Viral replicative capacity is the primary determinant of lymphocytic choriomeningitis virus persistence and immunosuppression. - Wikidata - awgldk - TriplyDB

Viral replicative capacity is the primary determinant of lymphocytic choriomeningitis virus persistence and immunosuppression.

Viral replicative capacity is the primary determinant of lymphocytic choriomeningitis virus persistence and immunosuppression.

http://www.wikidata.org/entity/Q34411316

about

Type I interferon suppresses virus-specific B cell responses by modulating CD8(+) T cell differentiation Platelets support a protective immune response to LCMV by preventing splenic necrosis.Type I interferon is a therapeutic target for virus-induced lethal vascular damage.Evolution of recombinant lymphocytic choriomeningitis virus/Lassa virus in vivo highlights the importance of the GPC cytosolic tail in viral fitness.Point mutation in the glycoprotein of lymphocytic choriomeningitis virus is necessary for receptor binding, dendritic cell infection, and long-term persistence.The role of dendritic cells in viral persistence Arenavirus variations due to host-specific adaptation.Early virus-host interactions dictate the course of a persistent infection Protective efficacy of individual CD8+ T cell specificities in chronic viral infection Blockade of interferon Beta, but not interferon alpha, signaling controls persistent viral infection Arenavirus Z protein controls viral RNA synthesis by locking a polymerase-promoter complex.Plasmacytoid dendritic cells control T-cell response to chronic viral infection.Toll-like receptor 7 is required for effective adaptive immune responses that prevent persistent virus infection.Plasmacytoid dendritic cells are productively infected and activated through TLR-7 early after arenavirus infection A Jekyll and Hyde Profile: Type 1 Interferon Signaling Plays a Prominent Role in the Initiation and Maintenance of a Persistent Virus Infection.Pathogenic Old World arenaviruses inhibit TLR2/Mal-dependent proinflammatory cytokines in vitro Therapeutic blockade of transforming growth factor beta fails to promote clearance of a persistent viral infection.Superoxide Dismutase 1 Protects Hepatocytes from Type I Interferon-Driven Oxidative Damage Role of lymphocytic choriomeningitis virus (LCMV) in understanding viral immunology: past, present and future.Type I interferons: diversity of sources, production pathways and effects on immune responses.Timing and magnitude of type I interferon responses by distinct sensors impact CD8 T cell exhaustion and chronic viral infection.PD-1 promotes immune exhaustion by inducing antiviral T cell motility paralysis Persistent LCMV infection is controlled by blockade of type I interferon signaling.Spatiotemporal interplay of severe acute respiratory syndrome coronavirus and respiratory mucosal cells drives viral dissemination in rhesus macaques.Networking at the level of host immunity: immune cell interactions during persistent viral infections Chronic but not acute virus infection induces sustained expansion of myeloid suppressor cell numbers that inhibit viral-specific T cell immunity.Targeting virulence mechanisms for the prevention and therapy of arenaviral hemorrhagic fever.Human hemorrhagic Fever causing arenaviruses: molecular mechanisms contributing to virus virulence and disease pathogenesis.Characterization of the Glycoprotein Stable Signal Peptide in Mediating Pichinde Virus Replication and Virulence.Residues K465 and G467 within the Cytoplasmic Domain of GP2 Play a Critical Role in the Persistence of Lymphocytic Choriomeningitis Virus in Mice.Resistance of human plasmacytoid dendritic CAL-1 cells to infection with lymphocytic choriomeningitis virus (LCMV) is caused by restricted virus cell entry, which is overcome by contact of CAL-1 cells with LCMV-infected cells.Evolutionary analysis of Old World arenaviruses reveals a major adaptive contribution of the viral polymerase.Human Plasmacytoid Dendritic Cells Elicited Different Responses after Infection with Pathogenic and Nonpathogenic Junin Virus Strains.Identification of virulence determinants within the L genomic segment of the pichinde arenavirus.The Z proteins of pathogenic but not nonpathogenic arenaviruses inhibit RIG-I-like receptor-dependent interferon production.Characterization of host proteins interacting with the lymphocytic choriomeningitis virus L protein.The Role of Interferon in Persistent Viral Infection: Insights from Murine Norovirus.The Janus Face of Follicular T Helper Cells in Chronic Viral Infections.Ancient Evolution of Mammarenaviruses: Adaptation via Changes in the L Protein and No Evidence for Host-Virus Codivergence.Xapuri virus, a novel mammarenavirus: natural reassortment and increased diversity between New World viruses.

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Viral replicative capacity is the primary determinant of lymphocytic choriomeningitis virus persistence and immunosuppression.

http://www.wikidata.org/entity/Q34411316

description

2010 nî lūn-bûn

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2010 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Viral replicative capacity is ...... istence and immunosuppression.

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Viral replicative capacity is ...... istence and immunosuppression.

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Viral replicative capacity is ...... istence and immunosuppression.

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Viral replicative capacity is ...... istence and immunosuppression.

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Viral replicative capacity is ...... istence and immunosuppression.

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Viral replicative capacity is ...... istence and immunosuppression.

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Viral replicative capacity is ...... istence and immunosuppression.

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Viral replicative capacity is ...... istence and immunosuppression.

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Viral replicative capacity is ...... istence and immunosuppression.

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Viral replicative capacity is ...... sistence and immunosuppression

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P2093

Ahmed N Hegazy

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Daniel D Pinschewer

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Edit Horvath

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Lukas Flatz

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Max Löhning

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Susan Johnson

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P2860

Dual stem loops within the poliovirus internal ribosomal entry site control neurovirulence

NP and L proteins of lymphocytic choriomeningitis virus (LCMV) are sufficient for efficient transcription and replication of LCMV genomic RNA analogs

Viral immune evasion due to persistence of activated T cells without effector function

Interleukin-10 determines viral clearance or persistence in vivo

The N-Terminal Domain of the Arenavirus L Protein Is an RNA Endonuclease Essential in mRNA Transcription

Analysis of successful immune responses in persons infected with hepatitis C virus

Determinants of viral clearance and persistence during acute hepatitis C virus infection

Restoring function in exhausted CD8 T cells during chronic viral infection

Z/EG, a double reporter mouse line that expresses enhanced green fluorescent protein upon Cre-mediated excision

Immunology of hepatitis B virus and hepatitis C virus infection

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107

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Andreas Bergthaler

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49629454

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immunosuppression

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3003068

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