Inhibition by curcumin of multiple sites of the transforming growth factor-beta1 signalling pathway ameliorates the progression of liver fibrosis induced by carbon tetrachloride in rats - Wikidata - awgldk - TriplyDB

Inhibition by curcumin of multiple sites of the transforming growth factor-beta1 signalling pathway ameliorates the progression of liver fibrosis induced by carbon tetrachloride in rats

Inhibition by curcumin of multiple sites of the transforming growth factor-beta1 signalling pathway ameliorates the progression of liver fibrosis induced by carbon tetrachloride in rats

http://www.wikidata.org/entity/Q34414756

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Hepatoprotective Effects of Chinese Medicinal Herbs: A Focus on Anti-Inflammatory and Anti-Oxidative Activities Therapeutic targeting of liver inflammation and fibrosis by nanomedicine Curcumin inhibits transforming growth factor-β1-induced EMT via PPARγ pathway, not Smad pathway in renal tubular epithelial cells Nanotechnology applications for the therapy of liver fibrosis.Ginseng essence, a medicinal and edible herbal formulation, ameliorates carbon tetrachloride-induced oxidative stress and liver injury in rats.Paeonol inhibits hepatic fibrogenesis via disrupting nuclear factor-κB pathway in activated stellate cells: in vivo and in vitro studies.Turmeric extract and its active compound, curcumin, protect against chronic CCl4-induced liver damage by enhancing antioxidation.Curcumin combined with FAPαc vaccine elicits effective antitumor response by targeting indolamine-2,3-dioxygenase and inhibiting EMT induced by TNF-α in melanoma.MicroRNA-17-5p-activated Wnt/β-catenin pathway contributes to the progression of liver fibrosis.TGF-β/SMAD Pathway and Its Regulation in Hepatic Fibrosis.Curcumin Suppresses Intestinal Fibrosis by Inhibition of PPARγ-Mediated Epithelial-Mesenchymal Transition Molecular basis of organ fibrosis: potential therapeutic approaches.Curcumin targets multiple pathways to halt hepatic stellate cell activation: updated mechanisms in vitro and in vivo.Protective mechanisms of medicinal plants targeting hepatic stellate cell activation and extracellular matrix deposition in liver fibrosis.Transcriptional profiling and co-expression network analysis identifies potential biomarkers to differentiate chronic hepatitis B and the caused cirrhosis.MicroRNA-17-5p activates hepatic stellate cells through targeting of Smad7.Hepatic stellate cell is activated by microRNA-181b via PTEN/Akt pathway.Diosgenin attenuates hepatic stellate cell activation through transforming growth factor-β/Smad signaling pathway.Curcumin up-regulates phosphatase and tensin homologue deleted on chromosome 10 through microRNA-mediated control of DNA methylation--a novel mechanism suppressing liver fibrosis.Branched chain amino acids supplementation modulates TGF-β1/Smad signaling pathway and interleukins in CCl4 -induced liver fibrosis.Curcumin downregulates the expression of Snail via suppressing Smad2 pathway to inhibit TGF-β1-induced epithelial-mesenchymal transitions in hepatoma cells.Food components with antifibrotic activity and implications in prevention of liver disease.Transforming Growth Factor-β1/Smad3 Signaling and Redox Status in Experimentally Induced Nephrotoxicity: Impact of Carnosine.Curcumin induces apoptosis in human leukemic cell lines through an IFIT2-dependent pathway.New mechanism in the modulation of carbon tetrachloride hepatotoxicity in rats using different natural antioxidants.Curcumin protects against CCl4-induced liver fibrosis in rats by inhibiting HIF-1α through an ERK-dependent pathway.Nanocurcumin-Mediated Down-Regulation of Telomerase Via Stimulating TGFβ1 Signaling Pathway in Hepatocellular Carcinoma Cells.Antifibrogenic Influence of Mentha piperita L. Essential Oil against CCl4-Induced Liver Fibrosis in Rats.Herbal remedies for liver fibrosis: A review on the mode of action of fifty herbs.Effect of Curcumin and Gliotoxin on Rat Liver Myofibroblast Culture

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Inhibition by curcumin of multiple sites of the transforming growth factor-beta1 signalling pathway ameliorates the progression of liver fibrosis induced by carbon tetrachloride in rats

http://www.wikidata.org/entity/Q34414756

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2012 nî lūn-bûn

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2012 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

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2012 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2012年の論文

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2012年論文

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2012年論文

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Inhibition by curcumin of mult ...... y carbon tetrachloride in rats

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Inhibition by curcumin of mult ...... y carbon tetrachloride in rats

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Inhibition by curcumin of mult ...... y carbon tetrachloride in rats

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Bei-li Xu

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Chuan-tao Tu

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Hong-chun Liu

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Ji-yao Wang

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Qun-yan Yao

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Shun-cai Zhang

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P2860

Mechanisms of TGF-beta signaling from cell membrane to the nucleus

Role and interaction of connective tissue growth factor with transforming growth factor-beta in persistent fibrosis: A mouse fibrosis model

Profibrogenic transforming growth factor-beta/activin receptor-like kinase 5 signaling via connective tissue growth factor expression in hepatocytes

Taking aim at the extracellular matrix: CCN proteins as emerging therapeutic targets

Modern pathogenetic concepts of liver fibrosis suggest stellate cells and TGF-beta as major players and therapeutic targets

Curcumin prevents and reverses murine cardiac hypertrophy

IL-13 induces connective tissue growth factor in rat hepatic stellate cells via TGF-β-independent Smad signaling

An algorithm for the grading of activity in chronic hepatitis C. The METAVIR Cooperative Study Group

Mechanisms of hepatic fibrogenesis

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