Bmi1 loss produces an increase in astroglial cells and a decrease in neural stem cell population and proliferation. - Wikidata - awgldk - TriplyDB

Bmi1 loss produces an increase in astroglial cells and a decrease in neural stem cell population and proliferation.

Bmi1 loss produces an increase in astroglial cells and a decrease in neural stem cell population and proliferation.

http://www.wikidata.org/entity/Q34426800

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Radioresistance of Brain Tumors Histone methylation makes its mark on longevity GSK3beta regulates differentiation and growth arrest in glioblastoma Switches, excitable responses and oscillations in the Ring1B/Bmi1 ubiquitination system Ink4a and Arf differentially affect cell proliferation and neural stem cell self-renewal in Bmi1-deficient mice.Ars2 maintains neural stem-cell identity through direct transcriptional activation of Sox2 Retinal stem cells transplanted into models of late stages of retinitis pigmentosa preferentially adopt a glial or a retinal ganglion cell fate.Prospective identification of functionally distinct stem cells and neurosphere-initiating cells in adult mouse forebrain.Epigenetic regulation of adult neural stem cells: implications for Alzheimer's disease.MiR-203 Interplays with Polycomb Repressive Complexes to Regulate the Proliferation of Neural Stem/Progenitor Cells Bmi1 is expressed in postnatal myogenic satellite cells, controls their maintenance and plays an essential role in repeated muscle regeneration Polycomb group proteins: multi-faceted regulators of somatic stem cells and cancer.Linking cell signaling and the epigenetic machinery.Bmi-1 absence causes premature brain degeneration Bmi1 is down-regulated in the aging brain and displays antioxidant and protective activities in neurons Identification and characterization of Bmi-1-responding element within the human p16 promoter.Comparative gene expression analysis in mouse models for multiple sclerosis, Alzheimer's disease and stroke for identifying commonly regulated and disease-specific gene changes Antagonistic interplay between necdin and Bmi1 controls proliferation of neural precursor cells in the embryonic mouse neocortex.Transcriptional silencing by polycomb-group proteins Nuclear-encoded cytochrome c oxidase subunit 4 regulates BMI1 expression and determines proliferative capacity of high-grade gliomas Radiosensitization of Primary Human Glioblastoma Stem-like Cells with Low-Dose AKT Inhibition.Bmi-1 is essential for the tumorigenicity of neuroblastoma cells.Ezh2 maintains retinal progenitor proliferation, transcriptional integrity, and the timing of late differentiation Bmi-1: At the crossroads of physiological and pathological biology.Deletion of Shp2 in the brain leads to defective proliferation and differentiation in neural stem cells and early postnatal lethality Upregulation of the proto-oncogene Bmi-1 predicts a poor prognosis in pediatric acute lymphoblastic leukemia.A Perspective on the Role of microRNA-128 Regulation in Mental and Behavioral Disorders.Single-cell mRNA profiling identifies progenitor subclasses in neurospheres.Retinal degeneration depends on Bmi1 function and reactivation of cell cycle proteins.Cathepsin B and uPAR regulate self-renewal of glioma-initiating cells through GLI-regulated Sox2 and Bmi1 expression NPV-LDE-225 (Erismodegib) inhibits epithelial mesenchymal transition and self-renewal of glioblastoma initiating cells by regulating miR-21, miR-128, and miR-200 Epigenetic regulation of mammalian stem cells Bmi-1 cooperates with Foxg1 to maintain neural stem cell self-renewal in the forebrain.Heterozygous knockout of the Bmi-1 gene causes an early onset of phenotypes associated with brain aging.Transcription factors and neural stem cell self-renewal, growth and differentiation Bmi-1, stem cells and cancer.MicroRNA regulation of cancer stem cells and therapeutic implications.Epigenetics of neurological cancers.miR128-1 inhibits the growth of glioblastoma multiforme and glioma stem-like cells via targeting BMI1 and E2F3.Epigenetic regulation of aging stem cells.

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Bmi1 loss produces an increase in astroglial cells and a decrease in neural stem cell population and proliferation.

http://www.wikidata.org/entity/Q34426800

description

2005 nî lūn-bûn

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2005 թուականի Յունիսին հրատարակուած գիտական յօդուած

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2005 թվականի հունիսին հրատարակված գիտական հոդված

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2005年の論文

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Bmi1 loss produces an increase ...... population and proliferation.

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Bmi1 loss produces an increase ...... population and proliferation.

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Bmi1 loss produces an increase ...... population and proliferation.

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Bmi1 loss produces an increase ...... population and proliferation.

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Bmi1 loss produces an increase ...... population and proliferation.

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Bmi1 loss produces an increase ...... population and proliferation.

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JNEUROSCI.3452-04.2005

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Journal of Neuroscience

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Dana Hornfeld

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Daniel F Schorderet

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Dusan Zencak

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Ellen Tanger

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Francis L Munier

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Maarten van Lohuizen

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Merel Lingbeek

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Meriem Tekaya

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Muriel Jaquet

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Yvan Arsenijevic

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10.1523/JNEUROSCI.3452-04.2005

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