Deceleration of fusion-fission cycles improves mitochondrial quality control during aging. - Wikidata - awgldk - TriplyDB

Deceleration of fusion-fission cycles improves mitochondrial quality control during aging.

Deceleration of fusion-fission cycles improves mitochondrial quality control during aging.

http://www.wikidata.org/entity/Q34325475

about

Integration of 'omics' data in aging research: from biomarkers to systems biology Mitochondrial Dysfunction Contributes to the Pathogenesis of Alzheimer's Disease What is the function of mitochondrial networks? A theoretical assessment of hypotheses and proposal for future research Agent-Based Modeling of Mitochondria Links Sub-Cellular Dynamics to Cellular Homeostasis and Heterogeneity Control of mitochondrial integrity in ageing and disease Emergence of the mitochondrial reticulum from fission and fusion dynamics.Mitochondria, energetics, epigenetics, and cellular responses to stress.Mitochondrial network morphology: building an integrative, geometrical view Optimal dynamics for quality control in spatially distributed mitochondrial networks.Mathematical modeling of the role of mitochondrial fusion and fission in mitochondrial DNA maintenance.Context-Dependent Role of Mitochondrial Fusion-Fission in Clonal Expansion of mtDNA Mutations Quality Saving Mechanisms of Mitochondria during Aging in a Fully Time-Dependent Computational Biophysical Model.Age-Related and Heteroplasmy-Related Variation in Human mtDNA Copy Number Evolution of Cell-to-Cell Variability in Stochastic, Controlled, Heteroplasmic mtDNA Populations.Inheritance of the fittest mitochondria in yeast.Identification of autophagy as a longevity-assurance mechanism in the aging model Podospora anserina.Mitochondrial dysfunction in psychiatric and neurological diseases: cause(s), consequence(s), and implications of antioxidant therapy.The systems biology of mitochondrial fission and fusion and implications for disease and aging.Aging and computational systems biology.Modelling the molecular mechanisms of aging.Fragmentation of the mitochondrial network in skin in vivo.Simultaneous impairment of mitochondrial fission and fusion reduces mitophagy and shortens replicative lifespan.Quality control of mitochondria during aging: is there a good and a bad side of mitochondrial dynamics?Pro-Inflammatory CXCR3 Impairs Mitochondrial Function in Experimental Non-Alcoholic Steatohepatitis.Simulation of Cellular Energy Restriction in Quiescence (ERiQ)-A Theoretical Model for Aging.Ectromelia Virus Affects Mitochondrial Network Morphology, Distribution, and Physiology in Murine Fibroblasts and Macrophage Cell Line.Mitochondrial Function and Dynamics Imaged In Vivo

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P2860

Deceleration of fusion-fission cycles improves mitochondrial quality control during aging.

http://www.wikidata.org/entity/Q34325475

description

2012 nî lūn-bûn

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2012 թուականի Յունիսին հրատարակուած գիտական յօդուած

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2012 թվականի հունիսին հրատարակված գիտական հոդված

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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name

Deceleration of fusion-fission ...... quality control during aging.

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Deceleration of fusion-fission ...... quality control during aging.

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Deceleration of fusion-fission ...... quality control during aging.

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type

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Deceleration of fusion-fission ...... quality control during aging.

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Deceleration of fusion-fission ...... quality control during aging.

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Deceleration of fusion-fission ...... quality control during aging.

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Deceleration of fusion-fission ...... quality control during aging.

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Deceleration of fusion-fission ...... quality control during aging.

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Deceleration of fusion-fission ...... quality control during aging.

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JOURNAL.PCBI.1002576

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PLOS Computational Biology

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Deceleration of fusion-fission ...... quality control during aging.

@en

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Andreas S Reichert

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Heinz D Osiewacz

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Michael Meyer-Hermann

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P2860

OPA1, encoding a dynamin-related GTPase, is mutated in autosomal dominant optic atrophy linked to chromosome 3q28

Premature ageing in mice expressing defective mitochondrial DNA polymerase

Mitochondrial fusion in human cells is efficient, requires the inner membrane potential, and is mediated by mitofusins.

Is the oxidative stress theory of aging dead?

Fission and selective fusion govern mitochondrial segregation and elimination by autophagy

Selective degradation of mitochondria by mitophagy

Oxidants, oxidative stress and the biology of ageing

Nuclear gene OPA1, encoding a mitochondrial dynamin-related protein, is mutated in dominant optic atrophy

Regulation of mitochondrial morphology through proteolytic cleavage of OPA1

SLP-2 is required for stress-induced mitochondrial hyperfusion

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10.1371/JOURNAL.PCBI.1002576

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6

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Marc Thilo Figge

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2012-06-28T00:00:00Z

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228116308

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quality control

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3386171

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