Krüppel-like factor 4, a tumor suppressor in hepatocellular carcinoma cells reverts epithelial mesenchymal transition by suppressing slug expression. - Wikidata - awgldk - TriplyDB

Krüppel-like factor 4, a tumor suppressor in hepatocellular carcinoma cells reverts epithelial mesenchymal transition by suppressing slug expression.

Krüppel-like factor 4, a tumor suppressor in hepatocellular carcinoma cells reverts epithelial mesenchymal transition by suppressing slug expression.

http://www.wikidata.org/entity/Q34399237

about

MiR-7 promotes epithelial cell transformation by targeting the tumor suppressor KLF4 KLF4 Plays an Essential Role in Corneal Epithelial Homeostasis by Promoting Epithelial Cell Fate and Suppressing Epithelial-Mesenchymal Transition.Krüppel-like factor 4 mediates lysophosphatidic acid-stimulated migration and proliferation of PC3M prostate cancer cells.Regulation of EMT by KLF4 in gastrointestinal cancer.Doxycycline inducible Krüppel-like factor 4 lentiviral vector mediates mesenchymal to epithelial transition in ovarian cancer cells.Heterogeneity of osteosarcoma cell lines led to variable responses in reprogramming Krüppel-like factor 4 induces apoptosis and inhibits tumorigenic progression in SK-BR-3 breast cancer cells.Suppression of epithelial-mesenchymal transition in hepatocellular carcinoma cells by Krüppel-like factor 4.Epigenetic signatures of alcohol abuse and hepatitis infection during human hepatocarcinogenesis.Attenuation of krüppel-like factor 4 facilitates carcinogenesis by inducing g1/s phase arrest in clear cell renal cell carcinoma Oncogenesis and induced pluripotency - commonalities of signalling pathways Slug contributes to cancer progression by direct regulation of ERα signaling pathway KLF4 Promotes Angiogenesis by Activating VEGF Signaling in Human Retinal Microvascular Endothelial Cells.Spatiotemporally Regulated Ablation of Klf4 in Adult Mouse Corneal Epithelial Cells Results in Altered Epithelial Cell Identity and Disrupted Homeostasis.F-box protein FBXO22 mediates polyubiquitination and degradation of KLF4 to promote hepatocellular carcinoma progression.Insulin-like growth factor-independent insulin-like growth factor binding protein 3 promotes cell migration and lymph node metastasis of oral squamous cell carcinoma cells by requirement of integrin β1.Basal/HER2 breast carcinomas: integrating molecular taxonomy with cancer stem cell dynamics to predict primary resistance to trastuzumab (Herceptin).Krüppel-like factor 2 promotes cell proliferation in hepatocellular carcinoma through up-regulation of c-myc.Switching metal ion coordination and DNA Recognition in a Tandem CCHHC-type zinc finger peptide p16 Stimulates CDC42-dependent migration of hepatocellular carcinoma cells Peroxisome Proliferator-Activated Receptor γ Expression Is Inversely Associated with Macroscopic Vascular Invasion in Human Hepatocellular Carcinoma.Differentiated epithelial- and mesenchymal-like phenotypes in subcutaneous mouse xenografts using diffusion weighted-magnetic resonance imaging.Endothelial cell colony forming units derived from malignant breast diseases are resistant to tumor necrosis factor-α-induced apoptosis Down-regulation of Krüppel-like factor-4 by microRNA-135a-5p promotes proliferation and metastasis in hepatocellular carcinoma by transforming growth factor-β1.Klf4 inhibits tumor growth and metastasis by targeting microRNA-31 in human hepatocellular carcinoma MircoRNA-145 promotes activation of hepatic stellate cells via targeting krüppel-like factor 4 Overexpression of KLF4 promotes cell senescence through microRNA-203-survivin-p21 pathway.Cellular reprogramming and hepatocellular carcinoma development.Krüppel-like factors in cancer progression: three fingers on the steering wheel.Role of Krüppel-like factors in cancer stem cells.Krüppel-like factors in hepatocellular carcinoma.Tumour cell-derived WNT5B modulates in vitro lymphangiogenesis via induction of partial endothelial-mesenchymal transition of lymphatic endothelial cells.Krüppel-Like Factor 4 Inhibits the Transforming Growth Factor-β1-Promoted Epithelial-to-Mesenchymal Transition via Downregulating Plasminogen Activator Inhibitor-1 in Lung Epithelial Cells Krüppel-like Factor 4 Blocks Hepatocellular Carcinoma Dedifferentiation and Progression through Activation of Hepatocyte Nuclear Factor-6.Current status in cancer cell reprogramming and its clinical implications.Loss of KLF4 and consequential downregulation of Smad7 exacerbate oncogenic TGF-β signaling in and promote progression of hepatocellular carcinoma.MicroRNA-9 enhances migration and invasion through KLF17 in hepatocellular carcinoma.SP and KLF Transcription Factors in Digestive Physiology and Diseases.Activation of pluripotent genes in hepatic progenitor cells in the transition of nonalcoholic steatohepatitis to pre-malignant lesions.Krüppel-like Transcription Factor KLF10 Suppresses TGFβ-Induced Epithelial-to-Mesenchymal Transition via a Negative Feedback Mechanism

