Atrial fibrillation and sinus node dysfunction in human ankyrin-B syndrome: a computational analysis. - Wikidata - awgldk - TriplyDB

Atrial fibrillation and sinus node dysfunction in human ankyrin-B syndrome: a computational analysis.

Atrial fibrillation and sinus node dysfunction in human ankyrin-B syndrome: a computational analysis.

http://www.wikidata.org/entity/Q34329504

about

Fibrosis: a structural modulator of sinoatrial node physiology and dysfunction Sinus Node Dysfunction and Atrial Fibrillation: A Reversible Phenomenon?Human sinoatrial node structure: 3D microanatomy of sinoatrial conduction pathways.Role of sinoatrial node architecture in maintaining a balanced source-sink relationship and synchronous cardiac pacemaking.Functional role of voltage gated Ca(2+) channels in heart automaticity.Cycle length restitution in sinoatrial node cells: a theory for understanding spontaneous action potential dynamics.Function and dysfunction of human sinoatrial node Malignant systemic hypertension, encephalopathy and bradycardia following splenectomy for hereditary spherocytosis.Modern perspectives on numerical modeling of cardiac pacemaker cell Two-Pore K+ Channel TREK-1 Regulates Sinoatrial Node Membrane Excitability.Patient-derived models link re-entrant driver localization in atrial fibrillation to fibrosis spatial pattern.Mechanistic inquiry into the role of tissue remodeling in fibrotic lesions in human atrial fibrillation.Towards personalized computational modelling of the fibrotic substrate for atrial arrhythmia.LongQt: A cardiac electrophysiology simulation platform.Atrial structure, function and arrhythmogenesis in aged and frail mice.Ca2+/calmodulin-dependent kinase II-dependent regulation of atrial myocyte late Na+ current, Ca2+ cycling, and excitability: a mathematical modeling study.Mechanism underlying impaired cardiac pacemaking rhythm during ischemia: A simulation study.The evolving role of ankyrin-B in cardiovascular disease.Impaired sinoatrial node function and increased susceptibility to atrial fibrillation in mice lacking natriuretic peptide receptor C.

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P2860

Atrial fibrillation and sinus node dysfunction in human ankyrin-B syndrome: a computational analysis.

http://www.wikidata.org/entity/Q34329504

description

2013 nî lūn-bûn

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2013 թուականի Փետրուարին հրատարակուած գիտական յօդուած

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2013年の論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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name

Atrial fibrillation and sinus ...... ome: a computational analysis.

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Atrial fibrillation and sinus ...... ome: a computational analysis.

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Atrial fibrillation and sinus ...... ome: a computational analysis.

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Atrial fibrillation and sinus ...... ome: a computational analysis.

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Atrial fibrillation and sinus ...... ome: a computational analysis.

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Atrial fibrillation and sinus ...... ome: a computational analysis.

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Atrial fibrillation and sinus ...... ome: a computational analysis.

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Atrial fibrillation and sinus ...... ome: a computational analysis.

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Atrial fibrillation and sinus ...... ome: a computational analysis.

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AJPHEART.00734.2012

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American Journal of Physiology - Heart and Circulatory Physiology

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Atrial fibrillation and sinus ...... ome: a computational analysis.

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Colleen C Mitchell

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Keyan Zarei

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Mark E Anderson

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Patric Glynn

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Peter J Mohler

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Roseanne M Wolf

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Seyed Hashemi

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Thomas J Hund

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P2860

Defining the cellular phenotype of "ankyrin-B syndrome" variants: human ANK2 variants associated with clinical phenotypes display a spectrum of activities in cardiomyocytes

Ankyrin-B mutation causes type 4 long-QT cardiac arrhythmia and sudden cardiac death

A cardiac arrhythmia syndrome caused by loss of ankyrin-B function

Ankyrin-B coordinates the Na/K ATPase, Na/Ca exchanger, and InsP3 receptor in a cardiac T-tubule/SR microdomain.

Dysfunction in ankyrin-B-dependent ion channel and transporter targeting causes human sinus node disease

Defects in ankyrin-based membrane protein targeting pathways underlie atrial fibrillation

Targeting and stability of Na/Ca exchanger 1 in cardiomyocytes requires direct interaction with the membrane adaptor ankyrin-B

Mapping of a gene for long QT syndrome to chromosome 4q25-27

Effects of electrical and structural remodeling on atrial fibrillation maintenance: a simulation study

Activation and inactivation of a non-selective cation conductance by local mechanical deformation of acutely isolated cardiac fibroblasts.

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atrial fibrillation

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