Grp78 heterozygosity regulates chaperone balance in exocrine pancreas with differential response to cerulein-induced acute pancreatitis - Wikidata - awgldk - TriplyDB

Grp78 heterozygosity regulates chaperone balance in exocrine pancreas with differential response to cerulein-induced acute pancreatitis

Grp78 heterozygosity regulates chaperone balance in exocrine pancreas with differential response to cerulein-induced acute pancreatitis

http://www.wikidata.org/entity/Q34358474

about

Adiponectin is essential for lipid homeostasis and survival under insulin deficiency and promotes β-cell regeneration The critical role of GRP78 in physiologic and pathologic stress.Inositol 1,4,5-trisphosphate receptor 1 mutation perturbs glucose homeostasis and enhances susceptibility to diet-induced diabetes.Endoplasmic reticulum stress may not be involved in intestinal epithelial cell apoptosis in experimental acute pancreatitis.Cell intrinsic role of COX-2 in pancreatic cancer development Role of the unfolded protein response, GRP78 and GRP94 in organ homeostasis.GRP78 plays an essential role in adipogenesis and postnatal growth in mice.Loss of acinar cell IKKα triggers spontaneous pancreatitis in mice.The 78-kD Glucose-Regulated Protein Regulates Endoplasmic Reticulum Homeostasis and Distal Epithelial Cell Survival during Lung Development C/EBP homologous protein deficiency aggravates acute pancreatitis and associated lung injury Calreticulin is highly expressed in pancreatic cancer stem-like cells.Effects of Prolonged GRP78 Haploinsufficiency on Organ Homeostasis, Behavior, Cancer and Chemotoxic Resistance in Aged Mice.GRP78 as a regulator of liver steatosis and cancer progression mediated by loss of the tumor suppressor PTEN.Environmental and genetic stressors and the unfolded protein response in exocrine pancreatic function - a hypothesis.Molecular mechanisms of pancreatic injury.Pathogenic mechanisms of acute pancreatitis.The role of the unfolded protein response in diabetes mellitus.Pathophysiological mechanisms in acute pancreatitis: Current understanding.Effect of emodin on endoplasmic reticulum stress in rats with severe acute pancreatitis.GRP78 haploinsufficiency suppresses acinar-to-ductal metaplasia, signaling, and mutant Kras-driven pancreatic tumorigenesis in mice.Hsp90 regulation affects the treatment of glucocorticoid for pancreatitis-induced lung injury.

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Grp78 heterozygosity regulates chaperone balance in exocrine pancreas with differential response to cerulein-induced acute pancreatitis

http://www.wikidata.org/entity/Q34358474

description

2010 nî lūn-bûn

@nan

2010 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի հոտեմբերին հրատարակված գիտական հոդված

@hy

2010年の論文

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2010年論文

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2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

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2010年論文

@zh-tw

2010年论文

@wuu

name

Grp78 heterozygosity regulates ...... ein-induced acute pancreatitis

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Grp78 heterozygosity regulates ...... ein-induced acute pancreatitis

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Grp78 heterozygosity regulates ...... ein-induced acute pancreatitis

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type

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Grp78 heterozygosity regulates ...... ein-induced acute pancreatitis

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Grp78 heterozygosity regulates ...... ein-induced acute pancreatitis

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Grp78 heterozygosity regulates ...... ein-induced acute pancreatitis

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prefLabel

Grp78 heterozygosity regulates ...... ein-induced acute pancreatitis

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Grp78 heterozygosity regulates ...... ein-induced acute pancreatitis

@en

Grp78 heterozygosity regulates ...... ein-induced acute pancreatitis

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P356

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The American Journal of Pathology

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Grp78 heterozygosity regulates ...... ein-induced acute pancreatitis

@en

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David R Hinton

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Ernesto Barron

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Miao Wang

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Olga A Mareninova

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Risheng Ye

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Stephen J Pandol

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P2860

Inflammation and insulin resistance

ATF6 as a transcription activator of the endoplasmic reticulum stress element: thapsigargin stress-induced changes and synergistic interactions with NF-Y and YY1

Endoplasmic reticulum stress activates cleavage of CREBH to induce a systemic inflammatory response

Essential role of the unfolded protein response regulator GRP78/BiP in protection from neuronal apoptosis

Inflammation, stress, and diabetes

Requirement of the p38 mitogen-activated protein kinase signalling pathway for the induction of the 78 kDa glucose-regulated protein/immunoglobulin heavy-chain binding protein by azetidine stress: activating transcription factor 6 as a target for st

Getting in and out from calnexin/calreticulin cycles

Hereditary pancreatitis caused by mutation-induced misfolding of human cationic trypsinogen: a novel disease mechanism

Structures of GRP94-nucleotide complexes reveal mechanistic differences between the hsp90 chaperones

ER chaperones in mammalian development and human diseases

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2827-2836

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scholarly article

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10.2353/AJPATH.2010.100368

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P433

6

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177

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2010-10-22T00:00:00Z

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47534534

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20971738

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P921

molecular chaperones

Aizkuņģa dziedzeris

P932

2993313

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