GDNF rescues hyperglycemia-induced diabetic enteric neuropathy through activation of the PI3K/Akt pathway - Wikidata - awgldk - TriplyDB

GDNF rescues hyperglycemia-induced diabetic enteric neuropathy through activation of the PI3K/Akt pathway

GDNF rescues hyperglycemia-induced diabetic enteric neuropathy through activation of the PI3K/Akt pathway

http://www.wikidata.org/entity/Q34331361

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FoxO Transcription Factors and Regenerative Pathways in Diabetes Mellitus Diabetic gastrointestinal motility disorders and the role of enteric nervous system: current status and future directions Targeted deletion of neuropeptide Y (NPY) modulates experimental colitis Dual angiogenic and neurotrophic effects of bone marrow-derived endothelial progenitor cells on diabetic neuropathy.Oxidative stress: Biomarkers and novel therapeutic pathways.Organotypic specificity of key RET adaptor-docking sites in the pathogenesis of neurocristopathies and renal malformations in mice.Preconditioning of adipose tissue-derived mesenchymal stem cells with deferoxamine increases the production of pro-angiogenic, neuroprotective and anti-inflammatory factors: Potential application in the treatment of diabetic neuropathy Colonic motor dysfunction in human diabetes is associated with enteric neuronal loss and increased oxidative stress.New strategies for Alzheimer's disease and cognitive impairment.Enteric nervous system abnormalities are present in human necrotizing enterocolitis: potential neurotransplantation therapy Diabetes mellitus: channeling care through cellular discovery The FOX transcription factors regulate vascular pathology, diabetes and Tregs Cellular pathogenesis of diabetic gastroenteropathy.The "O" class: crafting clinical care with FoxO transcription factors.Baicalin promotes the viability of Schwann cells in vitro by regulating neurotrophic factors Upper gastrointestinal function and glycemic control in diabetes mellitus.Wnt1 neuroprotection translates into improved neurological function during oxidant stress and cerebral ischemia through AKT1 and mitochondrial apoptotic pathways.Nerve injury induces glial cell line-derived neurotrophic factor (GDNF) expression in Schwann cells through purinergic signaling and the PKC-PKD pathway 2, 3, 5, 4'-Tetrahydroxystilbene-2-O-beta-D-glucoside improves gastrointestinal motility disorders in STZ-induced diabetic mice.Novel avenues of drug discovery and biomarkers for diabetes mellitus.Cellular changes in diabetic and idiopathic gastroparesis.Relationship between glycemic control and gastric emptying in poorly controlled type 2 diabetes.Impairment of nitrergic system and delayed gastric emptying in low density lipoprotein receptor deficient female mice.Smooth Muscle Hgs Deficiency Leads to Impaired Esophageal Motility Altered calcium signaling in colonic smooth muscle of type 1 diabetic mice.Diabetes-related alterations in the enteric nervous system and its microenvironment.Hepatic insulin gene therapy prevents diabetic enteropathy in STZ-treated CD-1 mice.High ω3-polyunsaturated fatty acids in fat-1 mice prevent streptozotocin-induced Purkinje cell degeneration through BDNF-mediated autophagy Gut microbial products regulate murine gastrointestinal motility via Toll-like receptor 4 signaling Diabetic peripheral neuropathy: should a chaperone accompany our therapeutic approach?Gastrointestinal motility and glycemic control in diabetes: the chicken and the egg revisited?Glial-derived neurotrophic factor modulates enteric neuronal survival and proliferation through neuropeptide Y.Insulin-like growth factor-I stimulates Shc-dependent phosphatidylinositol 3-kinase activation via Grb2-associated p85 in vascular smooth muscle cells Triple play: promoting neurovascular longevity with nicotinamide, WNT, and erythropoietin in diabetes mellitus.Aloperine protects mice against ischemia reperfusion (IR)-induced renal injury by regulating PI3K/AKT/mTOR signaling and AP-1 activity.The therapeutic potential of gene transfer for the treatment of peripheral neuropathies."Sly as a FOXO": new paths with Forkhead signaling in the brain Erythropoietin and oxidative stress.OutFOXOing disease and disability: the therapeutic potential of targeting FoxO proteins Diabetes and the enteric nervous system

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GDNF rescues hyperglycemia-induced diabetic enteric neuropathy through activation of the PI3K/Akt pathway

http://www.wikidata.org/entity/Q34331361

description

2006 nî lūn-bûn

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2006 թուականի Փետրուարին հրատարակուած գիտական յօդուած

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2006 թվականի փետրվարին հրատարակված գիտական հոդված

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2006年の論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年论文

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name

GDNF rescues hyperglycemia-ind ...... vation of the PI3K/Akt pathway

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GDNF rescues hyperglycemia-ind ...... vation of the PI3K/Akt pathway

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GDNF rescues hyperglycemia-ind ...... vation of the PI3K/Akt pathway

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type

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GDNF rescues hyperglycemia-ind ...... vation of the PI3K/Akt pathway

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GDNF rescues hyperglycemia-ind ...... vation of the PI3K/Akt pathway

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GDNF rescues hyperglycemia-ind ...... vation of the PI3K/Akt pathway

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GDNF rescues hyperglycemia-ind ...... vation of the PI3K/Akt pathway

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GDNF rescues hyperglycemia-ind ...... vation of the PI3K/Akt pathway

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GDNF rescues hyperglycemia-ind ...... vation of the PI3K/Akt pathway

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Journal of Clinical Investigation

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GDNF rescues hyperglycemia-ind ...... vation of the PI3K/Akt pathway

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P2093

Alexander Parsadanian

http://www.w3.org/2001/XMLSchema#string

Chetan Gondha

http://www.w3.org/2001/XMLSchema#string

Mallappa Anitha

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Roy Sutliff

http://www.w3.org/2001/XMLSchema#string

Shanthi Srinivasan

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Shanthi V Sitaraman

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Simon Mwangi

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P2860

Neural cells in the esophagus respond to glial cell line-derived neurotrophic factor and neurturin, and are RET-dependent

Akt, a pleckstrin homology domain containing kinase, is activated primarily by phosphorylation

The effect of intensive treatment of diabetes on the development and progression of long-term complications in insulin-dependent diabetes mellitus. The Diabetes Control and Complications Trial Research Group

Gene targeting reveals a critical role for neurturin in the development and maintenance of enteric, sensory, and parasympathetic neurons

Neurturin shares receptors and signal transduction pathways with glial cell line-derived neurotrophic factor in sympathetic neurons

Renal agenesis and the absence of enteric neurons in mice lacking GDNF

Retinopathy and nephropathy in patients with type 1 diabetes four years after a trial of intensive therapy.

GDNF rescues nonpeptidergic unmyelinated primary afferents in streptozotocin-treated diabetic mice.

Small-bowel motility.

The GDNF/RET signaling pathway and human diseases.

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344-356

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10.1172/JCI26295

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P433

2

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116

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2006-02-01T00:00:00Z

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7321241

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16453021

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1359053

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