A single nucleotide polymorphism in Tyk2 controls susceptibility to experimental allergic encephalomyelitis - Wikidata - awgldk - TriplyDB

A single nucleotide polymorphism in Tyk2 controls susceptibility to experimental allergic encephalomyelitis

A single nucleotide polymorphism in Tyk2 controls susceptibility to experimental allergic encephalomyelitis

http://www.wikidata.org/entity/Q34433877

about

Translational utility of experimental autoimmune encephalomyelitis: recent developments Forward genetic approaches for elucidation of novel regulators of Lyme arthritis severity Enabling structure-based drug design of Tyk2 through co-crystallization with a stabilizing aminoindazole inhibitor Identification of Orch3, a locus controlling dominant resistance to autoimmune orchitis, as kinesin family member 1C The postnatal maternal environment affects autoimmune disease susceptibility in A/J mice.Allelic variation in the Tyk2 and EGF genes as potential genetic determinants of CNS repair.Genetics of experimental allergic encephalomyelitis supports the role of T helper cells in multiple sclerosis pathogenesis.Impact of microbes on autoimmune diseases.Tyrosine kinases in inflammatory dermatologic disease.Inhibition of TYK2 and JAK1 ameliorates imiquimod-induced psoriasis-like dermatitis by inhibiting IL-22 and the IL-23/IL-17 axis.Exacerbation of autoimmune neuroinflammation by dietary sodium is genetically controlled and sex specific.Progress in multiple sclerosis genetics.Establishing the role of tyrosine kinase 2 in cancer.Sex-specific control of central nervous system autoimmunity by p38 mitogen-activated protein kinase signaling in myeloid cells.Histamine H₂ receptor signaling × environment interactions determine susceptibility to experimental allergic encephalomyelitis.Interferons in the central nervous system: a few instruments play many tunes.Tyrosine kinase 2 controls IL-1ß production at the translational level.Modeling month-season of birth as a risk factor in mouse models of chronic disease: from multiple sclerosis to autoimmune encephalomyelitis.

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P2860

A single nucleotide polymorphism in Tyk2 controls susceptibility to experimental allergic encephalomyelitis

http://www.wikidata.org/entity/Q34433877

description

2009 nî lūn-bûn

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2009 թուականի Յունիսին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի հունիսին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年论文

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name

A single nucleotide polymorphi ...... tal allergic encephalomyelitis

@ast

A single nucleotide polymorphi ...... tal allergic encephalomyelitis

@en

A single nucleotide polymorphi ...... tal allergic encephalomyelitis

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type

label

A single nucleotide polymorphi ...... tal allergic encephalomyelitis

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A single nucleotide polymorphi ...... tal allergic encephalomyelitis

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A single nucleotide polymorphi ...... tal allergic encephalomyelitis

@nl

prefLabel

A single nucleotide polymorphi ...... tal allergic encephalomyelitis

@ast

A single nucleotide polymorphi ...... tal allergic encephalomyelitis

@en

A single nucleotide polymorphi ...... tal allergic encephalomyelitis

@nl

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Journal of Immunology

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A single nucleotide polymorphi ...... tal allergic encephalomyelitis

@en

P2093

Ben McElvany

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Cory Teuscher

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Elizabeth P Blankenhorn

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Karen M Spach

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Rajkumar Noubade

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William F Hickey

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P2860

A receptor for the heterodimeric cytokine IL-23 is composed of IL-12Rbeta1 and a novel cytokine receptor subunit, IL-23R

Interleukin 12 (IL-12) induces tyrosine phosphorylation of JAK2 and TYK2: differential use of Janus family tyrosine kinases by IL-2 and IL-12

Polymorphisms in the tyrosine kinase 2 and interferon regulatory factor 5 genes are associated with systemic lupus erythematosus

Association scan of 14,500 nonsynonymous SNPs in four diseases identifies autoimmunity variants

A natural mutation in the Tyk2 pseudokinase domain underlies altered susceptibility of B10.Q/J mice to infection and autoimmunity

Signaling through the JAK/STAT pathway, recent advances and future challenges

Association and activation of Jak-Tyk kinases by CNTF-LIF-OSM-IL-6 beta receptor components

Pertussis toxin and target eukaryotic cells: binding, entry, and activation

Human tyrosine kinase 2 deficiency reveals its requisite roles in multiple cytokine signals involved in innate and acquired immunity

IL-23 drives a pathogenic T cell population that induces autoimmune inflammation

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scholarly article

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10.4049/JIMMUNOL.0900142

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12

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2009-06-01T00:00:00Z

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26264707

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19494301

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single-nucleotide polymorphism

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3008628

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