Bcl3 prevents acute inflammatory lung injury in mice by restraining emergency granulopoiesis.
about
NKT cell subsets as key participants in liver physiology and pathologyNF-κB, the first quarter-century: remarkable progress and outstanding questionsCutting edge: Pseudomonas aeruginosa abolishes established lung transplant tolerance by stimulating B7 expression on neutrophilsNovel multivariate methods for integration of genomics and proteomics data: applications in a kidney transplant rejection study.Modeling genome-wide dynamic regulatory network in mouse lungs with influenza infection using high-dimensional ordinary differential equations.The NF-κB regulator Bcl-3 governs dendritic cell antigen presentation functions in adaptive immunityThe oncoprotein and transcriptional regulator Bcl-3 governs plasticity and pathogenicity of autoimmune T cellsNeutrophil extracellular traps are pathogenic in primary graft dysfunction after lung transplantation.Alternative nuclear functions for NF-κB family membersDAP12 expression in lung macrophages mediates ischemia/reperfusion injury by promoting neutrophil extravasation.Cytokine systems approach demonstrates differences in innate and pro-inflammatory host responses between genetically distinct MERS-CoV isolates.Emergency granulopoiesis promotes neutrophil-dendritic cell encounters that prevent mouse lung allograft acceptance.The NF-κB regulator Bcl-3 modulates inflammation during contact hypersensitivity reactions in radioresistant cells.SUMOylation of sPRDM16 promotes the progression of acute myeloid leukemia.Meclizine Preconditioning Protects the Kidney Against Ischemia-Reperfusion Injury.Neutrophil expression of Fas ligand and perforin directs effector CD8 T cell infiltration into antigen-challenged skin.Sex-specific regulation of chemokine Cxcl5/6 controls neutrophil recruitment and tissue injury in acute inflammatory states.NADPH oxidase controls neutrophilic response to sterile inflammation in mice by regulating the IL-1α/G-CSF axis.Innate immunity and organ transplantation: focus on lung transplantation.Implications for Interleukin-33 in solid organ transplantationEndogenous memory CD8 T cells are activated within cardiac allografts without mediating rejection.Ischemia and reperfusion--from mechanism to translation.The innate immune system in transplantation.Role of Bcl-3 in solid tumorsIt takes two to tango: IκBs, the multifunctional partners of NF-κB.Five-year update on the mouse model of orthotopic lung transplantation: Scientific uses, tricks of the trade, and tips for success.Lung ischemia reperfusion injury: a bench-to-bedside review.Phosphatase Wip1 as a new therapeutic target for intestinal ischemia-reperfusion injury.Increased T cell glucose uptake reflects acute rejection in lung grafts.Peripheral Blood Gene Expression Changes Associated With Primary Graft Dysfunction After Lung Transplantation.The Diverse Biological Functions of Neutrophils, Beyond the Defense Against Infections.Mapping the Interaction of B Cell Leukemia 3 (BCL-3) and Nuclear Factor κB (NF-κB) p50 Identifies a BCL-3-mimetic Anti-inflammatory Peptide.Donor pulmonary intravascular nonclassical monocytes recruit recipient neutrophils and mediate primary lung allograft dysfunction.Noninvasive Imaging of CCR2+ Cells in Ischemia-Reperfusion Injury After Lung Transplantation.Longitudinal analysis of whole blood transcriptomes to explore molecular signatures associated with acute renal allograft rejection.Bcl3 regulates pro-survival and pro-inflammatory gene expression in cutaneous T-cell lymphoma.Gene expression in liver injury caused by long-term exposure to titanium dioxide nanoparticles in mice.Bcl-3 induced by IL-22 via STAT3 activation acts as a potentiator of psoriasis-related gene expression in epidermal keratinocytes.Report of the ISHLT Working Group on Primary Lung Graft Dysfunction Part III: Mechanisms: A 2016 Consensus Group Statement of the International Society for Heart and Lung Transplantation.Bcl-3 deficiency protects against dextran-sodium sulphate-induced colitis in the mouse.
P2860
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P2860
Bcl3 prevents acute inflammatory lung injury in mice by restraining emergency granulopoiesis.
description
2010 nî lūn-bûn
@nan
2010 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Bcl3 prevents acute inflammato ...... ning emergency granulopoiesis.
@ast
Bcl3 prevents acute inflammato ...... ning emergency granulopoiesis.
@en
Bcl3 prevents acute inflammato ...... ning emergency granulopoiesis.
@nl
type
label
Bcl3 prevents acute inflammato ...... ning emergency granulopoiesis.
@ast
Bcl3 prevents acute inflammato ...... ning emergency granulopoiesis.
@en
Bcl3 prevents acute inflammato ...... ning emergency granulopoiesis.
@nl
prefLabel
Bcl3 prevents acute inflammato ...... ning emergency granulopoiesis.
@ast
Bcl3 prevents acute inflammato ...... ning emergency granulopoiesis.
@en
Bcl3 prevents acute inflammato ...... ning emergency granulopoiesis.
@nl
P2093
P2860
P356
P1476
Bcl3 prevents acute inflammato ...... ning emergency granulopoiesis.
@en
P2093
Alexander S Krupnick
Andrew E Gelman
Daniel Kreisel
Jeremy Tietjens
Jihong Zhu
Ruaidhri J Carmody
Seiichiro Sugimoto
Sumiharu Yamamoto
P2860
P304
P356
10.1172/JCI42596
P407
P577
2010-12-13T00:00:00Z