Positive cross-talk between estrogen receptor and NF-kappaB in breast cancer.
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Carcinogenic effects of "whole-life" exposure to inorganic arsenic in CD1 miceThe Interactions of Obesity, Inflammation and Insulin Resistance in Breast CancerFucoxanthin and Its Metabolite Fucoxanthinol in Cancer Prevention and TreatmentMinireview: Inflammation: an instigator of more aggressive estrogen receptor (ER) positive breast cancersCyclopia extracts act as ERα antagonists and ERβ agonists, in vitro and in vivoSustained proliferation in cancer: Mechanisms and novel therapeutic targetsChIP-PED enhances the analysis of ChIP-seq and ChIP-chip dataLRP16 integrates into NF-κB transcriptional complex and is required for its functional activation.Palmitoylation: a protein S-acylation with implications for breast cancer.An integrated bioinformatics approach identifies elevated cyclin E2 expression and E2F activity as distinct features of tamoxifen resistant breast tumors.Estrogen signaling crosstalk: Implications for endocrine resistance in ovarian cancer.CBP mediates NF-κB-dependent histone acetylation and estrogen receptor recruitment to an estrogen response element in the BIRC3 promoter.The atypical ubiquitin ligase RNF31 stabilizes estrogen receptor α and modulates estrogen-stimulated breast cancer cell proliferationGenome-wide progesterone receptor binding: cell type-specific and shared mechanisms in T47D breast cancer cells and primary leiomyoma cells.Proinflammatory cytokines enhance estrogen-dependent expression of the multidrug transporter gene ABCG2 through estrogen receptor and NF{kappa}B cooperativity at adjacent response elements.Rb inactivation accelerates neoplastic growth and substitutes for recurrent amplification of cIAP1, cIAP2 and Yap1 in sporadic mammary carcinoma associated with p53 deficiency.Growth hormone potentiates 17β-estradiol-dependent breast cancer cell proliferation independently of IGF-I receptor signaling.Estrogen promotes breast cancer cell survival in an inhibitor of apoptosis (IAP)-dependent manner.Negative regulation of NF-κB by the ING4 tumor suppressor in breast cancerδEF1 down-regulates ER-α expression and confers tamoxifen resistance in breast cancerPHLDA1 expression is controlled by an estrogen receptor-NFκB-miR-181 regulatory loop and is essential for formation of ER+ mammospheresInnate immunity pathways and breast cancer Risk in African American and European-American women in the Women's Circle of Health Study (WCHS).Progesterone receptor-B induction of BIRC3 protects endometrial cancer cells from AP1-59-mediated apoptosis.TNFα signaling exposes latent estrogen receptor binding sites to alter the breast cancer cell transcriptomeRole of NF-κB in the skeletonNFκB affects estrogen receptor expression and activity in breast cancer through multiple mechanisms.Genomic instability influences the transcriptome and proteome in endometrial cancer subtypes.A novel aspirin prodrug inhibits NFκB activity and breast cancer stem cell properties.Associations of C-Reactive Protein, Granulocytes and Granulocyte-to-Lymphocyte Ratio with Mortality from Breast Cancer in Non-Institutionalized American Women.The scaffold protein MEK Partner 1 is required for the survival of estrogen receptor positive breast cancer cellsPrenatal exposure of mice to diethylstilbestrol disrupts T-cell differentiation by regulating Fas/Fas ligand expression through estrogen receptor element and nuclear factor-κB motifs.Estrogen receptor beta as target for colorectal cancer preventionStromal cells promote anti-estrogen resistance of breast cancer cells through an insulin-like growth factor binding protein 5 (IGFBP5)/B-cell leukemia/lymphoma 3 (Bcl-3) axisThe interaction between ER and NFκB in resistance to endocrine therapy.Nuclear factor-ĸB plays a critical role in both intrinsic and acquired resistance against endocrine therapy in human breast cancer cells.Role of nuclear factor-ĸB in breast and colorectal cancer.Methylation profiling with a panel of cancer related genes: association with estrogen receptor, TP53 mutation status and expression subtypes in sporadic breast cancer.Role of Estrogen Response Element in the Human Prolactin Gene: Transcriptional Response and TimingInhibitory effects of fucoxanthinol on the viability of human breast cancer cell lines MCF-7 and MDA-MB-231 are correlated with modulation of the NF-kappaB pathway.GR and ER Coactivation Alters the Expression of Differentiation Genes and Associates with Improved ER+ Breast Cancer Outcome.
P2860
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P2860
Positive cross-talk between estrogen receptor and NF-kappaB in breast cancer.
description
2009 nî lūn-bûn
@nan
2009 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
Positive cross-talk between estrogen receptor and NF-kappaB in breast cancer.
@ast
Positive cross-talk between estrogen receptor and NF-kappaB in breast cancer.
@en
Positive cross-talk between estrogen receptor and NF-kappaB in breast cancer.
@nl
type
label
Positive cross-talk between estrogen receptor and NF-kappaB in breast cancer.
@ast
Positive cross-talk between estrogen receptor and NF-kappaB in breast cancer.
@en
Positive cross-talk between estrogen receptor and NF-kappaB in breast cancer.
@nl
prefLabel
Positive cross-talk between estrogen receptor and NF-kappaB in breast cancer.
@ast
Positive cross-talk between estrogen receptor and NF-kappaB in breast cancer.
@en
Positive cross-talk between estrogen receptor and NF-kappaB in breast cancer.
@nl
P2093
P2860
P1433
P1476
Positive cross-talk between estrogen receptor and NF-kappaB in breast cancer.
@en
P2093
Adina Stanculescu
Aisha Weaver
Jonna Frasor
Lance D Miller
Madhumita Pradhan
P2860
P304
P356
10.1158/0008-5472.CAN-09-2608
P407
P50
P577
2009-11-17T00:00:00Z