Cag pathogenicity island-specific responses of gastric epithelial cells to Helicobacter pylori infection - Wikidata - awgldk - TriplyDB

Cag pathogenicity island-specific responses of gastric epithelial cells to Helicobacter pylori infection

Cag pathogenicity island-specific responses of gastric epithelial cells to Helicobacter pylori infection

http://www.wikidata.org/entity/Q34381529

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Helicobacter pylori persistence: biology and disease Activation of the abundant nuclear factor poly(ADP-ribose) polymerase-1 by Helicobacter pylori Helicobacter pylori-induced Sonic Hedgehog expression is regulated by NFκB pathway activation: the use of a novel in vitro model to study epithelial response to infection Toll-like receptor (TLR) 2 and TLR5, but not TLR4, are required for Helicobacter pylori-induced NF-kappa B activation and chemokine expression by epithelial cells Automated discovery of functional generality of human gene expression programs Helicobacter pylori-induced histone modification, associated gene expression in gastric epithelial cells, and its implication in pathogenesis.Toll-like receptor 2-mediated gene expression in epithelial cells during Helicobacter pylori infection.Helicobacter pylori CagA induces AGS cell elongation through a cell retraction defect that is independent of Cdc42, Rac1, and Arp2/3.Phylogeographic origin of Helicobacter pylori determines host-adaptive responses upon coculture with gastric epithelial cells.STEM: a tool for the analysis of short time series gene expression data.The impact of parietal cells on Helicobacter pylori tropism and host pathology: an analysis using gnotobiotic normal and transgenic mice.Global analysis of the human gastric epithelial transcriptome altered by Helicobacter pylori eradication in vivo.Extracting biologically significant patterns from short time series gene expression data.A Helicobacter pylori Homolog of Eukaryotic Flotillin Is Involved in Cholesterol Accumulation, Epithelial Cell Responses and Host Colonization Inhibition of heat shock protein expression by Helicobacter pylori.Functional antagonism between Helicobacter pylori CagA and vacuolating toxin VacA in control of the NFAT signaling pathway in gastric epithelial cells Helicobacter pylori interacts with the human single-domain trefoil protein TFF1.Helicobacter pylori exploits a unique repertoire of type IV secretion system components for pilus assembly at the bacteria-host cell interface Genes required for assembly of pili associated with the Helicobacter pylori cag type IV secretion system Interleukin-8 is the single most up-regulated gene in whole genome profiling of H. pylori exposed gastric epithelial cells.Helicobacter pylori infection, gastrin and cyclooxygenase-2 in gastric carcinogenesis Queuosine biosynthesis is required for sinorhizobium meliloti-induced cytoskeletal modifications on HeLa Cells and symbiosis with Medicago truncatula.The effect of Helicobacter pylori CagA on the HER-2 copy number and expression in gastric cancer.Helicobacter pylori persistence: an overview of interactions between H. pylori and host immune defenses Quantitative analysis of the effect of Helicobacter pylori on the expressions of SOX2, CDX2, MUC2, MUC5AC, MUC6, TFF1, TFF2, and TFF3 mRNAs in human gastric carcinoma cells.RNA profiling in host-pathogen interactions.Helicobacter pylori: Genomic Insight into the Host-Pathogen Interaction.Diagnostic methods for Helicobacter pylori detection and eradication Paxillin is a novel cellular target for converging Helicobacter pylori-induced cellular signaling.CD44 plays a functional role in Helicobacter pylori-induced epithelial cell proliferation.Molecular Koch's postulates applied to bacterial pathogenicity--a personal recollection 15 years later.H. pylori infection: bacterial virulence factors and cytokine gene polymorphisms as determinants of infection outcome.Peptidomimetic Small Molecules Disrupt Type IV Secretion System Activity in Diverse Bacterial Pathogens.Helicobacter pylori HP0231 Influences Bacterial Virulence and Is Essential for Gastric Colonization Insights into host responses against pathogens from transcriptional profiling.L-forms of H. pylori NF-kappaB translocation prevents host cell death after low-dose challenge by Legionella pneumophila.Association between cag-pathogenicity island in Helicobacter pylori isolates from peptic ulcer, gastric carcinoma, and non-ulcer dyspepsia subjects with histological changes Inactivation of the type IV secretion system reduces the Th1 polarization of the immune response to Brucella abortus infection.H. pylori virulence factor CagA increases intestinal cell proliferation by Wnt pathway activation in a transgenic zebrafish model.

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Cag pathogenicity island-specific responses of gastric epithelial cells to Helicobacter pylori infection

http://www.wikidata.org/entity/Q34381529

description

2002 nî lūn-bûn

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2002 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2002 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2002年の論文

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2002年論文

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2002年論文

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2002年论文

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Cag pathogenicity island-speci ...... Helicobacter pylori infection

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Cag pathogenicity island-speci ...... Helicobacter pylori infection

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Cag pathogenicity island-speci ...... Helicobacter pylori infection

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Cag pathogenicity island-speci ...... Helicobacter pylori infection

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Cag pathogenicity island-speci ...... Helicobacter pylori infection

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Cag pathogenicity island-speci ...... Helicobacter pylori infection

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Cag pathogenicity island-speci ...... Helicobacter pylori infection

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Cag pathogenicity island-speci ...... Helicobacter pylori infection

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Proceedings of the National Academy of Sciences of the United States of America

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Cag pathogenicity island-speci ...... Helicobacter pylori infection

@en

P2093

Karen Guillemin

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Lucy S Tompkins

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Nina R Salama

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Stanley Falkow

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P2860

A new family of Cdc42 effector proteins, CEPs, function in fibroblast and epithelial cell shape changes

The Borgs, a new family of Cdc42 and TC10 GTPase-interacting proteins.

Molecular characterization of the 128-kDa immunodominant antigen of Helicobacter pylori associated with cytotoxicity and duodenal ulcer

Significance analysis of microarrays applied to the ionizing radiation response

Cluster analysis and display of genome-wide expression patterns

Altered states: involvement of phosphorylated CagA in the induction of host cellular growth changes by Helicobacter pylori

c-Src/Lyn kinases activate Helicobacter pylori CagA through tyrosine phosphorylation of the EPIYA motifs.

Translocation of the Helicobacter pylori CagA protein in gastric epithelial cells by a type IV secretion apparatus.

SHP-2 tyrosine phosphatase as an intracellular target of Helicobacter pylori CagA protein.

Tyrosine-phosphorylated bacterial proteins: Trojan horses for the host cell.

P304

15136-15141

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scholarly article

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10.1073/PNAS.182558799

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23

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99

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12411577

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Helicobacter pylori

P932

137556

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