Modulation of endothelial cell adhesion by hevin, an acidic protein associated with high endothelial venules.
about
Hevin is down-regulated in many cancers and is a negative regulator of cell growth and proliferationHeterogeneity of endothelial cells: the specialized phenotype of human high endothelial venules characterized by suppression subtractive hybridizationMolecular characterization of NF-HEV, a nuclear factor preferentially expressed in human high endothelial venulesUpregulated genes in sporadic, idiopathic pulmonary arterial hypertension.SPARC, a matricellular glycoprotein with important biological functionsDifferential gene expression profile of human tonsil high endothelial cells: implications for lymphocyte traffickingSPARC (secreted protein acidic and rich in cysteine) induces apoptosis in ovarian cancer cellsNorrin, frizzled-4, and Lrp5 signaling in endothelial cells controls a genetic program for retinal vascularization.SC1/hevin. An extracellular calcium-modulated protein that binds collagen I.Tumor-suppressor function of SPARC-like protein 1/Hevin in pancreatic cancerRgcs1, a dominant QTL that affects retinal ganglion cell death after optic nerve crush in mice.Matricellular proteins in the trabecular meshwork: review and update.Matricellular hevin regulates decorin production and collagen assembly.SPARC fusion protein induces cellular adhesive signaling.The heparin-binding activity of secreted modular calcium-binding protein 1 (SMOC-1) modulates its cell adhesion propertiesInteraction of recombinant myocilin with the matricellular protein SPARC: functional implicationsExpression, functional, and structural analysis of proteins critical for otoconia developmentEffect of hevin deletion in mice and characterization in trabecular meshworkHevin plays a pivotal role in corneal wound healing.Matricellular homologs in the foreign body response: hevin suppresses inflammation, but hevin and SPARC together diminish angiogenesis.Cellular Responses Modulated by FGF-2 Adsorbed on Albumin/Heparin Layer-by-Layer AssembliesSecreted protein, acidic and rich in cysteine-like 1 (SPARCL1) is down regulated in aggressive prostate cancers and is prognostic for poor clinical outcome.Suppression subtractive hybridization identifies distinctive expression markers for coronary and internal mammary arteriesA BAC transgenic mouse model to analyze the function of astroglial SPARCL1 (SC1) in the central nervous system.Cloning and expression of murine SC1, a gene product homologous to SPARC.Matricellular proteins as regulators of cancer metastasis to bone.SPARCL1 suppresses metastasis in prostate cancer.Matricellular protein SPARCL1 regulates tumor microenvironment-dependent endothelial cell heterogeneity in colorectal carcinoma.Individualized analysis reveals CpG sites with methylation aberrations in almost all lung adenocarcinoma tissues.Signature patterns of gene expression in mouse atherosclerosis and their correlation to human coronary disease.Extracellular matrix-associated protein Sc1 is not essential for mouse development.Immunofibrogenic Gene Expression Patterns in Tanzanian Children with Ocular Chlamydia trachomatis Infection, Active Trachoma and Scarring: Baseline Results of a 4-Year Longitudinal Study.Crystal structure of a pair of follistatin-like and EF-hand calcium-binding domains in BM-40.Mice deficient for the secreted glycoprotein SPARC/osteonectin/BM40 develop normally but show severe age-onset cataract formation and disruption of the lens.Galectin-8 functions as a matricellular modulator of cell adhesion.Altered tissue repair in hevin-null mice: inhibition of fibroblast migration by a matricellular SPARC homolog.Expression and characterization of murine hevin (SC1), a member of the SPARC family of matricellular proteins.Brain endothelial cell expression of SPARCL-1 is specific to chronic multiple sclerosis lesions and is regulated by inflammatory mediators in vitro.Characterization of SMOC-1, a novel modular calcium-binding protein in basement membranes.SPARC-like 1 (SC1) is a diversely expressed and developmentally regulated matricellular protein that does not compensate for the absence of SPARC in the CNS.
P2860
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P2860
Modulation of endothelial cell adhesion by hevin, an acidic protein associated with high endothelial venules.
description
1996 nî lūn-bûn
@nan
1996 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
1996 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
1996年の論文
@ja
1996年論文
@yue
1996年論文
@zh-hant
1996年論文
@zh-hk
1996年論文
@zh-mo
1996年論文
@zh-tw
1996年论文
@wuu
name
Modulation of endothelial cell ...... with high endothelial venules.
@ast
Modulation of endothelial cell ...... with high endothelial venules.
@en
Modulation of endothelial cell ...... with high endothelial venules.
@nl
type
label
Modulation of endothelial cell ...... with high endothelial venules.
@ast
Modulation of endothelial cell ...... with high endothelial venules.
@en
Modulation of endothelial cell ...... with high endothelial venules.
@nl
prefLabel
Modulation of endothelial cell ...... with high endothelial venules.
@ast
Modulation of endothelial cell ...... with high endothelial venules.
@en
Modulation of endothelial cell ...... with high endothelial venules.
@nl
P2860
P356
P1476
Modulation of endothelial cell ...... with high endothelial venules.
@en
P2093
Springer TA
P2860
P304
P356
10.1074/JBC.271.8.4511
P407
P577
1996-02-01T00:00:00Z