Hormones and mammary carcinogenesis in mice, rats, and humans: a unifying hypothesis.
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Expression analysis of the mouse S100A7/psoriasin gene in skin inflammation and mammary tumorigenesisNovel estrogen and tamoxifen induced genes identified by SAGE (Serial Analysis of Gene Expression)Resveratrol, a polyphenolic compound found in grapes and wine, is an agonist for the estrogen receptorBisphenol-A and the great divide: a review of controversies in the field of endocrine disruptionInduction of mammary gland ductal hyperplasias and carcinoma in situ following fetal bisphenol A exposureMetabolic inactivation of estrogens in breast tissue by UDP-glucuronosyltransferase enzymes: an overview.Radiation and breast cancer: a review of current evidenceModels of breast cancer: is merging human and animal models the future?UDP-glucuronosyltransferase and sulfotransferase polymorphisms, sex hormone concentrations, and tumor receptor status in breast cancer patientsMultilocus analysis of SNP and metabolic data within a given pathway.Genetic polymorphisms in human SULT1A1 and UGT1A1 genes associate with breast tumor characteristics: a case-series study.Depurinating estrogen-DNA adducts, generators of cancer initiation: their minimization leads to cancer preventionThe role of androgens in experimental rodent mammary carcinogenesisThe molecular etiology and prevention of estrogen-initiated cancers: Ockham's Razor: Pluralitas non est ponenda sine necessitate. Plurality should not be posited without necessityExpression of prolactin receptors in normal, benign, and malignant breast tissue: an immunohistological studyMolecular origin of cancer: catechol estrogen-3,4-quinones as endogenous tumor initiatorsModified alternate-day fasting regimens reduce cell proliferation rates to a similar extent as daily calorie restriction in miceCancer target discovery using SAGE.Balance of beneficial and deleterious health effects of quinones: a case study of the chemical properties of genistein and estrone quinones.Responsiveness to PI3K and MEK inhibitors in breast cancer. Use of a 3D culture system to study pathways related to hormone independence in mice.Selective estrogen receptor modulator delivery of quinone warheads to DNA triggering apoptosis in breast cancer cells.Neoplasia as development gone awry: the role of endocrine disruptors.Ovarian hormones are not required for PRL-induced mammary tumorigenesis, but estrogen enhances neoplastic processesVascular endothelial growth factor secreted by activated stroma enhances angiogenesis and hormone-independent growth of estrogen receptor-positive breast cancerInhibition of cancer cell proliferation and metastasis by insulin receptor downregulationA (99m)Tc-labeled triphenylphosphonium derivative for the early detection of breast tumors.Insulin receptor functionally enhances multistage tumor progression and conveys intrinsic resistance to IGF-1R targeted therapy.Biological significance of prolactin in gynecologic cancers.Quantification of epithelial cell differentiation in mammary glands and carcinomas from DMBA- and MNU-exposed rats.Redox cycling of catechol estrogens generating apurinic/apyrimidinic sites and 8-oxo-deoxyguanosine via reactive oxygen species differentiates equine and human estrogens.Effects of estrogen metabolite 2-methoxyestradiol on tumor suppressor protein p53 and proliferation of breast cancer cells.Local feedback mechanisms in human breast cancer.HES-1 inhibits 17beta-estradiol and heregulin-beta1-mediated upregulation of E2F-1.Measurement in vivo of proliferation rates of slow turnover cells by 2H2O labeling of the deoxyribose moiety of DNA.Epstein-Barr virus as a marker of biological aggressiveness in breast cancer.A yeast estrogen screen for examining the relative exposure of cells to natural and xenoestrogens.Estradiol metabolism: an endocrine biomarker for modulation of human mammary carcinogenesis.Medical hypothesis: bifunctional genetic-hormonal pathways to breast cancerTesting electromagnetic fields for potential carcinogenic activity: a critical review of animal modelsGender bias in clinical research, pharmaceutical marketing, and the prescription of drugs.
P2860
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P2860
Hormones and mammary carcinogenesis in mice, rats, and humans: a unifying hypothesis.
description
1995 nî lūn-bûn
@nan
1995 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
1995 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
1995年の論文
@ja
1995年論文
@yue
1995年論文
@zh-hant
1995年論文
@zh-hk
1995年論文
@zh-mo
1995年論文
@zh-tw
1995年论文
@wuu
name
Hormones and mammary carcinogenesis in mice, rats, and humans: a unifying hypothesis.
@ast
Hormones and mammary carcinogenesis in mice, rats, and humans: a unifying hypothesis.
@en
Hormones and mammary carcinogenesis in mice, rats, and humans: a unifying hypothesis.
@nl
type
label
Hormones and mammary carcinogenesis in mice, rats, and humans: a unifying hypothesis.
@ast
Hormones and mammary carcinogenesis in mice, rats, and humans: a unifying hypothesis.
@en
Hormones and mammary carcinogenesis in mice, rats, and humans: a unifying hypothesis.
@nl
prefLabel
Hormones and mammary carcinogenesis in mice, rats, and humans: a unifying hypothesis.
@ast
Hormones and mammary carcinogenesis in mice, rats, and humans: a unifying hypothesis.
@en
Hormones and mammary carcinogenesis in mice, rats, and humans: a unifying hypothesis.
@nl
P2093
P2860
P356
P1476
Hormones and mammary carcinogenesis in mice, rats, and humans: a unifying hypothesis.
@en
P2093
P2860
P304
P356
10.1073/PNAS.92.9.3650
P407
P577
1995-04-01T00:00:00Z