Microglial activation underlies cerebellar deficits produced by repeated cannabis exposure - Wikidata - awgldk - TriplyDB

Microglial activation underlies cerebellar deficits produced by repeated cannabis exposure

Microglial activation underlies cerebellar deficits produced by repeated cannabis exposure

http://www.wikidata.org/entity/Q34363232

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Neural Plasticity in Multiple Sclerosis: The Functional and Molecular Background Inflammation and the neural diathesis-stress hypothesis of schizophrenia: a reconceptualization Synthetic Cannabinoid-Induced Immunosuppression Augments Cerebellar Dysfunction in Tetanus-Toxin Treated Mice.Ginsenoside Rb1 attenuates activated microglia-induced neuronal damage.Chronic THC intake modifies fundamental cerebellar functions New vistas on cannabis use disorder.CB1 receptor affects cortical plasticity and response to physiotherapy in multiple sclerosis.Species differences in cannabinoid receptor 2 and receptor responses to cocaine self-administration in mice and rats Endocannabinoid signalling and the deteriorating brain Neuropsychological performance in adolescent marijuana users with co-occurring alcohol use: A three-year longitudinal study Cerebellar endocannabinoids: retrograde signaling from purkinje cells.Cannabinoid Signaling and Neuroinflammatory Diseases: A Melting pot for the Regulation of Brain Immune Responses.Infectious immunity in the central nervous system and brain function.Microglial Acid Sensing Regulates Carbon Dioxide-Evoked Fear.Divergent effects of repeated restraint versus chronic variable stress on prefrontal cortical immune status after LPS injection.Cocaine promotes oxidative stress and microglial-macrophage activation in rat cerebellum Exposure of Adolescent Mice to Delta-9-Tetrahydrocannabinol Induces Long-Lasting Modulation of Pro- and Anti-Inflammatory Cytokines in Hypothalamus and Hippocampus Similar to that Observed for Peripheral Macrophages.Broad impact of deleting endogenous cannabinoid hydrolyzing enzymes and the CB1 cannabinoid receptor on the endogenous cannabinoid-related lipidome in eight regions of the mouse brain.Modulation of Myelopoiesis Progenitors Is an Integral Component of Trained Immunity.Octadecylpropyl Sulfamide Reduces Neurodegeneration and Restores the Memory Deficits Induced by Hypoxia-Ischemia in Mice.

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Microglial activation underlies cerebellar deficits produced by repeated cannabis exposure

http://www.wikidata.org/entity/Q34363232

description

2013 nî lūn-bûn

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2013 թուականի Յունիսին հրատարակուած գիտական յօդուած

@hyw

2013 թվականի հունիսին հրատարակված գիտական հոդված

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2013年の論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年论文

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name

Microglial activation underlie ...... by repeated cannabis exposure

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Microglial activation underlie ...... by repeated cannabis exposure

@en

Microglial activation underlie ...... by repeated cannabis exposure

@nl

type

label

Microglial activation underlie ...... by repeated cannabis exposure

@ast

Microglial activation underlie ...... by repeated cannabis exposure

@en

Microglial activation underlie ...... by repeated cannabis exposure

@nl

prefLabel

Microglial activation underlie ...... by repeated cannabis exposure

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Microglial activation underlie ...... by repeated cannabis exposure

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Microglial activation underlie ...... by repeated cannabis exposure

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Journal of Clinical Investigation

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Microglial activation underlie ...... by repeated cannabis exposure

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Agnès Gruart

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Emma Puighermanal

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José María Delgado-García

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Laura Cutando

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Maria Gomis-González

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Rafael Maldonado

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P2860

Toward a global view of alcohol, tobacco, cannabis, and cocaine use: findings from the WHO World Mental Health Surveys

Resting microglial cells are highly dynamic surveillants of brain parenchyma in vivo

CB1 cannabinoid receptors and on-demand defense against excitotoxicity

Cannabinoid modulation of hippocampal long-term memory is mediated by mTOR signaling

Endogenous cannabinoid signaling through the CB1 receptor is essential for cerebellum-dependent discrete motor learning

Identification and functional characterization of brainstem cannabinoid CB2 receptors

Molecular characterization of a peripheral receptor for cannabinoids

Evidence that MDMA ('ecstasy') increases cannabinoid CB2 receptor expression in microglial cells: role in the neuroinflammatory response in rat brain

The CB1 cannabinoid receptor is the major cannabinoid receptor at excitatory presynaptic sites in the hippocampus and cerebellum

Resting microglia directly monitor the functional state of synapses in vivo and determine the fate of ischemic terminals

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2816-2831

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10.1172/JCI67569

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7

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123

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Arnau Busquets-Garcia

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2013-06-24T00:00:00Z

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