Basal bioenergetic abnormalities in skeletal muscle from ryanodine receptor malignant hyperthermia-susceptible R163C knock-in mice.
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Perspectives on: SGP symposium on mitochondrial physiology and medicine: molecular identities of mitochondrial Ca2+ influx mechanism: updated passwords for accessing mitochondrial Ca2+-linked health and diseaseReview of RyR1 pathway and associated pathomechanismsMalignant hyperthermia susceptibility arising from altered resting coupling between the skeletal muscle L-type Ca2+ channel and the type 1 ryanodine receptorGene dose influences cellular and calcium channel dysregulation in heterozygous and homozygous T4826I-RYR1 malignant hyperthermia-susceptible muscle.Mice expressing T4826I-RYR1 are viable but exhibit sex- and genotype-dependent susceptibility to malignant hyperthermia and muscle damageOxidative stress and successful antioxidant treatment in models of RYR1-related myopathyImpact of a novel homozygous mutation in nicotinamide nucleotide transhydrogenase on mitochondrial DNA integrity in a case of familial glucocorticoid deficiency.Early mitochondrial abnormalities in hippocampal neurons cultured from Fmr1 pre-mutation mouse model.Nonspecific sarcolemmal cation channels are critical for the pathogenesis of malignant hyperthermia.Interactions between sarco-endoplasmic reticulum and mitochondria in cardiac and skeletal muscle - pivotal roles in Ca²⁺ and reactive oxygen species signaling.Unveiling the Metabolic Changes on Muscle Cell Metabolism Underlying p-Phenylenediamine Toxicity.Cardiac mitochondrial proteomic expression in inbred rat strains divergent in survival time after hemorrhage.Adiponectin inhibits neutrophil apoptosis via activation of AMP kinase, PKB and ERK 1/2 MAP kinase.Mitochondrial-nuclear crosstalk, haplotype and copy number variation distinct in muscle fiber type, mitochondrial respiratory and metabolic enzyme activities.Zdhhc13-dependent Drp1 S-palmitoylation impacts brain bioenergetics, anxiety, coordination and motor skills.Fiber hypertrophy and increased oxidative capacity can occur simultaneously in pig glycolytic skeletal muscle.Malignant Hyperthermia in the Post-Genomics Era: New Perspectives on an Old Concept.Mitochondrial cardiomyopathies feature increased uptake and diminished efflux of mitochondrial calcium.Molecular changes in mitochondrial respiratory activity and metabolic enzyme activity in muscle of four pig breeds with distinct metabolic types.
P2860
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P2860
Basal bioenergetic abnormalities in skeletal muscle from ryanodine receptor malignant hyperthermia-susceptible R163C knock-in mice.
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2010 nî lūn-bûn
@nan
2010 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2010年の論文
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2010年論文
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2010年論文
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2010年論文
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2010年論文
@zh-mo
2010年論文
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2010年论文
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name
Basal bioenergetic abnormaliti ...... sceptible R163C knock-in mice.
@ast
Basal bioenergetic abnormaliti ...... sceptible R163C knock-in mice.
@en
Basal bioenergetic abnormaliti ...... sceptible R163C knock-in mice.
@nl
type
label
Basal bioenergetic abnormaliti ...... sceptible R163C knock-in mice.
@ast
Basal bioenergetic abnormaliti ...... sceptible R163C knock-in mice.
@en
Basal bioenergetic abnormaliti ...... sceptible R163C knock-in mice.
@nl
prefLabel
Basal bioenergetic abnormaliti ...... sceptible R163C knock-in mice.
@ast
Basal bioenergetic abnormaliti ...... sceptible R163C knock-in mice.
@en
Basal bioenergetic abnormaliti ...... sceptible R163C knock-in mice.
@nl
P2093
P2860
P356
P1476
Basal bioenergetic abnormaliti ...... sceptible R163C knock-in mice.
@en
P2093
Alicja Omanska-Klusek
Catherine Ross-Inta
Danielle Sakaguchi
Eleonora Napoli
Genaro Barrientos
Isaac N Pessah
Paul D Allen
P2860
P304
P356
10.1074/JBC.M110.153247
P407
P577
2010-10-26T00:00:00Z