Chronic intermittent hypoxia alters density of aminergic terminals and receptors in the hypoglossal motor nucleus.
about
Abnormal myelin and axonal integrity in recently diagnosed patients with obstructive sleep apnea.Rats subjected to chronic-intermittent hypoxia have increased density of noradrenergic terminals in the trigeminal sensory and motor nuclei.Sleep-wake control of the upper airway by noradrenergic neurons, with and without intermittent hypoxia.Glucoregulatory consequences and cardiorespiratory parameters in rats exposed to chronic-intermittent hypoxia: effects of the duration of exposure and losartan.The effect of tongue exercise on serotonergic input to the hypoglossal nucleus in young and old rats.Effect of chronic intermittent hypoxia on noradrenergic activation of hypoglossal motoneuronsAntagonism of orexin receptors in the posterior hypothalamus reduces hypoglossal and cardiorespiratory excitation from the perifornical hypothalamus.Reduced c-Fos expression in medullary catecholaminergic neurons in rats 20 h after exposure to chronic intermittent hypoxia.Loss of motoneurons in the ventral compartment of the rat hypoglossal nucleus following early postnatal exposure to alcohol.Acute systemic hypoxia activates hypothalamic paraventricular nucleus-projecting catecholaminergic neurons in the caudal ventrolateral medulla.Central and peripheral factors contributing to obstructive sleep apneas.Neural Control of the Upper Airway: Respiratory and State-Dependent Mechanisms.Chronic intermittent hypoxia affects endogenous serotonergic inputs and expression of synaptic proteins in rat hypoglossal nucleus.Update in sleep medicine 2010.Catecholaminergic A1/C1 neurons contribute to the maintenance of upper airway muscle tone but may not participate in NREM sleep-related depression of these muscles.α1- and α2-adrenergic receptors in the retrotrapezoid nucleus differentially regulate breathing in anesthetized adult rats.AJRCCM: 100-Year Anniversary. Sleep-Disordered Breathing: Still the New Kid on the Block.Hypoglossal motoneurons are endogenously activated by serotonin during the active period of circadian cycle.Activation of α1-adrenoceptors facilitates excitatory inputs to medullary airway vagal preganglionic neurons.Intermittent hypercapnic hypoxia during sleep does not induce ventilatory long-term facilitation in healthy males.Effect of Gender on Chronic Intermittent Hypoxic Fosb Expression in Cardiorespiratory-Related Brain Structures in Mice.Neuroanatomical Basis of State-Dependent Activity of Upper Airway Muscles
P2860
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P2860
Chronic intermittent hypoxia alters density of aminergic terminals and receptors in the hypoglossal motor nucleus.
description
2010 nî lūn-bûn
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2010 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2010年の論文
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2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Chronic intermittent hypoxia a ...... the hypoglossal motor nucleus.
@ast
Chronic intermittent hypoxia a ...... the hypoglossal motor nucleus.
@en
Chronic intermittent hypoxia a ...... the hypoglossal motor nucleus.
@nl
type
label
Chronic intermittent hypoxia a ...... the hypoglossal motor nucleus.
@ast
Chronic intermittent hypoxia a ...... the hypoglossal motor nucleus.
@en
Chronic intermittent hypoxia a ...... the hypoglossal motor nucleus.
@nl
prefLabel
Chronic intermittent hypoxia a ...... the hypoglossal motor nucleus.
@ast
Chronic intermittent hypoxia a ...... the hypoglossal motor nucleus.
@en
Chronic intermittent hypoxia a ...... the hypoglossal motor nucleus.
@nl
P2093
P2860
P1476
Chronic intermittent hypoxia a ...... the hypoglossal motor nucleus.
@en
P2093
Andrea Price
Irma Rukhadze
Kate E Benincasa
Leszek Kubin
Victor B Fenik
P2860
P304
P356
10.1164/RCCM.200912-1884OC
P407
P50
P577
2010-07-09T00:00:00Z