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Krüppel-like factor 4, a tumor suppressor in hepatocellular carcinoma cells reverts epithelial mesenchymal transition by suppressing slug expression.

http://www.wikidata.org/entity/Q34399237

description

2012 nî lūn-bûn

@nan

2012 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2012 թվականի օգոստոսին հրատարակված գիտական հոդված

@hy

2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

@wuu

name

Krüppel-like factor 4, a tumor ...... y suppressing slug expression.

@ast

Krüppel-like factor 4, a tumor ...... y suppressing slug expression.

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Krüppel-like factor 4, a tumor ...... y suppressing slug expression.

@nl

type

label

Krüppel-like factor 4, a tumor ...... y suppressing slug expression.

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Krüppel-like factor 4, a tumor ...... y suppressing slug expression.

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Krüppel-like factor 4, a tumor ...... y suppressing slug expression.

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prefLabel

Krüppel-like factor 4, a tumor ...... y suppressing slug expression.

@ast

Krüppel-like factor 4, a tumor ...... y suppressing slug expression.

@en

Krüppel-like factor 4, a tumor ...... y suppressing slug expression.

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Krüppel-like factor 4, a tumor ...... y suppressing slug expression.

@en

P2093

Brian C Lewis

http://www.w3.org/2001/XMLSchema#string

Hsiao-Chien Chu

http://www.w3.org/2001/XMLSchema#string

Ya-Wen Chen

http://www.w3.org/2001/XMLSchema#string

Yi-Chen Yen

http://www.w3.org/2001/XMLSchema#string

Ze-Shiang Lin

http://www.w3.org/2001/XMLSchema#string

P2860

Global cancer statistics

Identification and characterization of a gene encoding a gut-enriched Krüppel-like factor expressed during growth arrest

Prognostic value and function of KLF4 in prostate cancer: RNAa and vector-mediated overexpression identify KLF4 as an inhibitor of tumor cell growth and migration

The diverse functions of Krüppel-like factors 4 and 5 in epithelial biology and pathobiology

Transcriptional profiling of Krüppel-like factor 4 reveals a function in cell cycle regulation and epithelial differentiation

Overexpression of Krüppel-like factor 4 in the human colon cancer cell line RKO leads to reduced tumorigenecity

The gut-enriched Kruppel-like factor (Kruppel-like factor 4) mediates the transactivating effect of p53 on the p21WAF1/Cip1 promoter

Sp1- and Krüppel-like transcription factors

Genes involved in viral carcinogenesis and tumor initiation in hepatitis C virus-induced hepatocellular carcinoma

Epithelial-mesenchymal transitions in tumour progression

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scholarly article

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10.1371/JOURNAL.PONE.0043593

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8

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230769797

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22937066

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3427336

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