H3K4 mono- and di-methyltransferase MLL4 is required for enhancer activation during cell differentiation. - Wikidata - awgldk - TriplyDB

H3K4 mono- and di-methyltransferase MLL4 is required for enhancer activation during cell differentiation.

H3K4 mono- and di-methyltransferase MLL4 is required for enhancer activation during cell differentiation.

http://www.wikidata.org/entity/Q34394362

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Molecular control of activation and priming in macrophages The epigenetic basis of diffuse large B-cell lymphoma Ex uno plures: molecular designs for embryonic pluripotency Hijacked in cancer: the KMT2 (MLL) family of methyltransferases The selection and function of cell type-specific enhancers A PTIP-PA1 subcomplex promotes transcription for IgH class switching independently from the associated MLL3/MLL4 methyltransferase complex Epigenetic regulation in the inner ear and its potential roles in development, protection, and regeneration.Lsh/HELLS regulates self-renewal/proliferation of neural stem/progenitor cells.Transcriptional and epigenetic regulation of PPARγ expression during adipogenesis.A non-active-site SET domain surface crucial for the interaction of MLL1 and the RbBP5/Ash2L heterodimer within MLL family core complexes MLL3/MLL4 are required for CBP/p300 binding on enhancers and super-enhancer formation in brown adipogenesis.PPARγ and the global map of adipogenesis and beyond.Critical role of histone demethylase RBP2 in human gastric cancer angiogenesis.Yorkie promotes transcription by recruiting a histone methyltransferase complex Gcn5 and PCAF regulate PPARγ and Prdm16 expression to facilitate brown adipogenesis Epigenomics of macrophages.MLL3 is a haploinsufficient 7q tumor suppressor in acute myeloid leukemia.Primordial germ cell specification: a context-dependent cellular differentiation event [corrected].The histone lysine methyltransferase KMT2D sustains a gene expression program that represses B cell lymphoma development.Disruption of KMT2D perturbs germinal center B cell development and promotes lymphomagenesis.KMT2D regulates specific programs in heart development via histone H3 lysine 4 di-methylation.Transcriptional and epigenetic control of brown and beige adipose cell fate and function.Enhancer priming by H3K4 methyltransferase MLL4 controls cell fate transition.Systematic Analysis of Known and Candidate Lysine Demethylases in the Regulation of Myoblast Differentiation.H3K4 Methyltransferase Activity Is Required for MLL4 Protein Stability.Mll3 and Mll4 Facilitate Enhancer RNA Synthesis and Transcription from Promoters Independently of H3K4 Monomethylation.Gcn5 and PCAF negatively regulate interferon-β production through HAT-independent inhibition of TBK1.TP53 engagement with the genome occurs in distinct local chromatin environments via pioneer factor activity DNA-damage-induced differentiation of leukaemic cells as an anti-cancer barrier.X-ray phase-contrast tomography with a compact laser-driven synchrotron source.De Novo Reconstruction of Adipose Tissue Transcriptomes Reveals Long Non-coding RNA Regulators of Brown Adipocyte Development.A protocol of histone modification-based mechanistic study of acupuncture in patients with stable angina pectoris.Establishing pluripotency in early development.Epigenetic priming of enhancers predicts developmental competence of hESC-derived endodermal lineage intermediates.Molecular Profiling of a Rare Rosette-Forming Glioneuronal Tumor Arising in the Spinal Cord Disrupted intricacy of histone H3K4 methylation in neurodevelopmental disorders.Paternal H3K4 methylation is required for minor zygotic gene activation and early mouse embryonic development Replication fork stability confers chemoresistance in BRCA-deficient cells RAP1-mediated MEK/ERK pathway defects in Kabuki syndrome.Long Noncoding RNA ADINR Regulates Adipogenesis by Transcriptionally Activating C/EBPα

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H3K4 mono- and di-methyltransferase MLL4 is required for enhancer activation during cell differentiation.

http://www.wikidata.org/entity/Q34394362

description

2013 nî lūn-bûn

@nan

2013 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2013 թվականի դեկտեմբերին հրատարակված գիտական հոդված

@hy

2013年の論文

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2013年論文

@yue

2013年論文

@zh-hant

2013年論文

@zh-hk

2013年論文

@zh-mo

2013年論文

@zh-tw

2013年论文

@wuu

name

H3K4 mono- and di-methyltransf ...... n during cell differentiation.

@ast

H3K4 mono- and di-methyltransf ...... n during cell differentiation.

@en

H3K4 mono- and di-methyltransf ...... n during cell differentiation.

@nl

type

label

H3K4 mono- and di-methyltransf ...... n during cell differentiation.

@ast

H3K4 mono- and di-methyltransf ...... n during cell differentiation.

@en

H3K4 mono- and di-methyltransf ...... n during cell differentiation.

@nl

prefLabel

H3K4 mono- and di-methyltransf ...... n during cell differentiation.

@ast

H3K4 mono- and di-methyltransf ...... n during cell differentiation.

@en

H3K4 mono- and di-methyltransf ...... n during cell differentiation.

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H3K4 mono- and di-methyltransf ...... n during cell differentiation.

@en

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Anne Baldridge

http://www.w3.org/2001/XMLSchema#string

Chaochen Wang

http://www.w3.org/2001/XMLSchema#string

Lenan Zhuang

http://www.w3.org/2001/XMLSchema#string

Lifeng Wang

http://www.w3.org/2001/XMLSchema#string

Shiliyang Xu

http://www.w3.org/2001/XMLSchema#string

Weiqun Peng

http://www.w3.org/2001/XMLSchema#string

Xuesong Feng

http://www.w3.org/2001/XMLSchema#string

Young-Wook Cho

http://www.w3.org/2001/XMLSchema#string

P2860

MyoD1: a nuclear phosphoprotein requiring a Myc homology region to convert fibroblasts to myoblasts

Transcription coactivator TRAP220 is required for PPAR gamma 2-stimulated adipogenesis

PTIP associates with MLL3- and MLL4-containing histone H3 lysine 4 methyltransferase complex

SET1 and p300 act synergistically, through coupled histone modifications, in transcriptional activation by p53

Simple combinations of lineage-determining transcription factors prime cis-regulatory elements required for macrophage and B cell identities

Dissecting the genomic complexity underlying medulloblastoma

Exome sequencing identifies MLL2 mutations as a cause of Kabuki syndrome

Histone H3K27ac separates active from poised enhancers and predicts developmental state

Frequent mutation of histone-modifying genes in non-Hodgkin lymphoma

FoxA1 translates epigenetic signatures into enhancer-driven lineage-specific transcription

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e01503

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scholarly article

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10.7554/ELIFE.01503

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2

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2013-12-24T00:00:00Z

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24368734

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3869375

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