IL-23 compensates for the absence of IL-12p70 and is essential for the IL-17 response during tuberculosis but is dispensable for protection and antigen-specific IFN-gamma responses if IL-12p70 is available.

IL-23 compensates for the absence of IL-12p70 and is essential for the IL-17 response during tuberculosis but is dispensable for protection and antigen-specific IFN-gamma responses if IL-12p70 is available.

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http://www.wikidata.org/entity/Q34432140

Q34432140

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IL-23 modulates CD56+/CD3- NK cell and CD56+/CD3+ NK-like T cell function differentially from IL-12 The roles and functional mechanisms of interleukin-17 family cytokines in mucosal immunity Th1 and Th17 Cells in Tuberculosis: Protection, Pathology, and Biomarkers Complexity and Controversies over the Cytokine Profiles of T Helper Cell Subpopulations in Tuberculosis IL-23/IL-17A Dysfunction Phenotypes Inform Possible Clinical Effects from Anti-IL-17A Therapies Synthetic double-stranded RNAs are adjuvants for the induction of T helper 1 and humoral immune responses to human papillomavirus in rhesus macaques Critical and independent role for SOCS3 in either myeloid or T cells in resistance to Mycobacterium tuberculosis Quest for correlates of protection against tuberculosis IL-23 promotes tumour incidence and growth T cell-mediated host immune defenses in the lung Mycolic acid modification by the mmaA4 gene of M. tuberculosis modulates IL-12 production The landscape of host transcriptional response programs commonly perturbed by bacterial pathogens: towards host-oriented broad-spectrum drug targets Surface phenotype and antigenic specificity of human interleukin 17-producing T helper memory cells Biologic therapies in the treatment of psoriasis: a comprehensive evidence-based basic science and clinical review and a practical guide to tuberculosis monitoring.CD4+ T cells contain early extrapulmonary tuberculosis (TB) dissemination and rapid TB progression and sustain multieffector functions of CD8+ T and CD3- lymphocytes: mechanisms of CD4+ T cell immunity.Profiling early lung immune responses in the mouse model of tuberculosis.Tracing functional antigen-specific CCR6 Th17 cells after vaccination.Interleukin-17A mediates acquired immunity to pneumococcal colonization In a murine tuberculosis model, the absence of homeostatic chemokines delays granuloma formation and protective immunity.Th17 cells at the crossroads of innate and adaptive immunity against infectious diseases at the mucosa.Unexpected role for IL-17 in protective immunity against hypervirulent Mycobacterium tuberculosis HN878 infection.Cigarette smoke-induced oxidative stress suppresses generation of dendritic cell IL-12 and IL-23 through ERK-dependent pathways.IL-23 mediates inflammatory responses to mucoid Pseudomonas aeruginosa lung infection in mice.Directing traffic: IL-17 and IL-22 coordinate pulmonary immune defense.Th17 cytokines and vaccine-induced immunity.Mycobacterium tuberculosis, macrophages, and the innate immune response: does common variation matter?Interleukin 12B (IL12B) genetic variation and pulmonary tuberculosis: a study of cohorts from The Gambia, Guinea-Bissau, United States and Argentina Phosphorylated STAT3 and PD-1 regulate IL-17 production and IL-23 receptor expression in Mycobacterium tuberculosis infection Potent intestinal Th17 priming through peripheral lipopolysaccharide-based immunization.Th17 cytokines in mucosal immunity and inflammation.IL-22 mediates mucosal host defense against Gram-negative bacterial pneumonia.The roles of IL-12 and IL-23 in CD8+ T cell-mediated immunity against Listeria monocytogenes: Insights from a DC vaccination model.Pathological role of interleukin 17 in mice subjected to repeated BCG vaccination after infection with Mycobacterium tuberculosis.The role of Th17 cytokines in primary mucosal immunity Mycobacterium bovis BCG-specific Th17 cells confer partial protection against Mycobacterium tuberculosis infection in the absence of gamma interferon IL-17 in protective immunity to intracellular pathogens.Altered CD8(+) T cell frequency and function in tuberculous lymphadenitis.The Th1 immune response to Plasmodium falciparum circumsporozoite protein is boosted by adenovirus vectors 35 and 26 with a homologous insert.IL-23 p19 knockout mice exhibit minimal defects in responses to primary and secondary infection with Francisella tularensis LVS Beyond macrophages: the diversity of mononuclear cells in tuberculosis.

P2860

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P2860

IL-23 compensates for the absence of IL-12p70 and is essential for the IL-17 response during tuberculosis but is dispensable for protection and antigen-specific IFN-gamma responses if IL-12p70 is available.

Item

http://www.wikidata.org/entity/Q34432140

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2005 nî lūn-bûn

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2005 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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2005 թվականի հուլիսին հրատարակված գիտական հոդված

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2005年の論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年论文

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