Systemic preconditioning by a prolyl hydroxylase inhibitor promotes prevention of skin flap necrosis via HIF-1-induced bone marrow-derived cells.

Systemic preconditioning by a prolyl hydroxylase inhibitor promotes prevention of skin flap necrosis via HIF-1-induced bone marrow-derived cells.

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http://www.wikidata.org/entity/Q34374873
Hypoxia-based strategies for angiogenic induction: the dawn of a new era for ischemia therapy and tissue regenerationMolecular mechanisms of action and therapeutic uses of pharmacological inhibitors of HIF-prolyl 4-hydroxylases for treatment of ischemic diseasesDimethyloxalylglycine prevents bone loss in ovariectomized C57BL/6J mice through enhanced angiogenesis and osteogenesis.HIF-1α transgenic bone marrow cells can promote tissue repair in cases of corticosteroid-induced osteonecrosis of the femoral head in rabbits.Preconditioning of bone marrow mesenchymal stem cells by prolyl hydroxylase inhibition enhances cell survival and angiogenesis in vitro and after transplantation into the ischemic heart of ratsMiRNA-Sequence Indicates That Mesenchymal Stem Cells and Exosomes Have Similar Mechanism to Enhance Cardiac Repair.Tumour blood vessel normalisation by prolyl hydroxylase inhibitor repaired sensitivity to chemotherapy in a tumour mouse model.Prolyl 4-hydroxylases, master regulators of the hypoxia response.Prolyl hydroxylase inhibitors act as agents to enhance the efficiency of cell therapy.Early upregulation of myocardial CXCR4 expression is critical for dimethyloxalylglycine-induced cardiac improvement in acute myocardial infarction.Therapeutic inhibition of prolyl hydroxylase domain-containing enzymes in surgery: putative applications and challenges.Overexpressed HIF-2α in Endothelial Cells Promotes Vascularization and Improves Random Pattern Skin Flap Survival.Topical application of nitrosonifedipine, a novel radical scavenger, ameliorates ischemic skin flap necrosis in a mouse model.Dimethyloxalylglycine lyophilized onto bone substitutes increase vessel area in rat calvarial defects
P2860

Systemic preconditioning by a prolyl hydroxylase inhibitor promotes prevention of skin flap necrosis via HIF-1-induced bone marrow-derived cells.

Item
http://www.wikidata.org/entity/Q34374873
description
2012 nî lūn-bûn
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2012 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2012 թվականի օգոստոսին հրատարակված գիտական հոդված
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2012年の論文
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2012年論文
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2012年論文
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2012年論文
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2012年論文
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2012年論文
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2012年论文
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Systemic preconditioning by a ...... ced bone marrow-derived cells.
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Systemic preconditioning by a ...... ced bone marrow-derived cells.
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Systemic preconditioning by a ...... ced bone marrow-derived cells.
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Systemic preconditioning by a ...... ced bone marrow-derived cells.
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Systemic preconditioning by a ...... ced bone marrow-derived cells.
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Systemic preconditioning by a ...... ced bone marrow-derived cells.
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Systemic preconditioning by a ...... ced bone marrow-derived cells.
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Systemic preconditioning by a ...... ced bone marrow-derived cells.
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Systemic preconditioning by a ...... ced bone marrow-derived cells.
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10.1371/JOURNAL.PONE.0042964
P407
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P932