Impact of corticosterone treatment on spontaneous seizure frequency and epileptiform activity in mice with chronic epilepsy.
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The role of stress as a trigger for epileptic seizures: a narrative review of evidence from human and animal studiesBiomarkers of epileptogenesis: psychiatric comorbidities (?)Common Mechanisms Underlying Epileptogenesis and the Comorbidities of Epilepsy.Hypothalamic-pituitary-adrenocortical axis dysfunction in epilepsy.Gabapentin attenuates hyperexcitability in the freeze-lesion model of developmental cortical malformation.Olfactory Bulbectomy Leads to the Development of Epilepsy in Mice.Clonal Analysis of Newborn Hippocampal Dentate Granule Cell Proliferation and Development in Temporal Lobe EpilepsyImpact of rapamycin on status epilepticus induced hippocampal pathology and weight gain.Accumulation of abnormal adult-generated hippocampal granule cells predicts seizure frequency and severity.Ablation of Newly Generated Hippocampal Granule Cells Has Disease-Modifying Effects in EpilepsySeizure-induced disinhibition of the HPA axis increases seizure susceptibility.Impact of inhibitory constraint of interneurons on neuronal excitability.RU486 Mitigates Hippocampal Pathology Following Status Epilepticus.Bumetanide reduces seizure progression and the development of pharmacoresistant status epilepticus.Stress is associated with an increased risk of recurrent seizures in adults.Compromised GABAergic inhibition contributes to tumor-associated epilepsy.Chronic stress shifts the GABA reversal potential in the hippocampus and increases seizure susceptibility.Stress and Seizures: Space, Time and Hippocampal Circuits.Ablation of peri-insult generated granule cells after epilepsy onset halts disease progression.Can Neurochemical Changes of Mood Disorders Explain the Increase Risk of Epilepsy or its Worse Seizure Control?Hippocampal corticotropin-releasing hormone neurons support recognition memory and modulate hippocampal excitability.Inhibition of anandamide hydrolysis dampens the neuroendocrine response to stress in neonatal rats subjected to suboptimal rearing conditions.Functional disruption of stress modulatory circuits in a model of temporal lobe epilepsy.
P2860
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P2860
Impact of corticosterone treatment on spontaneous seizure frequency and epileptiform activity in mice with chronic epilepsy.
description
2012 nî lūn-bûn
@nan
2012 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2012 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2012年の論文
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2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
name
Impact of corticosterone treat ...... in mice with chronic epilepsy.
@ast
Impact of corticosterone treat ...... in mice with chronic epilepsy.
@en
Impact of corticosterone treat ...... in mice with chronic epilepsy.
@nl
type
label
Impact of corticosterone treat ...... in mice with chronic epilepsy.
@ast
Impact of corticosterone treat ...... in mice with chronic epilepsy.
@en
Impact of corticosterone treat ...... in mice with chronic epilepsy.
@nl
prefLabel
Impact of corticosterone treat ...... in mice with chronic epilepsy.
@ast
Impact of corticosterone treat ...... in mice with chronic epilepsy.
@en
Impact of corticosterone treat ...... in mice with chronic epilepsy.
@nl
P2093
P2860
P1433
P1476
Impact of corticosterone treat ...... in mice with chronic epilepsy.
@en
P2093
Jessica M McKlveen
Katherine D Holland
Margaret Gardner
Matthew Batie
Norberto Garcia-Cairasco
Olagide W Castro
Raymund Y K Pun
Steve C Danzer
Victor R Santos
P2860
P304
P356
10.1371/JOURNAL.PONE.0046044
P407
P577
2012-09-28T00:00:00Z