Staphylococcus aureus protein A plays a critical role in mediating bone destruction and bone loss in osteomyelitis. - Wikidata - awgldk - TriplyDB

Staphylococcus aureus protein A plays a critical role in mediating bone destruction and bone loss in osteomyelitis.

Staphylococcus aureus protein A plays a critical role in mediating bone destruction and bone loss in osteomyelitis.

http://www.wikidata.org/entity/Q34336558

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Staphylococcus aureus vs. Osteoblast: Relationship and Consequences in Osteomyelitis PSMs of hypervirulent Staphylococcus aureus act as intracellular toxins that kill infected osteoblasts.Osteoclast profile of medication-related osteonecrosis of the jaw secondary to bisphosphonate therapy: a comparison with osteoradionecrosis and osteomyelitis.Staphylococcus aureus biofilms decrease osteoblast viability, inhibits osteogenic differentiation, and increases bone resorption in vitro Surgical Debridement Is Superior to Sole Antibiotic Therapy in a Novel Murine Posttraumatic Osteomyelitis Model.Toll-Like Receptor 2 Stimulation of Osteoblasts Mediates Staphylococcus Aureus Induced Bone Resorption and Osteoclastogenesis through Enhanced RANKL.Metabolic activity, urease production, antibiotic resistance and virulence in dual species biofilms of Staphylococcus epidermidis and Staphylococcus aureus.Impact of sarA and Phenol-Soluble Modulins on the Pathogenesis of Osteomyelitis in Diverse Clinical Isolates of Staphylococcus aureus.Staphylococcus aureus-dependent septic arthritis in murine knee joints: local immune response and beneficial effects of vaccination.The potential role of increasing the release of mouse β- defensin-14 in the treatment of osteomyelitis in mice: a primary study.Apoptosis-associated uncoupling of bone formation and resorption in osteomyelitis.The macrophage inflammatory proteins MIP1α (CCL3) and MIP2α (CXCL2) in implant-associated osteomyelitis: linking inflammation to bone degradation.In vitro and in vivo osteogenic activity of the novel vancomycin-loaded bone-like hydroxyapatite/poly(amino acid) scaffold.Primary acquired spondylodiscitis shows a more severe course than spondylodiscitis following spine surgery: a single-center retrospective study of 159 cases.The osteogenic potential of the marine-derived multi-mineral formula aquamin is enhanced by the presence of vitamin D.Inhibition of fracture healing in the presence of contamination by Staphylococcus aureus: Effects of growth state and immune response.The role of biofilms in persistent infections and factors involved in ica-independent biofilm development and gene regulation in Staphylococcus aureus.Atypical Osteomyelitis Caused by Mycobacterium chelonae-A Multimodal Imaging Approach.Diminished bone regeneration after debridement of posttraumatic osteomyelitis is accompanied by altered cytokine levels, elevated B cell activity, and increased osteoclast activity.Enterococcus faecalis attenuates osteogenesis through activation of p38 and ERK1/2 pathways in MC3T3-E1 cells.Staphylococcal protein A promotes osteoclastogenesis through MAPK signaling during bone infection.Staphylococcus aureus-mediated blood-brain barrier injury: an in vitro human brain microvascular endothelial cell model.Staphylococcus aureus peptidoglycan promotes osteoclastogenesis via TLR2-mediated activation of the NF-κB/NFATc1 signaling pathway.Short-term administration of small molecule phenamil induced a protracted osteogenic effect on osteoblast-like MC3T3-E1 cells.Bacterial modulators of bone remodeling in the periodontal pocket.Dual neutralization of TNFR-2 and MMP-2 regulates the severity of S. aureus induced septic arthritis correlating alteration in the level of interferon gamma and interleukin-10 in terms of TNFR2 blocking.Antibody-Based Agents in the Management of Antibiotic-Resistant Staphylococcus aureus Diseases.Staphylococcal Osteomyelitis: Disease Progression, Treatment Challenges, and Future Directions.Staphylococcus aureus protein A causes osteoblasts to hyper-mineralise in a 3D extra-cellular matrix environment.Cancer Metastases to Bone: Concepts, Mechanisms, and Interactions with Bone Osteoblasts.Raman Spectroscopy detects changes in Bone Mineral Quality and Collagen Cross-linkage in Staphylococcus Infected Human Bone.Innate Immunity to : Evolving Paradigms in Soft Tissue and Invasive Infections A potential key role for alpha-haemolysin of in mediating chondrocyte death in septic arthritis

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Staphylococcus aureus protein A plays a critical role in mediating bone destruction and bone loss in osteomyelitis.

http://www.wikidata.org/entity/Q34336558

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2012 nî lūn-bûn

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2012 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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Staphylococcus aureus protein ...... nd bone loss in osteomyelitis.

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Staphylococcus aureus protein ...... nd bone loss in osteomyelitis.

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Staphylococcus aureus protein ...... nd bone loss in osteomyelitis.

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Amro Widaa

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Steven W Kerrigan

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Tania Claro

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Timothy J Foster

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P2860

Staphylococcus aureus fibronectin binding proteins are essential for internalization by osteoblasts but do not account for differences in intracellular levels of bacteria

Crystal structure of a Staphylococcus aureus protein A domain complexed with the Fab fragment of a human IgM antibody: structural basis for recognition of B-cell receptors and superantigen activity

Staphylococcus aureus protein A induces airway epithelial inflammatory responses by activating TNFR1

Effect of parathyroid hormone on release of interleukin 1 and interleukin 6 from cultured mouse osteoblastic cells

Osteoclast differentiation and activation

Genetic regulation of osteoclast development and function

Staphylococcus aureus adhesion to bone matrix and bone-associated biomaterials.

Staphylococcus aureus protein A binds to osteoblasts and triggers signals that weaken bone in osteomyelitis.

Apoptosis induced by Staphylococcus aureus in epithelial cells utilizes a mechanism involving caspases 8 and 3.

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