Increased postischemic brain injury in mice deficient in uracil-DNA glycosylase. - Wikidata - awgldk - TriplyDB

Increased postischemic brain injury in mice deficient in uracil-DNA glycosylase.

Increased postischemic brain injury in mice deficient in uracil-DNA glycosylase.

http://www.wikidata.org/entity/Q34380700

about

The excitatory neurotransmitter glutamate stimulates DNA repair to increase neuronal resiliency Neuropharmacological Potential of Gastrodia elata Blume and Its Components Uracil DNA N-glycosylase promotes assembly of human centromere protein A DNA methylation, its mediators and genome integrity Base excision repair AID produces DNA double-strand breaks in non-Ig genes and mature B cell lymphomas with reciprocal chromosome translocations.C --> T mutagenesis and gamma-radiation sensitivity due to deficiency in the Smug1 and Ung DNA glycosylases Mitochondrial base excision repair assays DNA repair deficiency in neurodegeneration.Aag-initiated base excision repair promotes ischemia reperfusion injury in liver, brain, and kidney.RPA accumulation during class switch recombination represents 5'-3' DNA-end resection during the S-G2/M phase of the cell cycle Evidence that OGG1 glycosylase protects neurons against oxidative DNA damage and cell death under ischemic conditions.DNA repair mechanisms in dividing and non-dividing cells.Endonuclease VIII-like 3 (Neil3) DNA glycosylase promotes neurogenesis induced by hypoxia-ischemia.Essential role of interleukin-6 in post-stroke angiogenesis Mouse model of endemic Burkitt translocations reveals the long-range boundaries of Ig-mediated oncogene deregulation Role of genomic instability and p53 in AID-induced c-myc-Igh translocations.Folate and colorectal cancer in rodents: a model of DNA repair deficiency.Genetic variability in DNA repair proteins in age-related macular degeneration.Base excision repair in physiology and pathology of the central nervous system Combination treatment with progesterone and vitamin D hormone is more effective than monotherapy in ischemic stroke: the role of BDNF/TrkB/Erk1/2 signaling in neuroprotection.'To repair or not to repair - no longer a question': repair of mitochondrial DNA shielding against age and cancer.DNA damage and repair: relevance to mechanisms of neurodegeneration Base excision repair, aging and health span.Folate deficiency induces genomic uracil misincorporation and hypomethylation but does not increase DNA point mutations.Brain capacity for repair of oxidatively damaged DNA and preservation of neuronal function.Oxidative stress alters base excision repair pathway and increases apoptotic response in apurinic/apyrimidinic endonuclease 1/redox factor-1 haploinsufficient mice.Base excision repair in the mammalian brain: implication for age related neurodegeneration Folate deficiency induces neurodegeneration and brain dysfunction in mice lacking uracil DNA glycosylase.The cell cycle restricts activation-induced cytidine deaminase activity to early G1.DNA damage and impairment of DNA repair in Alzheimer's disease.The role of DNA base excision repair in brain homeostasis and disease.Development of a Cell-Based Assay for Measuring Base Excision Repair Responses.Mitochondrial DNA damage and the involvement of antioxidant defense and repair system in hippocampi of rats with chronic seizures.The ectonucleotidase cd39/ENTPDase1 modulates purinergic-mediated microglial migration.Phosphorylation promotes activation-induced cytidine deaminase activity at the Myc oncogene.Bone marrow-derived mesenchymal stem cells ameliorate sodium nitrite-induced hypoxic brain injury in a rat model.The subpopulation of microglia expressing functional muscarinic acetylcholine receptors expands in stroke and Alzheimer's disease.Novel roles for metallothionein-I + II (MT-I + II) in defense responses, neurogenesis, and tissue restoration after traumatic brain injury: insights from global gene expression profiling in wild-type and MT-I + II knockout mice.Quercetin inhibits sodium nitrite-induced inflammation and apoptosis in different rats organs by suppressing Bax, HIF1-α, TGF-β, Smad-2, and AKT pathways.

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Increased postischemic brain injury in mice deficient in uracil-DNA glycosylase.

http://www.wikidata.org/entity/Q34380700

description

2004 nî lūn-bûn

@nan

2004 թուականի Յունիսին հրատարակուած գիտական յօդուած

@hyw

2004 թվականի հունիսին հրատարակված գիտական հոդված

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2004年の論文

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2004年論文

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2004年論文

@zh-hant

2004年論文

@zh-hk

2004年論文

@zh-mo

2004年論文

@zh-tw

2004年论文

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name

Increased postischemic brain injury in mice deficient in uracil-DNA glycosylase.

@ast

Increased postischemic brain injury in mice deficient in uracil-DNA glycosylase.

@en

Increased postischemic brain injury in mice deficient in uracil-DNA glycosylase.

@nl

type

label

Increased postischemic brain injury in mice deficient in uracil-DNA glycosylase.

@ast

Increased postischemic brain injury in mice deficient in uracil-DNA glycosylase.

@en

Increased postischemic brain injury in mice deficient in uracil-DNA glycosylase.

@nl

prefLabel

Increased postischemic brain injury in mice deficient in uracil-DNA glycosylase.

@ast

Increased postischemic brain injury in mice deficient in uracil-DNA glycosylase.

@en

Increased postischemic brain injury in mice deficient in uracil-DNA glycosylase.

@nl

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Journal of Clinical Investigation

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Increased postischemic brain injury in mice deficient in uracil-DNA glycosylase.

@en

P2093

Andreas Lipski

http://www.w3.org/2001/XMLSchema#string

Detlev Biniszkiewicz

http://www.w3.org/2001/XMLSchema#string

Juri Katchanov

http://www.w3.org/2001/XMLSchema#string

Matthias Endres

http://www.w3.org/2001/XMLSchema#string

Michael Ahmadi

http://www.w3.org/2001/XMLSchema#string

Philipp Mergenthaler

http://www.w3.org/2001/XMLSchema#string

Robert W Sobol

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Samuel H Wilson

http://www.w3.org/2001/XMLSchema#string

P2860

Post-replicative base excision repair in replication foci

The spectrum of spontaneous mutations in a Saccharomyces cerevisiae uracil-DNA-glycosylase mutant limits the function of this enzyme to cytosine deamination repair.

Uracil-DNA glycosylase-deficient yeast exhibit a mitochondrial mutator phenotype.

The thymine glycosylase MBD4 can bind to the product of deamination at methylated CpG sites

Excision of deaminated cytosine from the vertebrate genome: role of the SMUG1 uracil-DNA glycosylase

Cloning and Expression of Human G/T Mismatch-specific Thymine-DNA Glycosylase

Mitochondria and apoptosis

Properties and functions of human uracil-DNA glycosylase from the UNG gene.

Differences in vulnerability to permanent focal cerebral ischemia among 3 common mouse strains.

Mitochondrial depolarization accompanies cytochrome c release during apoptosis in PC6 cells.

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1711-1721

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scholarly article

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10.1172/JCI200420926

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12

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113

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Christoph Harms

Rudolf Jaenisch

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2004-06-01T00:00:00Z

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15199406

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420508

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