NMDA receptor hypofunction produces concomitant firing rate potentiation and burst activity reduction in the prefrontal cortex.
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GABAergic interneuron origin of schizophrenia pathophysiology.Positive allosteric modulation of metabotropic glutamate 5 (mGlu5) receptors reverses N-Methyl-D-aspartate antagonist-induced alteration of neuronal firing in prefrontal cortexGroup II metabotropic glutamate receptor agonist ameliorates MK801-induced dysfunction of NMDA receptors via the Akt/GSK-3β pathway in adult rat prefrontal cortexNeural synchrony in schizophrenia: from networks to new treatmentsCircuit-based framework for understanding neurotransmitter and risk gene interactions in schizophreniaDistinct neuronal coding schemes in memory revealed by selective erasure of fast synchronous synaptic transmission.Contribution of NMDA receptors to dorsolateral prefrontal cortical networks in primatesReview of pharmacological treatment in mood disorders and future directions for drug development.Thinking outside the cleft to understand synaptic activity: contribution of the cystine-glutamate antiporter (System xc-) to normal and pathological glutamatergic signalingNeuromagnetic oscillations predict evoked-response latency delays and core language deficits in autism spectrum disorders.Knockout of NMDA-receptors from parvalbumin interneurons sensitizes to schizophrenia-related deficits induced by MK-801Excitation-inhibition discoordination in rodent models of mental disorders.The impact of NMDA receptor hypofunction on GABAergic neurons in the pathophysiology of schizophreniaXanthurenic Acid Activates mGlu2/3 Metabotropic Glutamate Receptors and is a Potential Trait Marker for SchizophreniaGlutamate concentration in the medial prefrontal cortex predicts resting-state cortical-subcortical functional connectivity in humansSpatial and temporal boundaries of NMDA receptor hypofunction leading to schizophrenia.Mechanisms of Functional Hypoconnectivity in the Medial Prefrontal Cortex of Mecp2 Null Mice.Selective activation of M4 muscarinic acetylcholine receptors reverses MK-801-induced behavioral impairments and enhances associative learning in rodents.Sustained N-methyl-d-aspartate receptor hypofunction remodels the dopamine system and impairs phasic signaling.How Nox2-containing NADPH oxidase affects cortical circuits in the NMDA receptor antagonist model of schizophrenia.Reduction in phencyclidine induced sensorimotor gating deficits in the rat following increased system xc⁻ activity in the medial prefrontal cortex.The electrophysiological signature of motivational salience in mice and implications for schizophrenia.Contribution of NMDA receptor hypofunction in prefrontal and cortical excitatory neurons to schizophrenia-like phenotypes.Genetic models of sensorimotor gating: relevance to neuropsychiatric disorders.Assessment of glutamate transporter GLAST (EAAT1)-deficient mice for phenotypes relevant to the negative and executive/cognitive symptoms of schizophrenia.Acute administration of typical and atypical antipsychotics reduces EEG γ power, but only the preclinical compound LY379268 reduces the ketamine-induced rise in γ power.The H3 antagonist, ciproxifan, alleviates the memory impairment but enhances the motor effects of MK-801 (dizocilpine) in ratsMethamphetamine self-administration produces attentional set-shifting deficits and alters prefrontal cortical neurophysiology in rats.The role of GluN2A and GluN2B subunits on the effects of NMDA receptor antagonists in modeling schizophrenia and treating refractory depressionNMDA Receptors Enhance Spontaneous Activity and Promote Neuronal Survival in the Developing CochleaKetamine Alters Outcome-Related Local Field Potentials in Monkey Prefrontal Cortex.Encoding of spatio-temporal input characteristics by a CA1 pyramidal neuron model.NMDA receptor subunit expression in GABAergic interneurons in the prefrontal cortex: application of laser microdissection technique.Dizocilpine (MK-801) induces distinct changes of N-methyl-D-aspartic acid receptor subunits in parvalbumin-containing interneurons in young adult rat prefrontal cortexPostnatal NMDA receptor ablation in corticolimbic interneurons confers schizophrenia-like phenotypes.Arc regulates experience-dependent persistent firing patterns in frontal cortex.Audiovisual Distraction Increases Prefrontal Cortical Neuronal Activity and Impairs Attentional Performance in the RatSubanesthetic ketamine treatment promotes abnormal interactions between neural subsystems and alters the properties of functional brain networks.Antipsychotics reverse abnormal EEG complexity in drug-naive schizophrenia: a multiscale entropy analysisCannabinoids and Vanilloids in Schizophrenia: Neurophysiological Evidence and Directions for Basic Research.
P2860
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P2860
NMDA receptor hypofunction produces concomitant firing rate potentiation and burst activity reduction in the prefrontal cortex.
description
2004 nî lūn-bûn
@nan
2004 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2004 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
name
NMDA receptor hypofunction pro ...... tion in the prefrontal cortex.
@ast
NMDA receptor hypofunction pro ...... tion in the prefrontal cortex.
@en
NMDA receptor hypofunction pro ...... tion in the prefrontal cortex.
@nl
type
label
NMDA receptor hypofunction pro ...... tion in the prefrontal cortex.
@ast
NMDA receptor hypofunction pro ...... tion in the prefrontal cortex.
@en
NMDA receptor hypofunction pro ...... tion in the prefrontal cortex.
@nl
prefLabel
NMDA receptor hypofunction pro ...... tion in the prefrontal cortex.
@ast
NMDA receptor hypofunction pro ...... tion in the prefrontal cortex.
@en
NMDA receptor hypofunction pro ...... tion in the prefrontal cortex.
@nl
P2860
P356
P1476
NMDA receptor hypofunction pro ...... tion in the prefrontal cortex.
@en
P2093
Houman Homayoun
Mark E Jackson
P2860
P304
P356
10.1073/PNAS.0308455101
P407
P577
2004-05-24T00:00:00Z