Targeted deletion of both thymidine phosphorylase and uridine phosphorylase and consequent disorders in mice
about
MitochondriaThymidine Phosphorylase Participates in Platelet Signaling and Promotes ThrombosisInherited mitochondrial DNA depletion.Modeling pathogenic mutations of human twinkle in Drosophila suggests an apoptosis role in response to mitochondrial defects.Long term expression of Drosophila melanogaster nucleoside kinase in thymidine kinase 2-deficient mice with no lethal effects caused by nucleotide pool imbalances.Mitochondrial Diseases Part II: Mouse models of OXPHOS deficiencies caused by defects in regulatory factors and other components required for mitochondrial function.Mitochondrial ATP transporter Ant2 depletion impairs erythropoiesis and B lymphopoiesisThe role of thymidine phosphorylase, an angiogenic enzyme, in tumor progression.A discovery resource of rare copy number variations in individuals with autism spectrum disorder.Mouse models of mitochondrial DNA defects and their relevance for human disease.Unbalanced deoxynucleotide pools cause mitochondrial DNA instability in thymidine phosphorylase-deficient miceTwo transgenic mouse models for β-subunit components of succinate-CoA ligase yielding pleiotropic metabolic alterationsScreen for abnormal mitochondrial phenotypes in mouse embryonic stem cells identifies a model for succinyl-CoA ligase deficiency and mtDNA depletion.Mitochondrial Neurogastrointestinal Encephalomyopathy Caused by Thymidine Phosphorylase Enzyme Deficiency: From Pathogenesis to Emerging Therapeutic Options.Functional analysis of pyrimidine biosynthesis enzymes using the anticancer drug 5-fluorouracil in Caenorhabditis elegans.Nutritional Interventions for Mitochondrial OXPHOS Deficiencies: Mechanisms and Model Systems.Mitochondrial dysfunction and ovarian aging.Relationship between biodistribution of a novel thymidine phosphorylase (TP) imaging probe and TP expression levels in normal mice.Immunohistochemical detection of platelet-derived endothelial cell growth factor/thymidine phosphorylase and angiopoietins in ameloblastic tumors.Capecitabine reverses tumor escape from anti-VEGF through the eliminating CD11bhigh/Gr1high myeloid cells.
P2860
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P2860
Targeted deletion of both thymidine phosphorylase and uridine phosphorylase and consequent disorders in mice
description
2002 nî lūn-bûn
@nan
2002 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2002 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2002年の論文
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2002年論文
@yue
2002年論文
@zh-hant
2002年論文
@zh-hk
2002年論文
@zh-mo
2002年論文
@zh-tw
2002年论文
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name
Targeted deletion of both thym ...... d consequent disorders in mice
@ast
Targeted deletion of both thym ...... d consequent disorders in mice
@en
Targeted deletion of both thym ...... d consequent disorders in mice
@nl
type
label
Targeted deletion of both thym ...... d consequent disorders in mice
@ast
Targeted deletion of both thym ...... d consequent disorders in mice
@en
Targeted deletion of both thym ...... d consequent disorders in mice
@nl
prefLabel
Targeted deletion of both thym ...... d consequent disorders in mice
@ast
Targeted deletion of both thym ...... d consequent disorders in mice
@en
Targeted deletion of both thym ...... d consequent disorders in mice
@nl
P2093
P2860
P1476
Targeted deletion of both thym ...... d consequent disorders in mice
@en
P2093
Atsuo Nakayama
Fuyuki Ishikawa
Hideo Toyoshima
Hideo Utsumi
Hiroaki Kawaguchi
Hiroaki Tsujimoto
Hiroki Yoshida
Hiromitsu Hara
Itsuro Higuchi
Junko Hirato
P2860
P304
P356
10.1128/MCB.22.14.5212-5221.2002
P407
P577
2002-07-01T00:00:00Z