Interaction between complement regulators and Streptococcus pyogenes: binding of C4b-binding protein and factor H/factor H-like protein 1 to M18 strains involves two different cell surface molecules.

Interaction between complement regulators and Streptococcus pyogenes: binding of C4b-binding protein and factor H/factor H-like protein 1 to M18 strains involves two different cell surface molecules.

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http://www.wikidata.org/entity/Q34370109
Virulence of Group A Streptococci Is Enhanced by Human Complement InhibitorsExtreme sequence divergence but conserved ligand-binding specificity in Streptococcus pyogenes M proteinStrain-specific virulence phenotypes of Streptococcus pneumoniae assessed using the Chinchilla laniger model of otitis mediaRelationship between expression of the family of M proteins and lipoteichoic acid to hydrophobicity and biofilm formation in Streptococcus pyogenesFactor H binds to the hypervariable region of many Streptococcus pyogenes M proteins but does not promote phagocytosis resistance or acute virulence.Evasion and interactions of the humoral innate immune response in pathogen invasion, autoimmune disease, and cancerFhb, a novel factor H-binding surface protein, contributes to the antiphagocytic ability and virulence of Streptococcus suis.Protease-dependent mechanisms of complement evasion by bacterial pathogens.Streptococcus pyogenes Employs Strain-dependent Mechanisms of C3b Inactivation to Inhibit Phagocytosis and Killing of Bacteria.Complement-mediated opsonization of invasive group A Streptococcus pyogenes strain AP53 is regulated by the bacterial two-component cluster of virulence responder/sensor (CovRS) system.Mechanisms of resistance of Porphyromonas gingivalis to killing by serum complement.Post-infectious Proliferative Glomerulonephritis with Monoclonal Immunoglobulin G Deposits Associated with Complement Factor H Mutation.Acquisition of factor H by a novel surface protein on group B Streptococcus promotes complement degradation.Variation, Indispensability, and Masking in the M protein.Complete deficiency of the sixth complement component (C6Q0), susceptibility to Neisseria meningitidis infections and analysis of the frequencies of C6Q0 gene defects in South Africans.Anti-phagocytic mechanisms of Streptococcus pyogenes: binding of fibrinogen to M-related protein.The antibacterial activity of peptides derived from human beta-2 glycoprotein I is inhibited by protein H and M1 protein from Streptococcus pyogenes.
P2860

Interaction between complement regulators and Streptococcus pyogenes: binding of C4b-binding protein and factor H/factor H-like protein 1 to M18 strains involves two different cell surface molecules.

Item
http://www.wikidata.org/entity/Q34370109
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2004 nî lūn-bûn
@nan
2004 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2004 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
name
Interaction between complement ...... ferent cell surface molecules.
@ast
Interaction between complement ...... ferent cell surface molecules.
@en
Interaction between complement ...... ferent cell surface molecules.
@nl
type
label
Interaction between complement ...... ferent cell surface molecules.
@ast
Interaction between complement ...... ferent cell surface molecules.
@en
Interaction between complement ...... ferent cell surface molecules.
@nl
prefLabel
Interaction between complement ...... ferent cell surface molecules.
@ast
Interaction between complement ...... ferent cell surface molecules.
@en
Interaction between complement ...... ferent cell surface molecules.
@nl
P1433
P1476
P2093
P304
P31
P356
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P433
P478
P577
P5875
P698
P356
P1433
P1476
Interaction between complement ...... ferent cell surface molecules.
@en
P2093
P304
P31
P356
10.4049/JIMMUNOL.173.11.6899
P407
P433
P478
P577
P5875
P698