West Nile virus (WNV) replication is independent of autophagy in mammalian cells.
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Biology of Zika Virus Infection in Human Skin CellsStress responses in flavivirus-infected cells: activation of unfolded protein response and autophagyThe Virus-Host Interplay: Biogenesis of +RNA Replication ComplexesApoptosis, autophagy and unfolded protein response pathways in Arbovirus replication and pathogenesisAdapting the Stress Response: Viral Subversion of the mTOR Signaling PathwayAutophagy is highly targeted among host comparative proteomes during infection with different virulent RABV strains.An essential role of PI3K in the control of West Nile virus infection.Regulation of cell survival and death during Flavivirus infections.Infection with Usutu virus induces an autophagic response in mammalian cells.Oxidative stress influences positive strand RNA virus genome synthesis and capping.Autophagy enhances the replication of classical swine fever virus in vitro.N-Desmethylclozapine, Fluoxetine, and Salmeterol Inhibit Postentry Stages of the Dengue Virus Life Cycle.Wild-type rabies virus induces autophagy in human and mouse neuroblastoma cell lines.Replication cycle and molecular biology of the West Nile virus.The Role of Secretory Autophagy in Zika Virus Transfer through the Placental Barrier.Antiviral autophagy restrictsRift Valley fever virus infection and is conserved from flies to mammals.Inhibition of autophagy limits vertical transmission of Zika virus in pregnant mice.Potential chemotherapeutic targets for Japanese encephalitis: current status of antiviral drug development and future challenges.Transcriptomic Analyses Reveal Differential Gene Expression of Immune and Cell Death Pathways in the Brains of Mice Infected with West Nile Virus and Chikungunya Virus.Reconciling West Nile virus with the autophagic pathwayTrehalose May Decrease the Transmission of Zika Virus to the Fetus by Activating Degradative Autophagy.Amino acid substitutions in the non-structural proteins 4A or 4B modulate the induction of autophagy in West Nile virus infected cells independently of the activation of the unfolded protein response.The Immune Responses of the Animal Hosts of West Nile Virus: A Comparison of Insects, Birds, and Mammals.Inflammation-Induced, STING-Dependent Autophagy Restricts Zika Virus Infection in the Drosophila BrainFlaviviruses and Autophagy
P2860
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P2860
West Nile virus (WNV) replication is independent of autophagy in mammalian cells.
description
2012 nî lūn-bûn
@nan
2012 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2012 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
name
West Nile virus (WNV) replication is independent of autophagy in mammalian cells.
@ast
West Nile virus (WNV) replication is independent of autophagy in mammalian cells.
@en
type
label
West Nile virus (WNV) replication is independent of autophagy in mammalian cells.
@ast
West Nile virus (WNV) replication is independent of autophagy in mammalian cells.
@en
prefLabel
West Nile virus (WNV) replication is independent of autophagy in mammalian cells.
@ast
West Nile virus (WNV) replication is independent of autophagy in mammalian cells.
@en
P2860
P1433
P1476
West Nile virus (WNV) replication is independent of autophagy in mammalian cells.
@en
P2093
Brenda L Fredericksen
Rianna Vandergaast
P2860
P304
P356
10.1371/JOURNAL.PONE.0045800
P407
P577
2012-09-21T00:00:00Z