Efficient, complete deletion of synaptic proteins using CRISPR.
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CRISPR/Cas9: Implications for Modeling and Therapy of Neurodegenerative DiseasesGenome Engineering with TALE and CRISPR Systems in NeuroscienceBrain tumor modeling using the CRISPR/Cas9 system: state of the art and view to the futureApplications of CRISPR-Cas systems in neuroscienceStrategies for precision modulation of gene expression by epigenome editing: an overviewSex steroid hormones matter for learning and memory: estrogenic regulation of hippocampal function in male and female rodentsCRISPR/Cas9: a powerful genetic engineering tool for establishing large animal models of neurodegenerative diseasesAssembly of hair bundles, an amazing problem for cell biologyA Toolkit for Orthogonal and in vivo Optical Manipulation of Ionotropic Glutamate Receptors.Editing the Neuronal Genome: a CRISPR View of Chromatin Regulation in Neuronal Development, Function, and PlasticityApplications of CRISPR/Cas9 for Gene Editing in Hereditary Movement DisordersN-terminal Huntingtin Knock-In Mice: Implications of Removing the N-terminal Region of Huntingtin for Therapy.An E3-ligase-based method for ablating inhibitory synapsesDeciphering the pathogenic consequences of chromosomal aberrations in human genetic disease.Tracking and transforming neocortical progenitors by CRISPR/Cas9 gene targeting and piggyBac transposase lineage labeling.α/β-Hydrolase domain-containing 6 (ABHD6) negatively regulates the surface delivery and synaptic function of AMPA receptors.High-Throughput, High-Resolution Mapping of Protein Localization in Mammalian Brain by In Vivo Genome Editing.Association of morphine-induced analgesic tolerance with changes in gene expression of GluN1 and MOR1 in rat spinal cord and midbrain.Surgical extraction of human dorsal root ganglia from organ donors and preparation of primary sensory neuron cultures.Genome Editing Reveals Glioblastoma Addiction to MicroRNA-10b.The application of CRISPR technology to high content screening in primary neurons.CrisprGE: a central hub of CRISPR/Cas-based genome editing.Desmocollin-2 alone forms functional desmosomal plaques, with the plaque formation requiring the juxtamembrane region and plakophilins.Use of CRISPR/Cas9 to model brain diseases.CRISPR/Cas9 system-mediated impairment of synaptobrevin/VAMP function in postmitotic hippocampal neurons.The schizophrenia- and autism-associated gene, transcription factor 4 regulates the columnar distribution of layer 2/3 prefrontal pyramidal neurons in an activity-dependent manner.Immunocytochemistry and fluorescence imaging efficiently identify individual neurons with CRISPR/Cas9-mediated gene disruption in primary cortical cultures.Identification of an elaborate complex mediating postsynaptic inhibition.Rapid and efficient CRISPR/Cas9 gene inactivation in human neurons during human pluripotent stem cell differentiation and direct reprogramming.Somatostatin and parvalbumin inhibitory synapses onto hippocampal pyramidal neurons are regulated by distinct mechanisms.Critical roles of αII spectrin in brain development and epileptic encephalopathy.Hepatic encephalopathy induces site-specific changes in gene expression of GluN1 subunit of NMDA receptor in rat brain.Deconstructing behavioral neuropharmacology with cellular specificity.CRISPR-Cas9-mediated genome editing in one blastomere of two-cell embryos reveals a novel Tet3 function in regulating neocortical development.[Application of Cas9/CRISPR to the study of synaptic function].Applications of CRISPR/Cas9 in the Mammalian Central Nervous System.Neuroepigenetic Editing.The CaMKII/NMDA receptor complex controls hippocampal synaptic transmission by kinase-dependent and independent mechanisms.Steroids and Brain, a Rising Bio-Medical Domain: a Perspective.Analgesia linked to Nav1.7 loss of function requires µ- and δ-opioid receptors
P2860
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P2860
Efficient, complete deletion of synaptic proteins using CRISPR.
description
2014 nî lūn-bûn
@nan
2014 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2014年の論文
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2014年学术文章
@wuu
2014年学术文章
@zh-cn
2014年学术文章
@zh-hans
2014年学术文章
@zh-my
2014年学术文章
@zh-sg
2014年學術文章
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name
Efficient, complete deletion of synaptic proteins using CRISPR.
@ast
Efficient, complete deletion of synaptic proteins using CRISPR.
@en
Efficient, complete deletion of synaptic proteins using CRISPR.
@nl
type
label
Efficient, complete deletion of synaptic proteins using CRISPR.
@ast
Efficient, complete deletion of synaptic proteins using CRISPR.
@en
Efficient, complete deletion of synaptic proteins using CRISPR.
@nl
prefLabel
Efficient, complete deletion of synaptic proteins using CRISPR.
@ast
Efficient, complete deletion of synaptic proteins using CRISPR.
@en
Efficient, complete deletion of synaptic proteins using CRISPR.
@nl
P2093
P2860
P1433
P1476
Efficient, complete deletion of synaptic proteins using CRISPR.
@en
P2093
Robert H Edwards
Roger A Nicoll
Salvatore Incontro
P2860
P304
P356
10.1016/J.NEURON.2014.07.043
P407
P577
2014-08-21T00:00:00Z