Potentiation of NMDA receptor-dependent cell responses by extracellular high mobility group box 1 protein. - Wikidata - awgldk - TriplyDB

Potentiation of NMDA receptor-dependent cell responses by extracellular high mobility group box 1 protein.

Potentiation of NMDA receptor-dependent cell responses by extracellular high mobility group box 1 protein.

http://www.wikidata.org/entity/Q34406782

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The Emerging Role of HMGB1 in Neuropathic Pain: A Potential Therapeutic Target for Neuroinflammation NK Cells, Tumor Cell Transition, and Tumor Progression in Solid Malignancies: New Hints for NK-Based Immunotherapy?The Role of HMGB1 Signaling Pathway in the Development and Progression of Hepatocellular Carcinoma: A Review Emerging roles for HMGB1 protein in immunity, inflammation, and cancer Extracellular high-mobility group box 1 protein (HMGB1) as a mediator of persistent pain Combinations of Ashwagandha leaf extracts protect brain-derived cells against oxidative stress and induce differentiation HMGB1 in health and disease.Interaction between calpain-1 and HSP90: new insights into the regulation of localization and activity of the protease.Physiological Roles of Calpain 1 Associated to Multiprotein NMDA Receptor Complex Does brain inflammation mediate pathological outcomes in epilepsy?Modulation of neuronal excitability by immune mediators in epilepsy.High mobility group box protein-1 in wound repair Natural Killer (NK)/melanoma cell interaction induces NK-mediated release of chemotactic High Mobility Group Box-1 (HMGB1) capable of amplifying NK cell recruitment.Response to “HMGB1 Mediates Cognitive Impairment in Sepsis Survivors”.

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Potentiation of NMDA receptor-dependent cell responses by extracellular high mobility group box 1 protein.

http://www.wikidata.org/entity/Q34406782

description

2012 nî lūn-bûn

@nan

2012 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2012 թվականի օգոստոսին հրատարակված գիտական հոդված

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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name

Potentiation of NMDA receptor- ...... mobility group box 1 protein.

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Potentiation of NMDA receptor- ...... mobility group box 1 protein.

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Potentiation of NMDA receptor- ...... mobility group box 1 protein.

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Potentiation of NMDA receptor- ...... mobility group box 1 protein.

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Potentiation of NMDA receptor- ...... mobility group box 1 protein.

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Potentiation of NMDA receptor- ...... mobility group box 1 protein.

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Potentiation of NMDA receptor- ...... mobility group box 1 protein.

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Potentiation of NMDA receptor- ...... mobility group box 1 protein.

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Potentiation of NMDA receptor- ...... mobility group box 1 protein.

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JOURNAL.PONE.0044518

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Potentiation of NMDA receptor- ...... mobility group box 1 protein.

@en

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Bianca Sparatore

http://www.w3.org/2001/XMLSchema#string

Chiara Cervetto

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Daniela Frattaroli

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Edon Melloni

http://www.w3.org/2001/XMLSchema#string

Franca Salamino

http://www.w3.org/2001/XMLSchema#string

Guido Maura

http://www.w3.org/2001/XMLSchema#string

Manuela Marcoli

http://www.w3.org/2001/XMLSchema#string

Marco Pedrazzi

http://www.w3.org/2001/XMLSchema#string

Mauro Patrone

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Monica Averna

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P2860

Receptor for advanced glycation end products (RAGE)-mediated neurite outgrowth and activation of NF-kappaB require the cytoplasmic domain of the receptor but different downstream signaling pathways

A critical cysteine is required for HMGB1 binding to Toll-like receptor 4 and activation of macrophage cytokine release

A nomenclature for ligand-gated ion channels

Glutamate and neurotrophic factors in neuronal plasticity and disease

HMGB1 loves company

Mini-review: The nuclear protein HMGB1 as a proinflammatory mediator

Glutamate receptor ion channels: structure, regulation, and function

NMDA and HIV-1 coat protein, GP120, produce necrotic but not apoptotic cell death in human CHP100 neuroblastoma cultures via a mechanism involving calpain

Masquerader: high mobility group box-1 and cancer

Extracellular processing of amphoterin generates a peptide active on erythroleukaemia cell differentiation

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e44518

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scholarly article

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10.1371/JOURNAL.PONE.0044518

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8

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2012-08-31T00:00:00Z

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