Abl kinases are required for invadopodia formation and chemokine-induced invasion. - Wikidata - awgldk - TriplyDB

Abl kinases are required for invadopodia formation and chemokine-induced invasion.

Abl kinases are required for invadopodia formation and chemokine-induced invasion.

http://www.wikidata.org/entity/Q34401096

about

Abl family kinases regulate endothelial barrier function in vitro and in mice Multifunctional Abl kinases in health and disease Tumor and stromal-based contributions to head and neck squamous cell carcinoma invasion Abl Kinases Regulate HGF/Met Signaling Required for Epithelial Cell Scattering, Tubulogenesis and Motility Role of ABL family kinases in cancer: from leukaemia to solid tumours Abl family kinases regulate FcγR-mediated phagocytosis in murine macrophages The oleocanthal-based homovanillyl sinapate as a novel c-Met inhibitor Abelson kinase acts as a robust, multifunctional scaffold in regulating embryonic morphogenesis.The NESH/Abi-3-based WAVE2 complex is functionally distinct from the Abi-1-based WAVE2 complex.Crk at the quarter century mark: perspectives in signaling and cancer.Met receptor tyrosine kinase signals through a cortactin-Gab1 scaffold complex, to mediate invadopodia Abl family kinases modulate T cell-mediated inflammation and chemokine-induced migration through the adaptor HEF1 and the GTPase Rap1 Nck2 promotes human melanoma cell proliferation, migration and invasion in vitro and primary melanoma-derived tumor growth in vivo Expression and Y435-phosphorylation of Abelson interactor 1 (Abi1) promotes tumour cell adhesion, extracellular matrix degradation and invasion by colorectal carcinoma cells.Invadopodia and basement membrane invasion in vivo.Targeting ABL1-mediated oxidative stress adaptation in fumarate hydratase-deficient cancer.Arg/Abl2 promotes invasion and attenuates proliferation of breast cancer in vivo.The Emerging Role of ABL Kinases in Solid Tumors.Protein tyrosine phosphatase SHP2 promotes invadopodia formation through suppression of Rho signaling Activation of abl family kinases in solid tumors.LPA-mediated migration of ovarian cancer cells involves translocalization of Gαi2 to invadopodia and association with Src and β-pix Signaling inputs to invadopodia and podosomes ABL kinases promote breast cancer osteolytic metastasis by modulating tumor-bone interactions through TAZ and STAT5 signaling.Inhibition of MMP14 potentiates the therapeutic effect of temozolomide and radiation in gliomas.Ableson kinases negatively regulate invadopodia function and invasion in head and neck squamous cell carcinoma by inhibiting an HB-EGF autocrine loop.Invading one step at a time: the role of invadopodia in tumor metastasis.Coordination of signalling networks and tumorigenic properties by ABL in glioblastoma cells.Regulation of the actin cytoskeleton in Helicobacter pylori-induced migration and invasive growth of gastric epithelial cells.Cortactin in cell migration and cancer at a glance Domain structure and function of matrix metalloprotease 23 (MMP23): role in potassium channel trafficking.Palladin regulation of the actin structures needed for cancer invasion.Arg kinase regulates epithelial cell polarity by targeting β1-integrin and small GTPase pathways.ABL2 suppresses FLT3-ITD-induced cell proliferation through negative regulation of AKT signaling.Cell adhesion and invasion mechanisms that guide developing axons Abl kinase regulation by BRAF/ERK and cooperation with Akt in melanoma.Membrane proteome analysis of glioblastoma cell invasion.Enterolobium contortisiliquum trypsin inhibitor (EcTI), a plant proteinase inhibitor, decreases in vitro cell adhesion and invasion by inhibition of Src protein-focal adhesion kinase (FAK) signaling pathways.c-Abl and Arg are activated in human primary melanomas, promote melanoma cell invasion via distinct pathways, and drive metastatic progression.Podocalyxin-like 1 promotes invadopodia formation and metastasis through activation of Rac1/Cdc42/cortactin signaling in breast cancer cells.Sos1 Regulates Macrophage Podosome Assembly and Macrophage Invasive Capacity.

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P2860

Abl kinases are required for invadopodia formation and chemokine-induced invasion.

http://www.wikidata.org/entity/Q34401096

description

2010 nî lūn-bûn

@nan

2010 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի հոտեմբերին հրատարակված գիտական հոդված

@hy

2010年の論文

@ja

2010年論文

@yue

2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

@zh-tw

2010年论文

@wuu

name

Abl kinases are required for invadopodia formation and chemokine-induced invasion.

@ast

Abl kinases are required for invadopodia formation and chemokine-induced invasion.

@en

Abl kinases are required for invadopodia formation and chemokine-induced invasion.

@nl

type

label

Abl kinases are required for invadopodia formation and chemokine-induced invasion.

@ast

Abl kinases are required for invadopodia formation and chemokine-induced invasion.

@en

Abl kinases are required for invadopodia formation and chemokine-induced invasion.

@nl

prefLabel

Abl kinases are required for invadopodia formation and chemokine-induced invasion.

@ast

Abl kinases are required for invadopodia formation and chemokine-induced invasion.

@en

Abl kinases are required for invadopodia formation and chemokine-induced invasion.

@nl

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JBC.M110.147330

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Journal of Biological Chemistry

P1476

Abl kinases are required for invadopodia formation and chemokine-induced invasion.

@en

P2093

Ann Marie Pendergast

http://www.w3.org/2001/XMLSchema#string

Emileigh K Greuber

http://www.w3.org/2001/XMLSchema#string

Gouri Yogalingam

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Pameeka S Smith-Pearson

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P2860

Involvement of chemokine receptors in breast cancer metastasis

Abi-2, a novel SH3-containing protein interacts with the c-Abl tyrosine kinase and modulates c-Abl transforming activity

Latent bone metastasis in breast cancer tied to Src-dependent survival signals

Wiskott-Aldrich syndrome protein regulates podosomes in primary human macrophages

Abelson-interactor-1 promotes WAVE2 membrane translocation and Abelson-mediated tyrosine phosphorylation required for WAVE2 activation

The Abl family kinases: mechanisms of regulation and signaling

Essential role of neural Wiskott-Aldrich syndrome protein in podosome formation and degradation of extracellular matrix in src-transformed fibroblasts

Assembly and biological role of podosomes and invadopodia

CXCR4 regulates growth of both primary and metastatic breast cancer

Cortactin is an essential regulator of matrix metalloproteinase secretion and extracellular matrix degradation in invadopodia

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40201-40211

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scholarly article

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10.1074/JBC.M110.147330

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51

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285

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2010-10-11T00:00:00Z

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3001002

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