Mechanisms of glioma formation: iterative perivascular glioma growth and invasion leads to tumor progression, VEGF-independent vascularization, and resistance to antiangiogenic therapy.
about
New perspectives in glioblastoma antiangiogenic therapySingle vs. combination immunotherapeutic strategies for glioma.Coibamide A, a natural lariat depsipeptide, inhibits VEGFA/VEGFR2 expression and suppresses tumor growth in glioblastoma xenografts.Effects of anti-angiogenesis on glioblastoma growth and migration: model to clinical predictions.Computational Trials: Unraveling Motility Phenotypes, Progression Patterns, and Treatment Options for Glioblastoma Multiforme.VB-111: a novel anti-vascular therapeutic for glioblastoma multiformeVEGF silencing inhibits human osteosarcoma angiogenesis and promotes cell apoptosis via PI3K/AKT signaling pathway.Single Cell Mathematical Model Successfully Replicates Key Features of GBM: Go-Or-Grow Is Not Necessary.Gene Therapy for the Treatment of Neurological Disorders: Central Nervous System Neoplasms.Isolation and Flow Cytometric Analysis of Glioma-infiltrating Peripheral Blood Mononuclear CellsDecrease of VEGF-A in myeloid cells attenuates glioma progression and prolongs survival in an experimental glioma modelNatural killer cells require monocytic Gr-1(+)/CD11b(+) myeloid cells to eradicate orthotopically engrafted glioma cellsSoluble Tie2 overrides the heightened invasion induced by anti-angiogenesis therapies in gliomas.Glutamate-Mediated Blood-Brain Barrier Opening: Implications for Neuroprotection and Drug Delivery.The brain-penetrating CXCR4 antagonist, PRX177561, increases the antitumor effects of bevacizumab and sunitinib in preclinical models of human glioblastoma.VEGF Promoter Polymorphism Confers an Increased Risk of Pulmonary Arterial Hypertension in a Chinese PopulationCXCR4 increases in-vivo glioma perivascular invasion, and reduces radiation induced apoptosis: A genetic knockdown study.Transfer of functional microRNAs between glioblastoma and microvascular endothelial cells through gap junctions.A role for ion channels in perivascular glioma invasion.RhoA/mDia-1/profilin-1 signaling targets microvascular endothelial dysfunction in diabetic retinopathy.Bevacizumab and radiotherapy for the treatment of glioblastoma: brothers in arms or unholy alliance?Strategies to enhance the distribution of nanotherapeutics in the brain.Shikonin Inhibits the Migration and Invasion of Human Glioblastoma Cells by Targeting Phosphorylated β-Catenin and Phosphorylated PI3K/Akt: A Potential Mechanism for the Anti-Glioma Efficacy of a Traditional Chinese Herbal MedicinePlexin-B2 promotes invasive growth of malignant gliomaThe Microvascular Gap Junction Channel: A Route to Deliver MicroRNAs for Neurological Disease TreatmentRecapitulating in vivo-like plasticity of glioma cell invasion along blood vessels and in astrocyte-rich stroma.EphrinB2 drives perivascular invasion and proliferation of glioblastoma stem-like cells.Cracking the glioma-NK inhibitory code: toward successful innate immunotherapy.Cell Migration in 1D and 2D Nanofiber Microenvironments.Immunosuppressive Myeloid Cells' Blockade in the Glioma Microenvironment Enhances the Efficacy of Immune-Stimulatory Gene Therapy.The Invasive Region of Glioblastoma Defined by 5ALA Guided Surgery Has an Altered Cancer Stem Cell Marker Profile Compared to Central Tumour.The biology and mathematical modelling of glioma invasion: a review.Awake craniotomy versus craniotomy under general anesthesia for the surgical treatment of insular glioma: choices and outcomes.Endothelium-induced three-dimensional invasion of heterogeneous glioma initiating cells in a microfluidic coculture platform.Hostile takeover: how