Correlation of disease severity with fecal toxin levels in patients with Clostridium difficile-associated diarrhea and distribution of PCR ribotypes and toxin yields in vitro of corresponding isolates.
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The Regulatory Networks That Control Clostridium difficile Toxin SynthesisUltrasensitive Detection and Quantification of Toxins for Optimized Diagnosis of Clostridium difficile InfectionClostridium difficile: epidemiology, diagnostic and therapeutic possibilities-a systematic reviewUV-visible marker confirms that environmental persistence of Clostridium difficile spores in toilets of patients with C. difficile-associated diarrhea is associated with lack of compliance with cleaning protocolClostridium difficile infection: An overview of the disease and its pathogenesis, epidemiology and interventionsGlobal transcriptional control by glucose and carbon regulator CcpA in Clostridium difficile.Ribotype 027 Clostridium difficile infections with measurable stool toxin have increased lactoferrin and are associated with a higher mortalityTruncation in the tcdC region of the Clostridium difficile PathLoc of clinical isolates does not predict increased biological activity of Toxin B or Toxin ADoes Adjunctive Tigecycline Improve Outcomes in Severe-Complicated, Nonoperative Clostridium difficile Infection?Analysis of ultra low genome conservation in Clostridium difficile.Procalcitonin levels associate with severity of Clostridium difficile infectionClostridium difficile transcriptome analysis using pig ligated loop model reveals modulation of pathways not modulated in vitroIn Vitro Selection of a Single-Stranded DNA Molecular Recognition Element against Clostridium difficile Toxin B and Sensitive Detection in Human Fecal Matter.Emergence of an outbreak-associated Clostridium difficile variant with increased virulence.Quantification of Clostridium difficile in antibiotic-associated-diarrhea patientsClostridium difficile isolates with increased sporulation: emergence of PCR ribotype 002 in Hong Kong.Humanized microbiota mice as a model of recurrent Clostridium difficile diseaseCefoperazone-treated mice as an experimental platform to assess differential virulence of Clostridium difficile strainsDevelopment and Validation of Digital Enzyme-Linked Immunosorbent Assays for Ultrasensitive Detection and Quantification of Clostridium difficile Toxins in StoolModulation of toxin production by the flagellar regulon in Clostridium difficile.Outcomes in patients tested for Clostridium difficile toxins.Clostridium difficile ribotype does not predict severe infection.Increased sporulation rate of epidemic Clostridium difficile Type 027/NAP1Performance of Clostridium difficile toxin enzyme immunoassay and nucleic acid amplification tests stratified by patient disease severity.Review article: tolevamer, a novel toxin-binding polymer: overview of preclinical pharmacology and physicochemical properties.A pilot study to assess bacterial and toxin reduction in patients with Clostridium difficile infection given fidaxomicin or vancomycin.Proteomic Analysis and Label-Free Quantification of the Large Clostridium difficile Toxins.Poor functional status as a risk factor for severe Clostridium difficile infection in hospitalized older adults.Clostridium difficile infection in non-HIV-immunocompromised patients and in HIV-infected patients.Hype or hypervirulence: a reflection on problematic C. difficile strains.Real-time cellular analysis for quantitative detection of functional Clostridium difficile toxin in stool.Description and validation of a new automated surveillance system for Clostridium difficile in Denmark.Control of Clostridium difficile Physiopathology in Response to Cysteine AvailabilityAn exploratory study to evaluate Clostridium difficile polymerase chain reaction ribotypes and infection outcomesTolevamer, an anionic polymer, neutralizes toxins produced by the BI/027 strains of Clostridium difficile.Lack of association between clinical outcome of Clostridium difficile infections, strain type, and virulence-associated phenotypes.The key sigma factor of transition phase, SigH, controls sporulation, metabolism, and virulence factor expression in Clostridium difficile.Human hypervirulent Clostridium difficile strains exhibit increased sporulation as well as robust toxin production.Characterization of the sporulation initiation pathway of Clostridium difficile and its role in toxin production.Diagnosing Clostridium difficile-associated diarrhea using enzyme immunoassay: the clinical significance of toxin negativity in glutamate dehydrogenase-positive patients
P2860
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P2860
Correlation of disease severity with fecal toxin levels in patients with Clostridium difficile-associated diarrhea and distribution of PCR ribotypes and toxin yields in vitro of corresponding isolates.
description
2006 nî lūn-bûn
@nan
2006 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2006 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
name
Correlation of disease severit ...... tro of corresponding isolates.
@ast
Correlation of disease severit ...... tro of corresponding isolates.
@en
Correlation of disease severit ...... tro of corresponding isolates.
@nl
type
label
Correlation of disease severit ...... tro of corresponding isolates.
@ast
Correlation of disease severit ...... tro of corresponding isolates.
@en
Correlation of disease severit ...... tro of corresponding isolates.
@nl
prefLabel
Correlation of disease severit ...... tro of corresponding isolates.
@ast
Correlation of disease severit ...... tro of corresponding isolates.
@en
Correlation of disease severit ...... tro of corresponding isolates.
@nl
P2093
P2860
P921
P1476
Correlation of disease severit ...... tro of corresponding isolates.
@en
P2093
Asa Lagergren
Bo Svenungsson
Lars G Burman
Thomas Akerlund
P2860
P304
P356
10.1128/JCM.44.2.353-358.2006
P407
P577
2006-02-01T00:00:00Z