about
CCN3 suppresses mitogenic signalling and reinstates growth control mechanisms in Chronic Myeloid LeukaemiaIGF2 is critical for tumorigenesis by synovial sarcoma oncoprotein SYT-SSX1Differential signaling networks of Bcr-Abl p210 and p190 kinases in leukemia cells defined by functional proteomics.Array-based genomic resequencing of human leukemia.Dissecting the molecular mechanism of chronic myelogenous leukemia using murine models.Enforced differentiation of Dnmt3a-null bone marrow leads to failure with c-Kit mutations driving leukemic transformationThe molecular mechanism of chronic myelogenous leukemia and its therapeutic implications: studies in a murine model.The molecular basis of myeloid malignancies.High affinity molecules disrupting GRB2 protein complexes as a therapeutic strategy for chronic myelogenous leukaemia.The role of DNA breaks in genomic instability and tumorigenesis.Split-signal FISH for detection of chromosome aberrations in acute lymphoblastic leukemia.Age-related defects in B lymphopoiesis underlie the myeloid dominance of adult leukemiaPhiladelphia positive acute lymphoblastic leukaemia of childhood.Sustained suppression of Bcr-Abl-driven lymphoid leukemia by microRNA mimics.Recent advances in targeted therapy of human myelogenous leukaemia.Role of Dok-1 and Dok-2 in leukemia suppression.Re-evaluating the role of BCR/ABL in chronic myelogenous leukemiaExpression of BCR/ABL p210 from a knockin allele enhances bone marrow engraftment without inducing neoplasiaTargeted therapy of chronic myeloid leukemia.Mouse models as tools to understand and study BCR-ABL1 diseases.Kinase-inhibitor-insensitive cancer stem cells in chronic myeloid leukemia.Novel working hypothesis for pathogenesis of hematological malignancies: combination of mutations-induced cellular phenotypes determines the disease (cMIP-DD).Mechanisms determining the fate of hematopoietic stem cells.CCN3: a key growth regulator in Chronic Myeloid Leukaemia.Role of Dok-1 and Dok-2 in myeloid homeostasis and suppression of leukemia.Establishment and phenotypic characterization of human U937 cells with inducible P210 BCR/ABL expression reveals upregulation of CEACAM1 (CD66a).Crk-associated substrate lymphocyte type regulates myeloid cell motility and suppresses the progression of leukemia induced by p210Bcr/Abl.
P2860
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P2860
description
2001 nî lūn-bûn
@nan
2001 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
name
Modeling Philadelphia chromosome positive leukemias.
@ast
Modeling Philadelphia chromosome positive leukemias.
@en
Modeling Philadelphia chromosome positive leukemias.
@nl
type
label
Modeling Philadelphia chromosome positive leukemias.
@ast
Modeling Philadelphia chromosome positive leukemias.
@en
Modeling Philadelphia chromosome positive leukemias.
@nl
prefLabel
Modeling Philadelphia chromosome positive leukemias.
@ast
Modeling Philadelphia chromosome positive leukemias.
@en
Modeling Philadelphia chromosome positive leukemias.
@nl
P2860
P356
P1433
P1476
Modeling Philadelphia chromosome positive leukemias.
@en
P2860
P2888
P304
P356
10.1038/SJ.ONC.1204638
P407
P577
2001-09-01T00:00:00Z
P6179
1022494522