P7C3 neuroprotective chemicals block axonal degeneration and preserve function after traumatic brain injury - Wikidata - awgldk - TriplyDB

P7C3 neuroprotective chemicals block axonal degeneration and preserve function after traumatic brain injury

P7C3 neuroprotective chemicals block axonal degeneration and preserve function after traumatic brain injury

http://www.wikidata.org/entity/Q34389585

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Traumatic Axonal Injury: Mechanisms and Translational Opportunities Increasing Adult Hippocampal Neurogenesis is Sufficient to Reduce Anxiety and Depression-Like Behaviors Protective efficacy of P7C3-S243 in the 6-hydroxydopamine model of Parkinson's disease.Recent Advances in Neurogenic Small Molecules as Innovative Treatments for Neurodegenerative Diseases The Eating-Disorder Associated HDAC4A778T Mutation Alters Feeding Behaviors in Female Mice.Behavioral disturbances in estrogen-related receptor alpha-null mice.NAD(+) Metabolism and the Control of Energy Homeostasis: A Balancing Act between Mitochondria and the Nucleus An Aminopropyl Carbazole Derivative Induces Neurogenesis by Increasing Final Cell Division in Neural Stem Cells.Attenuated traumatic axonal injury and improved functional outcome after traumatic brain injury in mice lacking Sarm1.Early detection of subclinical visual damage after blast-mediated TBI enables prevention of chronic visual deficit by treatment with P7C3-S243.Neuropsychiatric Symptom Modeling in Male and Female C57BL/6J Mice after Experimental Traumatic Brain Injury.Pyridine Dinucleotides from Molecules to Man.Neuroregeneration versus neurodegeneration: toward a paradigm shift in Alzheimer's disease drug discovery.P7C3 inhibits GSK3β activation to protect dopaminergic neurons against neurotoxin-induced cell death in vitro and in vivo.Degradomics in Neurotrauma: Profiling Traumatic Brain Injury.Targeting Nicotinamide Phosphoribosyltransferase as a Potential Therapeutic Strategy to Restore Adult Neurogenesis.Acute Axonal Degeneration Drives Development of Cognitive, Motor, and Visual Deficits after Blast-Mediated Traumatic Brain Injury in Mice.From Gene to Behavior: L-Type Calcium Channel Mechanisms Underlying Neuropsychiatric Symptoms.Visual system pathology in humans and animal models of blast injury.The Neuropsychiatric Disease-Associated Gene cacna1c Mediates Survival of Young Hippocampal Neurons Primary blast injury causes cognitive impairments and hippocampal circuit alterations.Isolated Primary Blast Inhibits Long-Term Potentiation in Organotypic Hippocampal Slice Cultures.Cacna1c: Protecting young hippocampal neurons in the adult brain.Cis P-tau is induced in clinical and preclinical brain injury and contributes to post-injury sequelae.Pharmacological augmentation of nicotinamide phosphoribosyltransferase (NAMPT) protects against paclitaxel-induced peripheral neuropathy.Prevention of vincristine-induced peripheral neuropathy by genetic deletion of SARM1 in mice.Beneficial Effects of Delayed P7C3-A20 Treatment After Transient MCAO in Rats.Phosphodiesterase-4 inhibition restored hippocampal long term potentiation after primary blast.Neuroprotective Efficacy of an Aminopropyl Carbazole Derivative P7C3-A20 in Ischemic Stroke.High-Content Microfluidic Screening Platform Used To Identify σ2R/Tmem97 Binding Ligands that Reduce Age-Dependent Neurodegeneration in C. elegans SC_APP Model.Protective effect of P7C3 on retinal ganglion cells from optic nerve injury.Targeting NAD+ in Metabolic Disease: New Insights Into an Old Molecule.Ca2+-Binding Protein 1 Regulates Hippocampal-dependent Memory and Synaptic Plasticity.Acute vitreoretinal trauma and inflammation after traumatic brain injury in mice.Phosphatidylserine is a marker for axonal debris engulfment but its exposure can be decoupled from degeneration Neuroprotective efficacy of P7C3 compounds in primate hippocampus P7C3 Inhibits LPS-Induced Microglial Activation to Protect Dopaminergic Neurons Against Inflammatory Factor-Induced Cell Death and

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P7C3 neuroprotective chemicals block axonal degeneration and preserve function after traumatic brain injury

http://www.wikidata.org/entity/Q34389585

description

2014 nî lūn-bûn

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2014 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի սեպտեմբերին հրատարակված գիտական հոդված

@hy

2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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name

P7C3 neuroprotective chemicals ...... n after traumatic brain injury

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P7C3 neuroprotective chemicals ...... n after traumatic brain injury

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P7C3 neuroprotective chemicals ...... n after traumatic brain injury

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P7C3 neuroprotective chemicals ...... n after traumatic brain injury

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P7C3 neuroprotective chemicals ...... n after traumatic brain injury

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P7C3 neuroprotective chemicals ...... n after traumatic brain injury

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P7C3 neuroprotective chemicals ...... n after traumatic brain injury

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P7C3 neuroprotective chemicals ...... n after traumatic brain injury

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J.CELREP.2014.08.030

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P1476

P7C3 neuroprotective chemicals ...... n after traumatic brain injury

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Aaron C Katzman

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Alexander G Bassuk

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Andrew A Pieper

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Cassie Wassink

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Héctor De Jesús-Cortés

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Jacinth Naidoo

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Jaymie R Voorhees

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Jeremiah K Britt

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Jianqiang Shao

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Joseph M Ready

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P2860

Suppression of acute proinflammatory cytokine and chemokine upregulation by post-injury administration of a novel small molecule improves long-term neurologic outcome in a mouse model of traumatic brain injury

Chronic traumatic encephalopathy in blast-exposed military veterans and a blast neurotrauma mouse model.

Discovery of a proneurogenic, neuroprotective chemical

A local mechanism mediates NAD-dependent protection of axon degeneration

Fluorescence imaging of blood-brain barrier disruption

Stimulation of nicotinamide adenine dinucleotide biosynthetic pathways delays axonal degeneration after axotomy

Increased nuclear NAD biosynthesis and SIRT1 activation prevent axonal degeneration

Neuroprotective efficacy of aminopropyl carbazoles in a mouse model of amyotrophic lateral sclerosis.

Neuroprotective efficacy of aminopropyl carbazoles in a mouse model of Parkinson disease

Early Neurodegeneration after Hypoxia-Ischemia in Neonatal Rat Is Necrosis while Delayed Neuronal Death Is Apoptosis.

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1731-1740

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scholarly article

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10.1016/J.CELREP.2014.08.030

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6

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8

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Matthew M Harper

Elizabeth A Newell

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2014-09-15T00:00:00Z

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265649732

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25220467

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traumatic brain injury

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4206693

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