tumours hijack pre-existing vascular environments to thrive.VEGF Silencing Inhibits Human Osteosarcoma Angiogenesis and Promotes Cell Apoptosis via PI3K/AKT Signaling Pathway.Pattern of relapse of glioblastoma multiforme treated with radical radio-chemotherapy: Could a margin reduction be proposed?Molecular Ablation of Tumor Blood Vessels Inhibits Therapeutic Effects of Radiation and Bevacizumab.The vesicular transfer of CLIC1 from glioblastoma to microvascular endothelial cells requires TRPM7Effects of NOTCH1 signaling inhibitor γ-secretase inhibitor II on growth of cancer stem cells
P2860
Q26746028-72B15A9B-5E44-4124-A7F3-3094042EA331Q33746041-4926B693-E650-48A3-8CDB-283A1BB3973AQ34501525-52A729A5-B38B-4177-9FB0-65F3D99CD38DQ34703888-CBE9818F-F7D6-4D6A-8C09-AB55EDC65AC1Q35891430-3CA414BB-6236-4C90-8957-C9CD509501C5Q36096024-0A0B277A-870D-4D4F-925F-0529AD4E57FAQ36182716-E357EE63-5243-479D-81CC-B403D39014E3Q36238522-099061CC-4F1F-4D91-B9CC-CBF41E619B07Q36367568-7723B14C-5591-4BD7-B06F-2946FFF49353Q36408364-EF602233-F045-4391-9E62-D7ED32981F6EQ36977700-4BC12C2A-745C-4416-99EF-7B1FC9978897Q37079098-1EB5DC33-5D8C-4A95-9463-D722A381A907Q37085978-DC0E5C36-387F-48E9-A406-B49EF5C91C65Q37110603-950A6529-7AD7-4CB4-939E-8E7F073B00B6Q37567174-AC2E065E-A141-4635-B745-A559539B4C05Q37622136-D4332EFF-05F9-4FB8-9099-C86DA290A077Q37686164-3492FC8A-E55F-47AA-AAD0-C859CADDB2A0Q37687500-B0F6A86D-18C1-4071-83A6-E5C32C4F417CQ37696664-B644C2A4-9310-4B55-9E80-289E9D85231FQ38384340-7D88898B-1587-443B-B463-29484C226E53Q38635290-8965FEA2-FBC3-4174-BDB6-E72B59F0CB27Q38663895-08EBB7B5-D02E-45A3-9AB8-2BF690B6549BQ38827440-C0640561-8DC4-4263-A120-7FF005E4B92CQ38900878-3E7F8492-FCBC-4BBB-B48A-C43C6030C195Q41243197-74626D39-E584-4FED-9333-D6E6FBA8970EQ42359895-C3AEC589-7FD3-4EC4-B311-943B4688E11EQ42529862-258601FE-65EE-4274-A742-7E8E6AFE7DC8Q43103834-2D66C234-D9C0-4B90-B014-EA38A284DDEEQ45073281-0A99D7D5-0577-4E6F-B6AB-16C1AAB23A8BQ45864012-1DA805F4-D478-42F1-BC75-68E2179A2025Q47104101-0638789C-7170-4A70-995B-96C1AD4ED20CQ47124238-27888BE3-B762-4369-9C58-CFAEEB6276C7Q47227956-2C7DF12D-0287-4B03-9AB3-DCA8C7139B52Q47870836-D4162450-D3B5-4102-8AB4-A4DEBDC230F7Q48301861-78399770-C5A9-4E0B-B8A8-A07872F00413Q51676545-63D8F02C-A073-4F21-B15F-8EC77FBD037BQ52143325-99CCDE4D-90FC-47CC-90D1-E2C8128AE010Q55457297-4569DBB7-E696-41D8-A0DE-67F2BEB1BC2DQ57117113-7873E38F-6FFD-4798-AFF4-BB0005459238Q58572964-B88A8917-4ED4-4A6F-A985-C093F0645710
P2860
Mechanisms of glioma formation: iterative perivascular glioma growth and invasion leads to tumor progression, VEGF-independent vascularization, and resistance to antiangiogenic therapy.
description
2014 nî lūn-bûn
@nan
2014 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
name
Mechanisms of glioma formation ...... nce to antiangiogenic therapy.
@ast
Mechanisms of glioma formation ...... nce to antiangiogenic therapy.
@en
Mechanisms of glioma formation ...... nce to antiangiogenic therapy.
@nl
type
label
Mechanisms of glioma formation ...... nce to antiangiogenic therapy.
@ast
Mechanisms of glioma formation ...... nce to antiangiogenic therapy.
@en
Mechanisms of glioma formation ...... nce to antiangiogenic therapy.
@nl
prefLabel
Mechanisms of glioma formation ...... nce to antiangiogenic therapy.
@ast
Mechanisms of glioma formation ...... nce to antiangiogenic therapy.
@en
Mechanisms of glioma formation ...... nce to antiangiogenic therapy.
@nl
P2093
P2860
P50
P1433
P1476
Mechanisms of glioma formation ...... ance to antiangiogenic therapy
@en
P2093
Carl Koschmann
Pedro R Lowenstein
Sandra Ines Camelo-Piragua
Sebastien Motsch
Serguei Bannykh
Tom Mikkelsen
Viveka Nand Yadav
Wesley S Nichols
Yohei Mineharu
P2860
P304
P356
10.1016/J.NEO.2014.06.003
P577
2014-07-01T00:00:00Z