Genistein depletes telomerase activity through cross-talk between genetic and epigenetic mechanisms. - Wikidata - awgldk - TriplyDB

Genistein depletes telomerase activity through cross-talk between genetic and epigenetic mechanisms.

Genistein depletes telomerase activity through cross-talk between genetic and epigenetic mechanisms.

http://www.wikidata.org/entity/Q34369723

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Epigenetic regulation of caloric restriction in aging Therapeutic Targeting of Telomerase Regulation of the Telomerase Reverse Transcriptase Subunit through Epigenetic Mechanisms Chemoprevention of Breast Cancer by Dietary Polyphenols Environmental Epigenetics: Crossroad between Public Health, Lifestyle, and Cancer Prevention Evasion of anti-growth signaling: A key step in tumorigenesis and potential target for treatment and prophylaxis by natural compounds.Environmental epigenetics and phytoestrogen/phytochemical exposures Dietary epigenetics in cancer and aging Impact of Nutrition on Non-Coding RNA Epigenetics in Breast and Gynecological Cancer Epigenetic diet: impact on the epigenome and cancer Disruptive chemicals, senescence and immortality Better Living through Chemistry: Caloric Restriction (CR) and CR Mimetics Alter Genome Function to Promote Increased Health and Lifespan Epigenetic mechanisms underlying diet-sourced compounds in the prevention and treatment of gastrointestinal cancer Sulforaphane causes epigenetic repression of hTERT expression in human breast cancer cell lines Epigenetics meets endocrinology.Reptin is required for the transcription of telomerase reverse transcriptase and over-expressed in gastric cancer Impact on DNA methylation in cancer prevention and therapy by bioactive dietary components.Induction of Apoptosis in Pancreatic Cancer Cells by CDDO-Me Involves Repression of Telomerase through Epigenetic Pathways.Seasonality modifies methylation profiles in healthy people.Epigenetic changes induced by curcumin and other natural compounds.Regulation of the human catalytic subunit of telomerase (hTERT).Synergistic epigenetic reactivation of estrogen receptor-α (ERα) by combined green tea polyphenol and histone deacetylase inhibitor in ERα-negative breast cancer cells Cancer chemoprevention by dietary polyphenols: promising role for epigenetics.Epigenetic regulation by selected dietary phytochemicals in cancer chemoprevention.Impact of soy isoflavones on the epigenome in cancer prevention.Epigenetic targets of bioactive dietary components for cancer prevention and therapy.Does consuming isoflavones reduce or increase breast cancer risk?Epigenetic regulation of multiple tumor-related genes leads to suppression of breast tumorigenesis by dietary genistein A novel prodrug of epigallocatechin-3-gallate: differential epigenetic hTERT repression in human breast cancer cells.Combined chromatin immunoprecipitation and bisulfite methylation sequencing analysis DNA Methylation and Flavonoids in Genitourinary Cancers.Therapeutic perspectives of epigenetically active nutrients.The DNA methylation inhibitor induces telomere dysfunction and apoptosis of leukemia cells that is attenuated by telomerase over-expression.Epigenetic activities of flavonoids in the prevention and treatment of cancer Endocrine disruption of the epigenome: a breast cancer link.Targeting the epigenome with bioactive food components for cancer prevention Genomic instability in human cancer: Molecular insights and opportunities for therapeutic attack and prevention through diet and nutrition.Natural products as potential cancer therapy enhancers: A preclinical update.Inhibition of cell proliferation and induction of apoptosis by oleanane triterpenoid (CDDO-Me) in pancreatic cancer cells is associated with the suppression of hTERT gene expression and its telomerase activity Epigenetic reactivation of estrogen receptor-α (ERα) by genistein enhances hormonal therapy sensitivity in ERα-negative breast cancer.

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P2860

Genistein depletes telomerase activity through cross-talk between genetic and epigenetic mechanisms.

http://www.wikidata.org/entity/Q34369723

description

2009 nî lūn-bûn

@nan

2009 թուականի Յուլիսին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի հուլիսին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年论文

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name

Genistein depletes telomerase ...... tic and epigenetic mechanisms.

@ast

Genistein depletes telomerase ...... tic and epigenetic mechanisms.

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Genistein depletes telomerase ...... tic and epigenetic mechanisms.

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type

label

Genistein depletes telomerase ...... tic and epigenetic mechanisms.

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Genistein depletes telomerase ...... tic and epigenetic mechanisms.

@en

Genistein depletes telomerase ...... tic and epigenetic mechanisms.

@nl

prefLabel

Genistein depletes telomerase ...... tic and epigenetic mechanisms.

@ast

Genistein depletes telomerase ...... tic and epigenetic mechanisms.

@en

Genistein depletes telomerase ...... tic and epigenetic mechanisms.

@nl

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International Journal of Cancer

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Genistein depletes telomerase ...... tic and epigenetic mechanisms.

@en

P2093

Liang Liu

http://www.w3.org/2001/XMLSchema#string

Lucy G Andrews

http://www.w3.org/2001/XMLSchema#string

Trygve O Tollefsbol

http://www.w3.org/2001/XMLSchema#string

Yuanyuan Li

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P2860

hEST2, the putative human telomerase catalytic subunit gene, is up-regulated in tumor cells and during immortalization

Common 5p15.33 and 6p21.33 variants influence lung cancer risk

Addressing the soy and breast cancer relationship: review, commentary, and workshop proceedings

The human telomerase catalytic subunit hTERT: organization of the gene and characterization of the promoter

Comparison of the ligand binding specificity and transcript tissue distribution of estrogen receptors alpha and beta

Identification of a specific telomere terminal transferase activity in Tetrahymena extracts

Prepubertal estradiol and genistein exposures up-regulate BRCA1 mRNA and reduce mammary tumorigenesis

Epigenetics in human disease and prospects for epigenetic therapy

Genistein, a specific inhibitor of tyrosine-specific protein kinases

Cell-cycle dysregulation in breast cancer: breast cancer therapies targeting the cell cycle.

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286-296

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10.1002/IJC.24398

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2

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125

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2009-07-01T00:00:00Z

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24268349

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19358274

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2995334

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