P2888
P356
10.1038/SREP06446
P407
P577
2014-10-10T00:00:00Z
P5875
P6179
1051734988