Troglitazone attenuates TGF-β1-induced EMT in alveolar epithelial cells via a PPARγ-independent mechanism. - Wikidata - awgldk - TriplyDB

Troglitazone attenuates TGF-β1-induced EMT in alveolar epithelial cells via a PPARγ-independent mechanism.

Troglitazone attenuates TGF-β1-induced EMT in alveolar epithelial cells via a PPARγ-independent mechanism.

http://www.wikidata.org/entity/Q34321160

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Mechanisms of TGFβ-Induced Epithelial-Mesenchymal Transition Chemical regulators of epithelial plasticity reveal a nuclear receptor pathway controlling myofibroblast differentiation Anti-fibrogenic effect of PPAR-γ agonists in human intestinal myofibroblasts.Crosstalk between TGF-β1 and complement activation augments epithelial injury in pulmonary fibrosis.Adenosine A2A receptor: a target for regulating renal interstitial fibrosis in obstructive nephropathy.Inhibitory effects of PPARγ ligands on TGF-β1-induced corneal myofibroblast transformation.Inhibitory effects of PPARγ ligands on TGF-β1-induced CTGF expression in cat corneal fibroblasts.Inhibition of epithelial-to-mesenchymal transition and pulmonary fibrosis by methacycline Biomechanics of TGFβ-induced epithelial-mesenchymal transition: implications for fibrosis and cancer Inhibition of Wnt/β-catenin signaling suppresses bleomycin-induced pulmonary fibrosis by attenuating the expression of TGF-β1 and FGF-2 Epithelial-mesenchymal transitions in bronchopulmonary dysplasia of newborn rats.Runx3 is a key modulator during the epithelial-mesenchymal transition of alveolar type II cells in animal models of BPD.Thalidomide Inhibits TGF-β1-induced Epithelial to Mesenchymal Transition in Alveolar Epithelial Cells via Smad-Dependent and Smad-Independent Signaling Pathways.Warburg effect hypothesis in autism Spectrum disorders.Trichostatin A inhibits radiation-induced epithelial-to-mesenchymal transition in the alveolar epithelial cells.Interactions between TGF-β1, canonical WNT/β-catenin pathway and PPAR γ in radiation-induced fibrosis.Thermodynamic Aspects and Reprogramming Cellular Energy Metabolism during the Fibrosis Process.Hyperoxia induces paracellular leak and alters claudin expression by neonatal alveolar epithelial cells.

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Troglitazone attenuates TGF-β1-induced EMT in alveolar epithelial cells via a PPARγ-independent mechanism.

http://www.wikidata.org/entity/Q34321160

description

2012 nî lūn-bûn

@nan

2012 թուականի Յունիսին հրատարակուած գիտական յօդուած

@hyw

2012 թվականի հունիսին հրատարակված գիտական հոդված

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2012年の論文

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年學術文章

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name

Troglitazone attenuates TGF-β1 ...... a PPARγ-independent mechanism.

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Troglitazone attenuates TGF-β1 ...... a PPARγ-independent mechanism.

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Troglitazone attenuates TGF-β1 ...... a PPARγ-independent mechanism.

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type

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Troglitazone attenuates TGF-β1 ...... a PPARγ-independent mechanism.

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Troglitazone attenuates TGF-β1 ...... a PPARγ-independent mechanism.

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Troglitazone attenuates TGF-β1 ...... a PPARγ-independent mechanism.

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prefLabel

Troglitazone attenuates TGF-β1 ...... a PPARγ-independent mechanism.

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Troglitazone attenuates TGF-β1 ...... a PPARγ-independent mechanism.

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Troglitazone attenuates TGF-β1 ...... a PPARγ-independent mechanism.

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JOURNAL.PONE.0038827

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Troglitazone attenuates TGF-β1 ...... a PPARγ-independent mechanism

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Beiyun Zhou

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Edward D Crandall

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Jiao Luo

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Kwang-Jin Kim

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Lucas DeMaio

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Manda Sai Krishnaveni

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Mihaela Ivan

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Stephen T Buckley

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Vipul Patel

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Yixin Liu

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Alveolar epithelial cell mesenchymal transition develops in vivo during pulmonary fibrosis and is regulated by the extracellular matrix

Interactions between β-catenin and transforming growth factor-β signaling pathways mediate epithelial-mesenchymal transition and are dependent on the transcriptional co-activator cAMP-response element-binding protein (CREB)-binding protein (CBP).

Peroxisome proliferator-activated receptor-gamma activation inhibits tumor metastasis by antagonizing Smad3-mediated epithelial-mesenchymal transition

Upregulation of PTEN involved in rosiglitazone-induced apoptosis in human hepatocellular carcinoma cells.

Effects of PPAR gamma ligands on TGF-beta1-induced epithelial-mesenchymal transition in alveolar epithelial cells.

PPAR-γ ligands repress TGFβ-induced myofibroblast differentiation by targeting the PI3K/Akt pathway: implications for therapy of fibrosis.

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Carsten Ehrhardt

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