Increased small intestinal apoptosis in coeliac disease. - Wikidata - awgldk - TriplyDB

Increased small intestinal apoptosis in coeliac disease.

Increased small intestinal apoptosis in coeliac disease.

http://www.wikidata.org/entity/Q34410636

about

Activation of caspases in intestinal villus epithelial cells of normal and nematode infected rats Permeability of human HT-29/B6 colonic epithelium as a function of apoptosis Parallels between pathogens and gluten peptides in celiac sprue Altered gene expression in highly purified enterocytes from patients with active coeliac disease.Intectin, a novel small intestine-specific glycosylphosphatidylinositol-anchored protein, accelerates apoptosis of intestinal epithelial cells.Attenuation of apoptosis in enterocytes by blockade of potassium channels.Broad MICA/B expression in the small bowel mucosa: a link between cellular stress and celiac disease.Immunohistochemical analysis of candidate gene product expression in the duodenal epithelium of children with coeliac sprue Nodular regenerative hyperplasia of the liver and coeliac disease: potential role of IgA anticardiolipin antibody.MeDiA: Mean Distance Association and Its Applications in Nonlinear Gene Set Analysis The Interaction among Microbiota, Immunity, and Genetic and Dietary Factors Is the Condicio Sine Qua Non Celiac Disease Can Develop.Genes involved in muscle contractility and nutrient signaling pathways within celiac disease risk loci show differential mRNA expression Actin reorganization as the molecular basis for the regulation of apoptosis in gastrointestinal epithelial cells.Regulation of apoptosis during homeostasis and disease in the intestinal epithelium.Significance of increased apoptosis and Bax expression in human small intestinal adenocarcinoma.Cell proliferation within small intestinal crypts is the principal driving force for cell migration on villi.Architectural and functional alterations of the small intestinal mucosa in classical Whipple's disease.Cyclic AMP activation of the extracellular signal-regulated kinases 1 and 2: implications for intestinal cell survival through the transient inhibition of apoptosis.Phosphatidylinositol 3-kinase-dependent pathways oppose Fas-induced apoptosis and limit chloride secretion in human intestinal epithelial cells. Implications for inflammatory diarrheal states.Susceptibility to FAS-induced apoptosis in human nontumoral enterocytes: role of costimulatory factors.Intestinal and Circulating MicroRNAs in Coeliac Disease.Defining the roles of perforin, Fas/FasL, and tumour necrosis factor alpha in T cell induced mucosal damage in the mouse intestine FAS engagement drives apoptosis of enterocytes of coeliac patients.Intraepithelial and lamina propria lymphocytes show distinct patterns of apoptosis whereas both populations are active in Fas based cytotoxicity in coeliac disease.Effect of fat feeding on pro-oxidant and anti-oxidant enzyme systems in rat intestine: possible role in the turnover of enterocytes Intestinal crypt cell apoptosis in murine acute graft versus host disease is mediated by tumour necrosis factor alpha and not by the FasL-Fas interaction: effect of pentoxifylline on the development of mucosal atrophy.Effect of ectopic expression of rat trefoil factor family 3 (intestinal trefoil factor) in the jejunum of transgenic mice.Collagen IV regulates Caco-2 migration and ERK activation via alpha1beta1- and alpha2beta1-integrin-dependent Src kinase activation.Intestinal parameters of oxidative imbalance in celiac adults with extraintestinal manifestations.Sphingolipid de novo biosynthesis is essential for intestine cell survival and barrier function.The role of corticosteroids in celiac disease.DNA fragmentation is a feature of cystic fibrosis epithelial cells: a disease with inappropriate apoptosis?Defective tight junctions in refractory celiac disease.Evaluation of the ultrastructure of the small intestine of hiv infected children by transmission and scanning electronic microscopy.Cytolytic mechanisms of intraepithelial lymphocytes in coeliac disease (CoD).Immunohistochemical analysis of coeliac mucosa following ingestion of oats.Gliadin intake alters the small intestinal mucosa in indomethacin-treated HLA-DQ8 transgenic mice

P2860

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P2860

Increased small intestinal apoptosis in coeliac disease.

http://www.wikidata.org/entity/Q34410636

description

1996 nî lūn-bûn

@nan

1996 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

1996 թվականի դեկտեմբերին հրատարակված գիտական հոդված

@hy

1996年の論文

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1996年論文

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1996年論文

@zh-hant

1996年論文

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1996年論文

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1996年論文

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1996年论文

@wuu

name

Increased small intestinal apoptosis in coeliac disease.

@ast

Increased small intestinal apoptosis in coeliac disease.

@en

Increased small intestinal apoptosis in coeliac disease.

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type

label

Increased small intestinal apoptosis in coeliac disease.

@ast

Increased small intestinal apoptosis in coeliac disease.

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Increased small intestinal apoptosis in coeliac disease.

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prefLabel

Increased small intestinal apoptosis in coeliac disease.

@ast

Increased small intestinal apoptosis in coeliac disease.

@en

Increased small intestinal apoptosis in coeliac disease.

@nl

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Increased small intestinal apoptosis in coeliac disease.

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Attia L

http://www.w3.org/2001/XMLSchema#string

Holt PR

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Moss SF

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Scholes JV

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Walters JR

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P2860

Identification of programmed cell death in situ via specific labeling of nuclear DNA fragmentation

Apoptosis, oncosis, and necrosis. An overview of cell death

Apoptosis in the pathogenesis and treatment of disease

Distinction of apoptotic and necrotic cell death by in situ labelling of fragmented DNA

The Fas death factor

Regulation of cell number in the mammalian gastrointestinal tract: the importance of apoptosis.

Gluten specific, HLA-DQ restricted T cells from coeliac mucosa produce cytokines with Th1 or Th0 profile dominated by interferon gamma.

Induction of gastric epithelial apoptosis by Helicobacter pylori

Cell kinetics in flat (avillous) mucosa of the human small intestine.

Review article: manipulation of cell death--the development of novel strategies for the treatment of gastrointestinal disease.

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